GPCR Agonist Activation Prediction via Minimum Free Energy Paths

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Solution Overview

Problem

Current technologies struggle to predict the activation potency of agonist molecules on G Protein-Coupled Receptors (GPCRs) efficiently and accurately, lacking an algorithmic framework to reveal the activation mechanism and specificity, which is crucial for rational drug design.

Innovation Solution

A method and system utilizing global molecular docking, enhanced sampling algorithms, and free energy calculations to determine the minimum free energy path of agonist activation on GPCRs, including blind speculation of complex structures and local structural optimization, without requiring prior information about the ligand structure.

Engineering Contradictions & Design Principles

VSEngineering Contradiction Analysis

1Measurement precision

If functional assays are used to determine activation, then activation occurrence can be detected, but detailed mechanistic information and activation potency prediction are not obtained

Engineering Contradiction:
Improveactivation potency predictionVSAvoidmechanistic information
Core Design Contradiction:
Measurement precisionVSLoss of information

Solution Approach 1:

The patent introduces computational modeling and simulation as an intermediary between functional assays and structural biology. The system uses molecular dynamics simulations, free energy calculations, and machine learning models to predict activation potency and reveal mechanistic details that neither functional assays nor static structural data can provide alone.

Inventive Principle:
Principle #24Intermediary (Mediator)

Solution Approach 2:

The patent replaces wet-lab functional assays with in-silico computational predictions for activation potency assessment. The system uses physics-based molecular simulations and data-driven machine learning models to substitute experimental measurement, enabling detailed mechanistic insights without the limitations of traditional functional assays.

Inventive Principle:
Principle #28Mechanics substitution (Replace mechanical system)

2Measurement precision

If structural biology methods are used, then atomic-level details of GPCR states are obtained, but activation difficulty level and mechanistic origin are not indicated

Engineering Contradiction:
Improveatomic-level structural detailsVSAvoidactivation mechanism and difficulty level
Core Design Contradiction:
Measurement precisionVSLoss of information

Solution Approach 1:

The patent transforms static structural biology data into dynamic information through molecular dynamics simulations. The system analyzes conformational transitions, energy landscapes, and temporal evolution of GPCR states to extract mechanistic insights and activation difficulty levels that cannot be obtained from static atomic structures alone.

Inventive Principle:
Principle #15Dynamics

Solution Approach 2:

The patent extends structural parameters by calculating free energy differences, activation barriers, and conformational transition probabilities. The system transforms spatial coordinates into thermodynamic and kinetic parameters that quantify activation mechanism and difficulty, providing a comprehensive view beyond atomic positions.

Inventive Principle:
Principle #35Parameter changes

3Measurement precision

If site-directed mutagenesis is performed to identify key residues, then interaction details are obtained, but the process is excessively costly and cumbersome

Engineering Contradiction:
Improvekey residue identificationVSAvoidresearch efficiency
Core Design Contradiction:
Measurement precisionVSProductivity

Solution Approach 1:

The patent performs preliminary computational screening to identify key residues and interaction hotspots before any experimental work. The system uses molecular docking, interaction energy calculations, and machine learning predictions to pre-identify critical residues, guiding subsequent experiments and reducing the need for exhaustive site-directed mutagenesis.

Inventive Principle:
Principle #10Preliminary action

Solution Approach 2:

The patent creates computational models and simulations as virtual copies of the GPCR-ligand system. These in-silico models replicate molecular interactions, enabling virtual screening and analysis of residue importance without physical manipulation, thereby replacing costly and time-consuming mutagenesis experiments.

Inventive Principle:
Principle #26Copying

4Measurement precision

If current CADD technologies are used, then ligand binding strength can be calculated, but the activation process and receptor conformational changes are not involved

Engineering Contradiction:
Improvebinding strength calculationVSAvoidactivation process and conformational changes
Core Design Contradiction:
Measurement precisionVSLoss of information

Solution Approach 1:

The patent extends static binding affinity calculations to dynamic activation process simulations. The system employs molecular dynamics to model receptor conformational transitions from inactive to active states, capturing the temporal and spatial evolution of binding and activation that traditional CADD methods miss.

Inventive Principle:
Principle #15Dynamics

Solution Approach 2:

The patent creates a unified computational framework that simultaneously calculates binding strength, predicts activation potency, and models conformational changes. The system integrates multiple functions (docking, free energy calculations, dynamics simulation, machine learning) into a single platform that provides comprehensive activation analysis beyond binding affinity alone.

Inventive Principle:
Principle #6Universality (Multi-functionality)

Data Source

PatentEP4730337A1Method and system for predicting GPCR activation ability of agonist molecules
Publication Date: 2026.04.22 THE CHINESE UNIV OF HONG KONG (SHENZHEN)
  • EP4730337A1 patent drawingFigure 1
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  • EP4730337A1 patent drawingFigure 3

AI summary

Provided are a method and system for predicting an activation potency of agonist molecules on G Protein-Coupled Receptors (GPCRs). The method includes: blindly speculating complex structures formed by binding of a ligand to an activated receptor structure and an inactivated receptor structure, respectively, via global molecular docking; extracting an initial path enabling an inactivated complex structure to be activated to an activated complex structure based on an enhanced sampling algorithm; searching for a minimum free energy path closest to the initial path by applying an automatic path optimization algorithm; calculating a free energy distribution curve along the minimum free energy path by employing umbrella sampling and determining an energy barrier height and a free energy difference before and after activation, thereby determining the activation potency of the ligand structure on the GPCRs.