Human Milk Oligosaccharides Inhibit Coronavirus Binding

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Solution Overview

Problem

Current treatments lack effective solutions for preventing or attenuating coronavirus-induced inflammation, particularly in the respiratory system and gastrointestinal tract, where coronaviruses like SARS-CoV-2 bind to the ACE2 receptor, leading to severe inflammation and immune responses.

Innovation Solution

Compositions of human milk oligosaccharides (hMOS), such as 2′-fucosyllactose, are used to prevent viral binding, promote gut commensal bacteria growth, and directly reduce inflammation by administering them alone or in combination with other agents to the respiratory and gastrointestinal systems.

Engineering Contradictions & Design Principles

VSEngineering Contradiction Analysis

1Object-affected harmful factors

If conventional treatments are used for coronavirus infection, then viral infection can be addressed, but host inflammation is not effectively prevented or attenuated

Engineering Contradiction:
Improvehost inflammationVSAvoideffectiveness of treatment
Core Design Contradiction:
Object-affected harmful factorsVSReliability

Solution Approach 1:

Human milk oligosaccharides act as intermediary molecules that bind to coronaviruses in the gut, preventing direct interaction between the virus and host ACE2 receptors. This intermediary binding mechanism effectively blocks viral attachment and entry into host cells, thereby preventing host inflammation without requiring direct antiviral intervention

Inventive Principle:
Principle #24Intermediary (Mediator)

Solution Approach 2:

The patent extracts and utilizes specific functional components (human milk oligosaccharides) from human milk to isolate and amplify the protective anti-inflammatory effects. These extracted oligosaccharides are then administered as concentrated therapeutic compositions to specifically target and prevent coronavirus-induced inflammation

Inventive Principle:
Principle #2Taking out (Extraction)

2Object-affected harmful factors

If human milk oligosaccharides are administered to prevent viral binding, then host inflammation is reduced, but the mechanism requires specific oligosaccharide structures

Engineering Contradiction:
Improveviral bindingVSAvoidoligosaccharide structure specificity
Core Design Contradiction:
Object-affected harmful factorsVSDevice complexity

Solution Approach 1:

The patent identifies and utilizes specific structural parameters of human milk oligosaccharides (such as fucosylated and sialylated structures) that are critical for their protective function. By focusing on these specific structural parameters and producing oligosaccharides with defined configurations, the patent achieves effective viral binding prevention while managing structural complexity through targeted synthesis or selection

Inventive Principle:
Principle #35Parameter changes

Applied Scientific Principles

This section explains which scientific principles are used to turn an abstract innovation direction into a practical engineering solution.

Function Achieved in This Case

The hMOS compositions effectively inhibit coronavirus binding, reduce inflammation, and promote recovery from pathogen-induced immune responses, offering a natural and effective treatment for coronavirus-induced inflammation.

Implementation Method 1

preventing viruses, or other respiratory pathogens, from binding to host cells or tissue

Methodology Applied
Scientific EffectViral binding inhibition: Absorption (physical)

Implementation Method 2

reducing host inflammation by, e.g., promoting growth and/or functions of common gut commensal bacteria

Methodology Applied
Scientific EffectPrebiotic effect: Fermentation

Data Source

PatentUS11324766B22'-fucosyllactose for the prevention and treatment of coronavirus-induced inflammation
Publication Date: 2022.05.10 GLYCOSYN LLC

AI summary

The invention provides compositions and methods for utilizing oligosaccharides, such as isolated human milk oligosaccharides, to attenuate a respiratory pathogen infection-induced host inflammation and/or to promote recovery from a respiratory pathogen infection-induced host inflammation in a subject.