An implant for repairing a heart valve

Abstract

The application relates to the field of medical devices, in particular to an implant for repairing a heart valve, which comprises a fixing support, an anti-reflux prosthesis, a fixing member and a limiting mechanism, the anti-reflux prosthesis is anchored on atrial tissue through the fixing member, one end of the limiting mechanism is fixed on ventricular tissue or apical tissue, characterized in that the anti-reflux prosthesis comprises a closing aid and an adjusting framework, the adjusting framework comprises a plurality of adjusting units and connecting sections connected between the adjusting units, and the other end of the limiting mechanism is connected with the adjusting units respectively; the application can effectively avoid the occurrence of adverse conditions such as wrinkles, thrombus accumulation and calcification of the closing aid, and has good clinical significance.

Description

Technical Field

[0001] This application relates to the field of medical devices, and more particularly to an implant for repairing heart valves. Background Technology

[0002] The mitral valve is located at the left atrioventricular orifice and consists of five parts: the valve annulus, leaflets, chordae tendineae, papillary muscles, and commissural junction. Its accurate anatomical name is the mitral apparatus or mitral complex.

[0003] The tricuspid valve is located at the right atrioventricular orifice and has three approximately triangular velamentous valves. The base of the valves is attached to the fibrous annulus at the atrioventricular orifice. Functionally, the fibrous annulus, valves, chordae tendineae, and papillary muscles are visible, forming the tricuspid valve complex.

[0004] Mitral regurgitation can be divided into two types: 1. Rheumatic mitral regurgitation, mainly caused by mitral valve insufficiency, which can cause blood to flow in the opposite direction, resulting in the mixing of different blood types and a decrease in the heart's pumping and oxygen-carrying capacity. 2. Non-rheumatic mitral regurgitation, which usually refers to varying degrees of mitral regurgitation caused by abnormalities in the mitral valve itself and its surrounding anatomical structures, excluding rheumatic valvular disease. There are many causes of non-rheumatic mitral regurgitation, the most common being: mitral valve prolapse, papillary muscle dysfunction or chordae tendineae rupture, left atrial myxoma, valvular annular calcification, congenital valvular malformations, and infective endocarditis. Mitral regurgitation can also be classified into three types: functional, degenerative, and mixed. The most common are degenerative and functional mitral regurgitation. Functional mitral regurgitation is generally secondary to impaired left ventricular wall motion function, left ventricular dilation, and papillary muscle dysfunction, and is commonly seen in patients with heart failure. This group of patients also includes ischemic mitral regurgitation secondary to coronary artery disease and mitral regurgitation related to non-ischemic cardiomyopathy. Degenerative mitral regurgitation is generally considered to be caused by pathological changes in the valve structure or subvalvular structures, including abnormal extension or rupture of the chordae tendineae.

[0005] Patent CN202210259928.6 A repair device for preventing valvular regurgitation is disclosed, comprising a fixation unit, a closure aid, and an adjustment unit. The fixation unit is connected to the closure aid and anchored to atrial tissue or the autologous valve annulus. The closure aid has a first frame, and one end of the adjustment unit is connected to the first frame. The proximal end of the adjustment unit is fixed to the apex of the heart or ventricular tissue. When the closure aid moves with the autologous valve leaflet, the adjustment unit slides relative to the first frame. This relative sliding allows the closure aid to adjust its position in real time according to the direction of blood flow impact during torsion or return. Although the adjustment unit in this patented solution can adapt to the torsional stroke of the closure aid and make adaptive stroke changes, because the closure aid is a single structure, the adjustment unit exerts a pulling force on the closure aid when subjected to blood flow impact. This only causes the closure aid to be pulled as a whole, rather than allowing for relatively independent adjustments to local areas. This can lead to local wrinkles in the closure aid, which, over time, can cause blood accumulation, resulting in thrombosis, calcification, and other adverse conditions.

