CD33-targeted chimeric antigen receptor-modified t cells for treating cd33-positive malignancies

By using CD33-targeted chimeric antigen receptor-modified T cells, the low cure rate of AML treatment has been solved, achieving specific killing and therapeutic effects on CD33-positive cells, significantly reducing tumor burden and prolonging survival time.

CN114040978BActive Publication Date: 2025-10-17CITY OF HOPE
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Patent Information

Application Number
CN202080047912.8
Authority / Receiving Office
CN · China
Patent Type
Patents(China)
Current Assignee / Owner
Priority Date
2019-05-31
Filing Date
2020-06-01
Publication Date
2025-10-17
Estimated Expiration
2040-06-01

AI Technical Summary

Technical Problem

In the current technology, the cure rate of acute myeloid leukemia (AML) is low, especially for patients with relapsed or refractory AML, there is a lack of effective treatments, and CD33 positive cells are widely expressed in leukemia and other cancers, but there is a lack of specific treatments.

Method used

T cells modified with CD33-targeting chimeric antigen receptor (CAR) are transduced by nucleic acid molecules encoding specific amino acid sequences and combined with hypomethylating agents such as decitabine to treat CD33-positive cells, including local or systemic administration of CD33-expressing cancerous T cells.

Benefits of technology

It achieves specific killing of CD33-positive leukemia and other cancers, significantly reduces tumor burden and prolongs patient survival time, reduces myeloid-derived suppressor cells, and improves treatment efficacy.

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Abstract

Chimeric antigen receptors targeting CD33 and their use in treating various cancers and reducing myeloid-derived suppressor cells in patients are described.
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