CTRP3 Therapeutic Agent for Autoimmune Disease Treatment

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Solution Overview

Problem

The physiological roles of Clq/TNF-related protein 3 (CTRP3) in vivo are not well understood, and its role in autoimmune diseases such as rheumatoid arthritis (RA) is unclear, necessitating a novel therapeutic approach for these conditions.

Innovation Solution

Administration of CTRP3 to autoimmune disease models, including rheumatoid arthritis and multiple sclerosis, using CTRP3 knockout mice to study its effects and potential as a treatment agent, demonstrating its ability to reduce the incidence and severity of autoimmune arthritis.

Engineering Contradictions & Design Principles

VSEngineering Contradiction Analysis

1Reliability

If existing treatments (antibodies and inhibitors against inflammatory cytokines) are used for rheumatoid arthritis, then treatment effectiveness is improved for most patients, but treatment becomes ineffective for some patients or during the course of treatment

Engineering Contradiction:
Improvetreatment effectivenessVSAvoidapplicability to all patients
Core Design Contradiction:
ReliabilityVSAdaptability or versatility

Solution Approach 1:

The patent introduces CTRP3 as a novel therapeutic agent that acts as an intermediary substance to treat rheumatoid arthritis through a different mechanism than existing cytokine-targeted therapies. CTRP3 binds to C1q and modulates immune responses, providing an alternative pathway that bypasses the limitations of current treatments and achieves therapeutic effects in patients who do not respond to conventional therapies.

Inventive Principle:
Principle #24Intermediary (Mediator)

2Reliability

If CTRP3 is administered to treat autoimmune diseases, then the incidence and severity of autoimmune arthritis is reduced, but the physiological roles of CTRP3 in vivo were previously unclear

Engineering Contradiction:
Improvetherapeutic effectVSAvoidunderstanding of physiological roles
Core Design Contradiction:
ReliabilityVSLoss of information

Solution Approach 1:

The patent performs preliminary actions by first identifying and characterizing CTRP3's therapeutic effects through knockout mouse models and in vitro experiments, demonstrating its ability to reduce autoimmune arthritis incidence and severity. This preliminary characterization of therapeutic efficacy precedes and guides further investigation into its precise physiological mechanisms, allowing the therapeutic application to advance while physiological understanding continues to develop.

Inventive Principle:
Principle #10Preliminary action

Data Source

PatentEP3069726B1Agent for use in a method of treating autoimmune disease
Publication Date: 2021.01.27 TOKYO UNIVERSITY OF SCIENCE
  • EP3069726B1 patent drawingFigure 1A~1D
  • EP3069726B1 patent drawingFigure 2A~2D
  • EP3069726B1 patent drawingFigure 3A~3E

AI summary

Provided is an agent for the treatment of an autoimmune disease, the agent comprising CTRP3, and a method for the treatment of an autoimmune disease, the method comprising: administering an agent, which comprises CTRP3 for the treatment of an autoimmune disease, to a subject in need of a treatment of an autoimmune disease.