[0006] In summary, the existing repair devices have at least the following technical drawbacks: when subjected to blood flow impact, the existing closure aids can only be adjusted by the overall swing of the closure aid, which means that the adjustment cannot specifically adjust the local areas that need adjustment. This results in the closure aid not being properly adjusted, causing wrinkles to form in the closure aid, which in turn affects the repair effect. Summary of the Invention

[0007] This application is made in view of the above and other ideas.

[0008] One of the purposes of this application is to overcome the shortcomings of the prior art, such as the inability of the adjustment component of the closure aid to make targeted local adjustments to the closure aid when subjected to blood flow impact, which leads to wrinkles in the closure aid and subsequently problems such as thrombosis and calcification. This application provides an implant for repairing heart valves.

[0009] The technical solution adopted to solve the technical problem of the present invention is to provide an implant for repairing heart valves, including a fixation stent, an anti-reflux prosthesis, a fixation element, and a restraining mechanism. The anti-reflux prosthesis is anchored to the atrial tissue by the fixation element, and one end of the restraining mechanism is fixed to the ventricular tissue or the apical tissue. Due to the characteristics of the physiological anatomy of the heart, the P2 region of the autologous valve annulus (i.e., the anchoring position of the fixation stent) forms a 6-8° inclination angle to the apex of the heart (i.e., the location where the restraining mechanism is fixed). Therefore, after the anti-reflux prosthesis is implanted, the closure aid will deflect to one side. The anti-reflux prosthesis includes a closure aid and an adjustment frame. The adjustment frame includes multiple adjustment units and connecting segments connecting the adjustment units. The other end of the restraining mechanism is respectively connected to multiple of the aforementioned components. The adjustment unit connection specifically includes a first adjustment unit, a second adjustment unit, a third adjustment unit, a first connecting segment, and a second connecting segment. The second and third adjustment units are respectively located on the left and right sides of the first adjustment unit. The first adjustment unit is connected to the second and third adjustment units through the first and second connecting segments. When the closure aid is subjected to blood flow impact, the first, second, and third adjustment units can adjust the corresponding areas on the closure aid. At the same time, the connecting segment can both support the distal part of the closure aid and maintain the relative independence of the first, second, and third adjustment units, so as to facilitate targeted adjustment of local areas of the closure aid.

[0010] As a further improvement of the present invention, the first adjustment unit is disposed in the central region of the closure aid, and the second and third adjustment units are disposed on the left and right sides of the closure aid. Due to the characteristics of the physiological and anatomical structure of the mitral valve, when the valve is closed, the autologous anterior leaflet is divided into three main occlusive regions (A1, A2, and A3) from a top view. Therefore, when the closure aid occludes with the autologous anterior leaflet after implantation, the central region and the left and right sides of the closure aid need to extensively adhere to the autologous anterior leaflet. Therefore, the second and third adjustment units are disposed on the left and right sides of the first adjustment unit, respectively.

[0011] As a further improvement of the present invention, the stiffness of the first adjustment unit, the second adjustment unit and the third adjustment unit is greater than the stiffness of the first connecting segment and the second connecting segment; the purpose of this design is that when the limiting mechanism pulls the first adjustment unit, the second adjustment unit and the third adjustment unit, the three adjustment units can make a powerful adjustment to the closure aid, so as to avoid the closure aid from wrinkling.

[0012] As a further improvement of the present invention, the first connecting segment and the second connecting segment are flexible segments, and when the closure aid is subjected to blood flow impact, the first connecting segment and the second connecting segment will deform in accordance with the impact of blood flow; this makes the three adjustment units relatively independent, and each adjustment unit can adjust the corresponding adjustment area of ​​the closure aid in a targeted manner.

