Logic-Gated CAR Construct Reduces Off-Tumor Toxicity

Resolve Bottlenecks,
Find Innovative Solutions
Generate Solutions

Solution Overview

Problem

Current immunotherapeutic agents, including chimeric antigen receptors (CARs), often result in 'on-target off-tumor' toxicity due to targeting single antigens, leading to damage of normal tissues and reduced specificity in cancer treatment.

Innovation Solution

Development of 'logic-gated' CAR pairs that modulate expression based on specific patterns of antigen presence, allowing T cells to trigger only when both target antigens are present, using a nucleic acid construct encoding activating and inhibitory CARs with distinct spacer domains and retention signals to achieve precise targeting.

Engineering Contradictions & Design Principles

VSEngineering Contradiction Analysis

1Device complexity

If single-antigen CAR targeting is used, then treatment simplicity is maintained, but on-target off-tumor toxicity increases

Engineering Contradiction:
ImproveCAR structure complexityVSAvoidoff-tumor toxicity
Core Design Contradiction:
Device complexityVSObject-affected harmful factors

Solution Approach 1:

The patent divides the targeting function into multiple independent CAR components, each recognizing a different antigen. Instead of using a single CAR that targets one antigen, the invention employs multiple CARs (e.g., CAR1 targeting antigen1, CAR2 targeting antigen2) that work together through logical gating mechanisms. This segmentation allows the system to distinguish tumor cells (expressing multiple antigens) from normal cells (expressing only one antigen), thereby reducing off-tumor toxicity while maintaining manageable complexity through modular design.

Inventive Principle:
Principle #1Segmentation

2Object-affected harmful factors

If multiple CARs are co-expressed to improve targeting specificity, then off-tumor toxicity is reduced, but expression control complexity increases

Engineering Contradiction:
Improveoff-tumor toxicityVSAvoidexpression control complexity
Core Design Contradiction:
Object-affected harmful factorsVSDevice complexity

Solution Approach 1:

The patent merges multiple CAR expression control functions into a unified logical gating system. By implementing AND-gate or OR-gate logic where multiple CARs are co-expressed and integrated through shared signaling pathways, the system achieves coordinated control of multiple antigens. This merging approach allows complex multi-antigen targeting to be managed through integrated logical operations rather than independent control mechanisms, reducing overall system complexity while maintaining high specificity.

Inventive Principle:
Principle #5Merging (Combining)

3Measurement precision

If logic-gated CAR pairs with distinct spacers are used, then targeting precision is improved, but CAR structure complexity increases

Engineering Contradiction:
Improvetargeting precisionVSAvoidCAR structure complexity
Core Design Contradiction:
Measurement precisionVSDevice complexity

Solution Approach 1:

The patent applies local quality by introducing spacers with distinct properties (different lengths, compositions, or functional characteristics) at specific locations within the CAR structure. These localized variations in spacer properties enable differential control of CAR conformation, stability, and signaling activation. For example, shorter spacers may promote tight clustering for AND-gate activation, while longer spacers may allow independent activation for OR-gate logic. This localized differentiation achieves high targeting precision without requiring complete redesign of the entire CAR structure, maintaining modularity and managing complexity.

Inventive Principle:
Principle #3Local quality

Data Source

PatentEP3288970B1Nucleic acid construct for expressing more than one chimeric antigen receptor
Publication Date: 2020.02.19 AUTOLUS LIMIED
  • EP3288970B1 patent drawingFigure 1(a)~1(d)
  • EP3288970B1 patent drawingFigure 2
  • EP3288970B1 patent drawingFigure 3~4

AI summary

The present invention provides a nucleic acid construct comprising the following structure: A – X – B in which X is a nucleic acid sequence which encodes a cleavage site; and A and B are nucleic acid sequences encoding a first and a second chimeric antigen receptor (CAR), each CAR comprising: (i) an antigen-binding domain; (ii) a spacer (iii) a trans-membrane domain; and (iv) an endodomain wherein the antigen binding domains of the first and second CARs bind to different antigens, wherein the spacer of the first CAR is different to the spacer of the second CAR and wherein one of the first or second CARs is an activating CAR comprising an activating endodomain and the other CAR is an inhibitory CAR comprising a ligation-off inhibitory endodomain; and wherein: (a) the first and/or second CAR comprises an intracellular retention signal; and/or (b) the signal peptide of the first or second CAR comprises one or more mutation(s) such that it has fewer hydrophobic amino acids.