Cancer Peptide Therapeutics Targeting caPCNA Isoforms

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Solution Overview

Problem

Current therapies lack effective methods to selectively inhibit cancer cell proliferation by targeting cancer-specific isoforms of proliferating cell nuclear antigen (PCNA), which are unique to malignant cells, without affecting non-malignant cells.

Innovation Solution

Development of peptide variants derived from specific regions of cancer-specific PCNA that interfere with the interaction of cellular proteins with PCNA, including amino acid substitutions and cell-permeable sequences to enhance cytotoxic effects, which can be administered alone or in combination with chemotherapeutic agents.

Engineering Contradictions & Design Principles

VSEngineering Contradiction Analysis

1Reliability

If conventional chemotherapeutic agents are used to inhibit cancer cell proliferation, then cancer cell growth is suppressed, but non-malignant cells are also affected causing harmful side effects

Engineering Contradiction:
Improveselectivity of cancer cell inhibitionVSAvoidimpact on non-malignant cells
Core Design Contradiction:
ReliabilityVSObject-affected harmful factors

Solution Approach 1:

The patent applies local quality by designing peptides with specific amino acid sequences that recognize and bind only to the cancer-specific PCNA isoform (caPCNA) with altered isoelectric point, while leaving non-malignant PCNA unaffected. This localized specificity at the molecular level enables selective cancer cell inhibition without harming normal cells

Inventive Principle:
Principle #3Local quality

Solution Approach 2:

The invention exploits parameter changes in the PCNA protein's isoelectric point between cancer and non-malignant forms. By targeting this specific biophysical parameter difference, the peptides achieve selective binding to caPCNA, resolving the contradiction between effective cancer cell suppression and preservation of non-malignant cell function

Inventive Principle:
Principle #35Parameter changes

2Reliability

If peptide variants with enhanced cytotoxic effects are developed, then cancer cell proliferation is more effectively inhibited, but the complexity of peptide design and manufacturing increases

Engineering Contradiction:
Improveefficacy of cancer cell inhibitionVSAvoidpeptide structure complexity
Core Design Contradiction:
ReliabilityVSDevice complexity

Solution Approach 1:

The patent segments the complex problem of cancer cell inhibition into modular peptide components with specific amino acid sequences. Each peptide variant is designed as a discrete molecular entity targeting a specific region of PCNA, simplifying the design process while maintaining high efficacy through combinatorial optimization of sequence variants

Inventive Principle:
Principle #1Segmentation

Solution Approach 2:

The invention uses copying by creating multiple peptide variants based on a core sequence template that targets caPCNA. These variants are generated through systematic amino acid substitutions and modifications, allowing efficient exploration of structure-activity relationships without requiring de novo design of each peptide

Inventive Principle:
Principle #26Copying

Data Source

PatentEP2913057B1Cancer peptide therapeutics
Publication Date: 2017.09.06 INDIANA UNIVERSITY RESEARCH & TECHNOLOGY CORP
  • EP2913057B1 patent drawingFigure 1
  • EP2913057B1 patent drawingFigure 2A~2B
  • EP2913057B1 patent drawingFigure 3

AI summary

Peptide variants corresponding to cancer specific isoform of proliferating cell nuclear antigen (caPCNA) interfere with intracellular protein-protein interaction, thereby causing a reduction in the proliferative potential of cancer. Cell-permeable caPCNA-derived peptides and their variants serve as therapeutic compositions to reduce the proliferation of cancer cells and also augment cytotoxic effects of existing cancer therapy. These compositions are also useful for chemoprevention of cancer.