[0013] As a further improvement of the present invention, the first connecting segment and the second connecting segment are elastic, so that the first connecting segment and the second connecting segment can recover to the preset shape after being deformed by blood flow impact; the first connecting segment and the second connecting segment are both flexible segments, which can maintain the relative independence between the three adjustment units. At the same time, the first connecting segment and the second connecting segment are elastic, which allows the connecting segment to recover the closure aid to its original shape after deforming in accordance with blood flow impact. This ensures that even if the closure aid is subjected to different degrees of blood flow impact (the heart pumps blood at different rates under different conditions), it can still recover to its initial shape, effectively avoiding wrinkles in the closure aid after implantation, which could lead to adverse conditions such as thrombosis and calcification.

[0014] As a further improvement of the present invention, the width of the adjustment frame does not exceed 35mm. The setting value of the width of the adjustment frame can be determined according to the size of the ventricles of different patients, but it usually does not exceed 35mm (refer to the values ​​of a large number of existing cardiac physiological and anatomical measurements). This is because if the width of the adjustment frame is too wide during ventricular contraction, the adjustment frame will collide with the myocardium in the ventricle, causing damage to the myocardium.

[0015] As a further improvement of the present invention, the first adjustment unit, the second adjustment unit, and the third adjustment unit are configured with an arc-shaped structure.

[0016] As a further improvement of the present invention, the first adjustment unit, the second adjustment unit, and the third adjustment unit are configured with an elliptical structure.

[0017] As a further improvement of the present invention, the first connecting segment and the second connecting segment are configured with a wavy or "Z" shaped structure.

[0018] As a further improvement of the present invention, the limiting mechanism includes a first limiting member, a second limiting member, and a third limiting member, wherein the other ends of the first limiting member, the second limiting member, and the third limiting member are respectively connected to the first adjusting unit, the second adjusting unit, and the third adjusting unit.

[0019] As a further improvement of the present invention, the length of the first limiting member is greater than the length of the third limiting member, and the length of the third limiting member is greater than the length of the second limiting member.

[0020] As a further improvement of the present invention, the first limiting member, the second limiting member and the third limiting member are merged into one in the proximal portion and fixed to the apical tissue or papillary muscle.

[0021] As a further improvement of the present invention, the proximal portion of the closure aid is provided with a shape retention rod, which is arranged in an arched structure.

[0022] As a further improvement of the present invention, the left and right sides of the proximal portion of the closure aid are provided with curling structures, the curling structures causing the left and right sides of the proximal portion of the closure aid to curl backward.

[0023] As a further improvement of the present invention, the curling structure is disposed on the left and right sides of the connection between the closure aid and the fixed bracket.

[0024] Compared with the prior art, the advantages of the technical solution of this application include at least the following: In existing technologies, when closure aids are subjected to blood flow impact, their adjustment components cannot specifically adjust localized areas of the aid, leading to wrinkles and subsequent problems such as thrombosis and calcification. According to a concept proposed in this application, the adjustment framework consists of multiple adjustment units and connecting segments linking these units. Furthermore, multiple limiting components are connected to each adjustment unit, allowing the limiting components to work in conjunction with the corresponding adjustment units to adjust specific areas of the aid when subjected to blood flow impact. This adjustment method is more targeted than existing technologies and can precisely address the specific needs. The adjustment of the local area is effective. At the same time, the connecting segment can ensure that each adjustment unit maintains relative independence and can also provide effective support for the distal part of the closure aid. This further ensures that the closure aid can buffer and adapt to deformation after being subjected to different degrees of blood flow impact, avoiding stress concentration in certain local areas of the closure aid and causing damage. Moreover, after the blood flow impact subsides, the closure aid can quickly return to its initial state, effectively preventing wrinkles in the closure aid. Over time, this can lead to adverse conditions such as thrombus accumulation and calcification, which has great clinical significance.

[0025] According to a concept of this application, the stiffness of the first adjustment unit, the second adjustment unit, and the third adjustment unit is greater than the stiffness of the first connecting section and the second connecting section. The purpose of this design is that when the limiting mechanism pulls the first adjustment unit, the second adjustment unit, and the third adjustment unit, the three adjustment units can make a powerful adjustment to the closure aid, so as to avoid the closure aid from wrinkling.

