Immunogenic Peptides for Tumour Control via CD4+ T Cell Activation

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Solution Overview

Problem

Current immunotherapy approaches for cancer treatment, particularly targeting tumour-specific antigens, have limited success and high relapse rates, with a focus on CD8+ T cells and MHC class I presentation, while the potential of CD4+ T cells through MHC class II presentation has been underexplored due to the belief that most tumours do not express MHC class II determinants and CD4+ T cells are not potent anti-tumour cells.

Innovation Solution

The use of isolated immunogenic peptides comprising a T-cell epitope derived from a tumour-associated antigen combined with a [CST]-(X)2-[CST] motif, specifically a C-(X)2-[CST] or [CST]-(X)2-C motif, to induce CD4+ regulatory T cells that are cytotoxic to cells presenting the tumour-associated antigen, potentially overcoming the limitations of existing therapies by targeting MHC class II presentation.

Engineering Contradictions & Design Principles

VSEngineering Contradiction Analysis

1Reliability

If immunotherapy focuses on CD8+ T cells and MHC class I presentation, then tumour-specific antigen recognition is achieved, but treatment success is limited and relapse rates are high

Engineering Contradiction:
Improvetreatment success rateVSAvoidrelapse rate
Core Design Contradiction:
ReliabilityVSProductivity

Solution Approach 1:

The patent segments the immunotherapy approach by dividing the traditional single-pathway (CD8+ T cells only) into multiple pathways: activating both CD8+ T cells through MHC class I and CD4+ T cells through MHC class II presentation. This segmentation allows simultaneous engagement of different immune cell types to achieve more reliable tumour elimination and reduce relapse

Inventive Principle:
Principle #1Segmentation

Solution Approach 2:

The patent merges two previously separate immunotherapy pathways into a unified approach: combining MHC class I-restricted CD8+ T cell responses with MHC class II-restricted CD4+ T cell responses. This combination therapy integrates the cytolytic activity of CD8+ T cells with the helper and regulatory functions of CD4+ T cells, improving overall treatment efficacy and reducing relapse

Inventive Principle:
Principle #5Merging (Combining)

2Adaptability or versatility

If CD4+ T cells are targeted through MHC class II presentation, then new anti-tumour mechanisms are activated, but this approach has been underexplored due to beliefs about tumour MHC class II expression

Engineering Contradiction:
Improveimmunotherapy strategy diversityVSAvoidestablished therapy effectiveness
Core Design Contradiction:
Adaptability or versatilityVSReliability

Solution Approach 1:

The patent inverts the conventional assumption that tumours do not express MHC class II molecules. By designing peptides that specifically target MHC class II presentation and demonstrating their efficacy, the patent challenges and reverses the established belief, opening new avenues for immunotherapy that were previously considered invalid

Inventive Principle:
Principle #13The other way round (Inversion)

Solution Approach 2:

The patent changes the key parameter of MHC class expression consideration in tumour immunotherapy. Instead of assuming MHC class II absence or low expression, the patent designs and tests peptides optimized for MHC class II binding and presentation, thereby changing the fundamental parameter of antigen presentation pathway utilization and demonstrating enhanced anti-tumour immunity

Inventive Principle:
Principle #35Parameter changes

Data Source

PatentUS10617748B2Immunogenic control of tumours and tumour cells
Publication Date: 2020.04.14 IMCYSE SA

AI summary

The present invention relates to the use of immunogenic peptides comprising a T-cell epitope derived from a tumor-associated antigen and a redox motif such as C-(X)2-[CST] or [CST]-(X)2-C in the treatment of a tumor or in the treatment or prevention of a tumor relapse, and in the manufacture of medicaments therefore.