Modified Peptide Ligands for Autoimmune Disorder Treatment

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Solution Overview

Problem

Current therapies for Multiple Sclerosis (MS) like interferons and glatiramer acetate have limited effectiveness and significant side effects, and there is a need for improved immunotherapeutic approaches that can effectively modulate the immune response to prevent disease progression.

Innovation Solution

Development of altered peptide ligands and conjugates, specifically modified versions of the MBP 83-99 peptide, which are either in linear or cyclic forms and conjugated with mannan, to divert immune responses from a Th1 to a Th2 profile, reducing inflammation and preventing autoimmune damage in MS.

Engineering Contradictions & Design Principles

VSEngineering Contradiction Analysis

1Reliability

If current peptide therapies (interferons and glatiramer acetate) are used for MS treatment, then immune response modulation is achieved, but side effects increase and long-term effectiveness decreases

Engineering Contradiction:
Improvelong-term effectivenessVSAvoidside effects
Core Design Contradiction:
ReliabilityVSObject-affected harmful factors

Solution Approach 1:

The patent modifies the MBP peptide sequence by substituting specific amino acids (e.g., position 91 in the MBP 83-99 epitope) to create altered peptide ligands that maintain immunogenicity while reducing cross-reactivity with native MBP, thereby improving long-term effectiveness and reducing side effects

Inventive Principle:
Principle #35Parameter changes

Solution Approach 2:

The patent conjugates altered peptide ligands to carrier proteins (keyhole limpet hemocyanin, bovine serum albumin, or rat serum albumin) to create composite immunogenic compositions that enhance stability, immunogenicity, and therapeutic effectiveness while reducing adverse reactions

Inventive Principle:
Principle #40Composite materials

2Reliability

If orally administered antigens are used to suppress autoimmunity, then regulatory T-cell induction is improved, but peptide stability and cost-effectiveness deteriorate

Engineering Contradiction:
Improveantigen-specific regulatory T-cell inductionVSAvoidpeptide stability
Core Design Contradiction:
ReliabilityVSStability of the object's composition

Solution Approach 1:

The patent introduces non-natural amino acid substitutions in the peptide sequence to enhance resistance to proteolytic degradation, thereby improving oral stability while maintaining the ability to induce regulatory T-cells

Inventive Principle:
Principle #35Parameter changes

Solution Approach 2:

The patent forms conjugates with carrier proteins to protect the peptide from degradation in the gastrointestinal tract, enabling stable oral administration and improving cost-effectiveness through enhanced bioavailability

Inventive Principle:
Principle #40Composite materials

3Reliability

If peptide analogues are designed to cross-react with native MBP for therapeutic effect, then immune suppression is achieved, but off-target effects and harmful factors increase

Engineering Contradiction:
Improveimmune suppression efficacyVSAvoidoff-target effects
Core Design Contradiction:
ReliabilityVSObject-generated harmful factors

Solution Approach 1:

The patent introduces subtle local modifications at specific positions (e.g., position 91) in the peptide sequence that preserve binding to the target antigen-presenting cells while eliminating cross-reactivity with native MBP, thereby reducing off-target effects

Inventive Principle:
Principle #3Local quality

Solution Approach 2:

The patent creates peptide analogues that copy the essential immunogenic features of native MBP epitopes while introducing modifications to prevent harmful cross-reactivity, achieving selective immune suppression

Inventive Principle:
Principle #26Copying

Data Source

PatentEP2227486B1Peptide analogues and conjugates thereof
Publication Date: 2017.03.08 VIANEX SA
  • EP2227486B1 patent drawingFigure 1
  • EP2227486B1 patent drawingFigure 2
  • EP2227486B1 patent drawingFigure 3

AI summary

One aspect of the invention relates to a peptide comprising the amino acid sequence of formula (I), ENPVVHFFK91NIVTP96RTP (I) wherein at least one of K91 and P96 is substituted by a natural or unnatural amino acid, wherein said peptide is in linear or cyclic form. Another aspect of the invention relates to a peptide comprising the amino acid sequence of formula (Ia), ENPWHFFK91NrVTP96RTP (Ia), wherein each of K91 and P96 is substituted by a natural or unnatural amino acid. Another aspect of the invention relates to a peptide comprising the amino acid sequence of formula (Ib), ENPVVHFFK91NIVTP96RTP (Ib) wherein at least one of K91 and P96 is substituted by an amino acid selected from R, E, F and Y. Yet another aspect of the invention relates to a peptide comprising the amino acid sequence of formula (II), VHFFK91NrVTP96RTP (II) wherein K91 is substituted by the amino acid A and P96 is substituted by the amino acid A, wherein said peptide is in linear or cyclic form. Further aspects of the invention relate to conjugates of the above described peptides with mannan, pharmaceutical compositions and methods of treating autoimmune disorders.