[0026] According to a concept of this application, the first connecting segment and the second connecting segment are flexible segments. The purpose is that when the closure aid is subjected to blood flow impact, the first connecting segment and the second connecting segment will deform in accordance with the impact of blood flow. This allows the three adjustment units to maintain relative independence, and each adjustment unit can specifically adjust the corresponding adjustment area of ​​the closure aid. At the same time, the first connecting segment and the second connecting segment are also elastic, so that even if the closure aid is subjected to different degrees of blood flow impact (the heart pumps blood at different rates under different conditions), it can still return to its initial shape, effectively avoiding wrinkles in the closure aid after implantation, which could lead to adverse conditions such as thrombosis and calcification.

[0027] According to a concept of this application, the width of the adjustable skeleton does not exceed 35mm. The purpose of this design is to effectively prevent the adjustable skeleton from colliding with the myocardium during the heart's systolic phase, thus effectively protecting the heart tissue.

[0028] According to a concept of this application, the left and right sides of the proximal portion of the closure aid are provided with coiled structures. The coiled structures cause the left and right sides of the proximal portion of the closure aid to curl backward. When the ventricle is in systole, blood rushes towards the central region of the closure aid and flows to both sides along the central region of the closure aid. The coiled structures on the left and right sides of the closure aid allow the blood flowing to both sides to meander, effectively preventing blood from leaking out from the left and right sides of the closure aid, avoiding backflow, and ensuring its repair effect.

[0029] The embodiments of this application can achieve other advantageous technical effects not listed one by one. These other technical effects may be partially described below and can be expected and understood by those skilled in the art after reading this application. Attached Figure Description

[0030] The above-described features and advantages, as well as other features and advantages, and the ways in which they are implemented, of these embodiments will become more apparent and the embodiments of this application will be better understood by referring to the following description in conjunction with the accompanying drawings, in which: Figure 1 This is a front view of the overall structure of the implant of the present invention.

[0031] Figure 2 This is a rear view of the implant of the present invention.

[0032] Figure 3 This is a side view of the implant of the present invention.

[0033] Figure 4 This is a schematic diagram of the overall structure of the implant of the present invention positioned in the heart.

[0034] Figure 5This is a schematic diagram illustrating the principle of adjusting the corresponding area on the closure aid when it is subjected to blood flow impact.

[0035] Figure 6 for Figure 5 Enlarged view of part A in the middle.

[0036] Figure 7 for Figure 5 Rear view.

[0037] Figure 8 This is a schematic diagram of the mitral valve region.

[0038] Figure 9 A diagram illustrating the alignment of the closure element with the anterior leaflet after implantation of the anti-reflux prosthesis. The features represented by the numbers in the attached diagram are as follows: 1-Fixed bracket, 2-Anti-backflow prosthesis, 21-Closing aid, 22-Adjusting frame, 221-First adjustment unit, 222-Second adjustment unit, 223-Third adjustment unit, 224-First connecting section, 225-Second connecting section, 3-Fixed component, 4-Restriction mechanism, 41-First restriction component, 42-Second restriction component, 43-Third restriction component, 5-Shape holding rod, 6-Curled structure. Implementation

[0039] The details of one or more embodiments of this application will be set forth in the following description of the accompanying drawings and specific embodiments. Other features, objects, and advantages of this application will become clear from these descriptions, drawings, and claims.

[0040] It should be understood that the illustrated and described embodiments are not limited in application to the details of the construction and arrangement of the components set forth in the following description or illustrated in the accompanying drawings. The illustrated embodiments may be other embodiments and can be implemented or performed in various ways. The examples are provided by way of explanation rather than limitation of the disclosed embodiments. In fact, it will be apparent to those skilled in the art that various modifications and variations can be made to the embodiments of this application without departing from the scope or spirit of this disclosure. For example, features illustrated or described as part of one embodiment may be used with another embodiment to still produce another embodiment. Therefore, this disclosure covers such modifications and variations that fall within the scope of the appended claims and their equivalents.

