Dexpramipexole Targets Eosinophils to Reduce Plasma Cell Levels

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Solution Overview

Problem

Current treatments for plasma cell disorders and B-cell disorders, such as multiple myeloma and lymphomas, face challenges in effectively reducing elevated plasma cell and B-cell levels due to drug resistance mechanisms and the supportive role of eosinophils and basophils in cell survival and proliferation.

Innovation Solution

Dexpramipexole, a synthetic aminobenzothiazole derivative, is administered therapeutically to decrease plasma cell and B-cell levels by targeting eosinophils and basophils, potentially overcoming cell adhesion-mediated drug resistance and promoting apoptosis.

Engineering Contradictions & Design Principles

VSEngineering Contradiction Analysis

1Reliability

If current treatments are used to reduce plasma cell and B-cell levels, then cell levels may be reduced, but drug resistance mechanisms develop and treatment effectiveness decreases

Engineering Contradiction:
Improvetreatment effectivenessVSAvoiddrug resistance
Core Design Contradiction:
ReliabilityVSObject-affected harmful factors

Solution Approach 1:

The patent extracts and targets the specific cellular mechanism (eosinophils and basophils) that supports plasma cell and B-cell survival. By removing this supportive element through dexpramipexole treatment, the patent addresses drug resistance by attacking the disease through a different pathway rather than directly targeting the resistant plasma cells themselves.

Inventive Principle:
Principle #2Taking out (Extraction)

Solution Approach 2:

The patent uses dexpramipexole as an intermediary agent that indirectly reduces plasma cell and B-cell levels by targeting eosinophils and basophils. This intermediary approach bypasses direct drug resistance mechanisms that plasma cells may have developed against conventional therapies.

Inventive Principle:
Principle #24Intermediary (Mediator)

2Reliability

If conventional therapies are administered to decrease plasma cell levels, then cell proliferation may be inhibited, but adverse effects increase due to lack of selectivity

Engineering Contradiction:
Improvecell level reductionVSAvoidadverse effects
Core Design Contradiction:
ReliabilityVSObject-generated harmful factors

Solution Approach 1:

The patent applies local quality by targeting a specific cell type (eosinophils and basophils) with a specific drug mechanism (dexpramipexole) rather than using broad-spectrum immunosuppressants. This localized approach reduces adverse effects by sparing other healthy cell populations from unnecessary suppression.

Inventive Principle:
Principle #3Local quality

Solution Approach 2:

The patent changes the therapeutic parameter from direct plasma cell targeting to eosinophil/basophil targeting. This parameter change allows for more selective treatment that reduces adverse effects while maintaining effectiveness in decreasing plasma cell and B-cell levels.

Inventive Principle:
Principle #35Parameter changes

3Reliability

If treatment duration is extended to overcome drug resistance, then treatment effectiveness may improve, but treatment complexity and patient burden increase

Engineering Contradiction:
Improvetreatment effectivenessVSAvoidtreatment regimen complexity
Core Design Contradiction:
ReliabilityVSDevice complexity

Solution Approach 1:

Instead of extending treatment duration to overcome resistance, the patent inverts the approach by using a completely different mechanism of action (dexpramipexole targeting eosinophils/basophils) that may work more quickly and effectively, potentially shortening the treatment course required.

Inventive Principle:
Principle #13The other way round (Inversion)

Data Source

PatentEP3038467B1Compositions and methods for treating plasma cell disorders and b-cell prolymphocytic disorders
Publication Date: 2020.07.29 KNOPP BIOSCIENCES LLC
  • EP3038467B1 patent drawingFigure 1
  • EP3038467B1 patent drawingFigure 2A~2B
  • EP3038467B1 patent drawingFigure 3A~3B

AI summary

Disclosed herein are methods of treating conditions, which may be associated with elevated levels of plasma cells and/or B-cells, with a therapeutically effective amount of dexpramipexole or pharmaceutical acceptable salt thereof.