[0041] Similarly, it is understood that the phrases and terms used in this document are for descriptive purposes and should not be considered restrictive. The use of “including,” “contains,” or “has,” and their variations, in this document is intended to include, in an open-ended manner, the items listed thereafter, their equivalents, and any additional items.

[0042] The present application will now be described in more detail with reference to various embodiments and examples of several aspects thereof.

[0043] In this application, the terms "other end" or "proximal end" or "proximal side" refer to the end or side closer to the centrifuge tip, and "one end" or "distal end" or "distal side" refer to the end or side farther from the centrifuge tip.

[0044] One of the objectives of the embodiments described below is to address the aforementioned deficiencies, as well as other problems. Example

[0045] like Figures 1-3 The illustration shows an implant for repairing a heart valve according to an embodiment of this application, used to repair the mitral valve. Common methods for mitral valve repair include leaflet clamping and annular reduction. This solution uses a repair approach that replaces the posterior leaflet of the autologous valve, increasing the contact area with the anterior leaflet to reduce regurgitation. Specifically, the implant for repairing the heart valve includes a fixation stent 1, an anti-regurgitation prosthesis 2, a fixation element 3, and a restraining mechanism 4. The anti-regurgitation prosthesis 2 is anchored to the atrial tissue by the fixation element 3, and one end of the restraining mechanism 4 is fixed to the ventricular tissue or the apical tissue. Figure 4 As shown, due to the characteristics of the heart's physiological anatomy, the P2 region of the autologous valve annulus (i.e., the anchoring position of the fixation stent 1) is inclined at a 6-8° angle to the apex of the heart (i.e., the location where the restricting mechanism 4 is fixed). Therefore, after the anti-reflux prosthesis 2 is implanted, the closure aid 21 will deflect to one side. The anti-reflux prosthesis 2 includes the closure aid 21 and an adjusting frame 22. The adjusting frame 22 includes a first adjusting unit 221, a second adjusting unit 222, a third adjusting unit 223, a first connecting segment 224, and a second connecting segment 225. The second adjusting unit 222 and the third adjusting unit 223 are respectively located on the left and right sides of the first adjusting unit 221. The first adjusting unit 221 is connected to the first connecting segment 225. Section 224 and the second connecting section 225 are connected to the second adjusting unit 222 and the third adjusting unit 223, and the other end of the limiting mechanism 4 is connected to the first adjusting unit 221, the second adjusting unit 222 and the third adjusting unit 223 respectively. When the closure aid 21 is subjected to blood flow impact, the first adjusting unit 221, the second adjusting unit 222 and the third adjusting unit 223 can adjust the corresponding area on the closure aid 21. At the same time, the connecting section can not only support the distal part of the closure aid 21, but also allow the first adjusting unit 221, the second adjusting unit 222 and the third adjusting unit 223 to maintain relative independence, so as to make targeted adjustments to local areas of the closure aid 21.

[0046] In this embodiment, the first adjustment unit 221 is disposed in the central region of the closure aid 21, and the second adjustment unit 222 and the third adjustment unit 223 are disposed on the left and right sides of the closure aid 21, as shown below. Figure 2 As mentioned above; due to the physiological and anatomical characteristics of the mitral valve, when the valve is closed, the anterior leaflet, viewed from above, consists of three main occlusal regions (A1, A2, and A3), as... Figure 8 Therefore, when the closure aid 21 aligns with the autologous anterior leaflet after implantation, the central area and both sides of the closure aid 21 need to extensively conform to the autologous anterior leaflet. Therefore, the second adjustment unit 222 and the third adjustment unit 223 are respectively located on the left and right sides of the first adjustment unit 221. Figure 9 As shown.

[0047] In this embodiment, the stiffness of the first adjustment unit 221, the second adjustment unit 222, and the third adjustment unit 223 is greater than the stiffness of the first connecting segment 224 and the second connecting segment 225. The purpose of this design is that when the limiting mechanism 4 pulls the first adjustment unit 221, the second adjustment unit 222, and the third adjustment unit 223, the three adjustment units can make a powerful adjustment to the closure aid 21, so as to avoid the closure aid 21 from wrinkling.

[0048] In this embodiment, the first connecting segment 224 and the second connecting segment 225 are flexible segments, and when the closure aid 21 is subjected to blood flow impact, the first connecting segment 224 and the second connecting segment 225 will deform in accordance with the impact of the blood flow, such as... Figures 5-7 As shown; this allows the three adjustment units to maintain relative independence, and each adjustment unit can specifically adjust the adjustment area corresponding to the auxiliary closing element 21.

[0049] In this embodiment, the first connecting segment 224 and the second connecting segment 225 are elastic, so that after the first connecting segment 224 and the second connecting segment 225 are deformed due to blood flow impact, they can return to a preset shape. The first connecting segment 224 and the second connecting segment 225 are both flexible segments, which can maintain relative independence between the three adjustment units. At the same time, the first connecting segment 224 and the second connecting segment 225 are also elastic, which allows the connecting segments to restore the closure aid 21 to its original shape after deforming in accordance with blood flow impact. This ensures that even if the closure aid 21 is subjected to different degrees of blood flow impact (the heart pumps blood at different rates under different conditions), it can still return to its initial shape, effectively preventing the closure aid 21 from wrinkling after implantation, which could lead to adverse conditions such as thrombosis and calcification.

[0050] In this embodiment, the width of the adjustment frame 22 does not exceed 35mm. The setting value of the width of the adjustment frame 22 can be determined according to the size of the ventricles of different patients, but it usually does not exceed 35mm (refer to the values ​​of a large number of existing cardiac physiological and anatomical measurements). This is because if the width of the adjustment frame 22 is too wide during ventricular contraction, the adjustment frame 22 will collide with the myocardium in the ventricle, causing damage to the myocardium.

[0051] In this embodiment, the first adjustment unit 221, the second adjustment unit 222, and the third adjustment unit 223 are configured with an arc-shaped structure, such as... Figure 2 As shown.

[0052] In this embodiment, the first connecting segment 224 and the second connecting segment 225 are configured with a wavy or "Z" shaped structure, such as... Figure 2 As shown.

[0053] In this embodiment, the limiting mechanism 4 includes a first limiting member 41, a second limiting member 42, and a third limiting member 43, wherein the other ends of the first limiting member 41, the second limiting member 42, and the third limiting member 43 are respectively connected to the first adjusting unit 221, the second adjusting unit 222, and the third adjusting unit 223, as shown below. Figure 1 and Figure 2 As shown.

[0054] In this embodiment, the length of the first limiting member 41 is greater than the length of the third limiting member 43, and the length of the third limiting member 43 is greater than the length of the second limiting member 42.

[0055] In this embodiment, the first limiting member 41, the second limiting member 42, and the third limiting member 43 are merged into one at the proximal portion and fixed to the papillary muscle, as shown below. Figure 4 As shown.

[0056] In this embodiment, the proximal portion of the closure aid 21 is provided with a shape-retaining rod 5, which is arranged in an arched structure, such as... Figure 1 and Figure 3 As shown.

[0057] In this embodiment, the left and right sides of the proximal portion of the closure aid 21 are provided with curling structures 6, which cause the left and right sides of the proximal portion of the closure aid 21 to curl backward, such as... Figure 2 and Figure 3 As shown.

[0058] In this embodiment, the curling structure 6 is disposed on the left and right sides of the connection between the closure aid 21 and the fixed bracket 1.

[0059] In this embodiment, the closure aid 21 is made of biological pericardial material, such as porcine pericardium or bovine pericardium.

[0060] An exemplary procedure for repairing the mitral valve using an implant for repairing heart valves, as described in Embodiment 1, is as follows: 1. The delivery system is accessed via the transapical approach, passing through the patient's left ventricle to reach the left atrium; 2. Position the fixation bracket 1 at the autologous valve annulus, and further release the fixation member 3 so that the fixation member 3 passes through the fixation bracket 1 and inserts into the autologous valve annulus tissue to achieve anchoring; 3. The closure aid 21 is gradually released from the distal end to the proximal end, and the proximal end of the restraint mechanism 4 is fixed to the papillary muscle. At this time, the anti-reflux prosthesis 2 replaces the function of a single autologous flap and achieves closure with the adjacent autologous flap leaflets. 4. Remove the delivery device for the implanted heart valve repair to complete the surgery.

[0061] The foregoing description of the embodiments described above is provided for illustrative purposes. This foregoing description is not intended to be exhaustive, nor is it intended to limit the application to the precise configurations, constructions, and / or steps disclosed. Clearly, many modifications and variations can be made in light of the teachings above. The scope of the invention and all its equivalents are intended to be defined by the appended claims.

Claims

1. An implant for repairing a heart valve, comprising a fixation stent, an anti-reflux prosthesis, a fixation element, and a restraining mechanism, wherein the anti-reflux prosthesis is anchored to atrial tissue via the fixation element, and one end of the restraining mechanism is fixed to ventricular tissue or apical tissue, characterized in that: The anti-reflux prosthesis includes an apoplexy component and an adjustment frame. The adjustment frame includes multiple adjustment units and connecting segments connecting the adjustment units. The other end of the limiting mechanism is connected to each of the adjustment units. The adjustment frame includes a first adjustment unit, a second adjustment unit, a third adjustment unit, a first connecting segment, and a second connecting segment. The second and third adjustment units are respectively located on the left and right sides of the first adjustment unit. The first adjustment unit is connected to the second and third adjustment units via the first and second connecting segments, respectively. The first and second connecting segments are flexible segments, and when the apoplexy component is subjected to blood flow impact, the first and second connecting segments deform in accordance with the impact of the blood flow.

2. The implant for repairing heart valves according to claim 1, characterized in that: The stiffness of the first adjustment unit, the second adjustment unit, and the third adjustment unit must be greater than the stiffness of the first connecting segment and the second connecting segment.

3. An implant for repairing heart valves according to claim 2, characterized in that: The first connecting segment and the second connecting segment are elastic, so that the first connecting segment and the second connecting segment can recover to a preset shape after being deformed by blood flow impact.

4. An implant for repairing heart valves according to claim 1, characterized in that: The width of the adjustable frame shall not exceed 35mm.

5. An implant for repairing heart valves according to claim 1, characterized in that: The first adjustment unit, the second adjustment unit, and the third adjustment unit are configured with an arc-shaped structure.

6. An implant for repairing heart valves according to claim 1, characterized in that: The first connecting segment and the second connecting segment are configured with a wavy or "Z" shaped structure.

7. An implant for repairing heart valves according to claim 1, characterized in that: The limiting mechanism includes a first limiting member, a second limiting member, and a third limiting member, wherein the other ends of the first limiting member, the second limiting member, and the third limiting member are respectively connected to the first adjusting unit, the second adjusting unit, and the third adjusting unit.

8. An implant for repairing heart valves according to claim 7, characterized in that: The length of the first limiting member is greater than the length of the third limiting member, and the length of the third limiting member is greater than the length of the second limiting member.

9. An implant for repairing heart valves according to claim 7, characterized in that: The proximal portion of the closure aid is provided with a shape retention rod, which is arranged in an arched structure.

10. An implant for repairing heart valves according to claim 1, characterized in that: The proximal portion of the closure aid has a curled structure on both sides, which causes the proximal portion of the closure aid to curl backward.

11. An implant for repairing heart valves according to claim 10, characterized in that: The curled structure is located on the left and right sides of the connection between the closure aid and the fixed bracket.

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