Intraocular tissue disruptor

Abstract

This invention relates to an intraocular tissue disruptor comprising a clamping and fragmenting section, a connecting mechanism, a storage sheath, a control mechanism, and a handle. The connecting mechanism is housed within the storage sheath, with the clamping and fragmenting section located at its distal end and the handle at its proximal end. The control mechanism controls the opening and closing of the clamping and fragmenting section. When the control mechanism closes the clamping and fragmenting section, the connecting mechanism moves the clamping and fragmenting section into the storage sheath, causing the working part of the clamping and fragmenting section to close. Under the combined action of the cutting edge of the storage sheath and the working part, the intraocular tissue is fragmented. When this invention includes a negative pressure suction mechanism, the negative pressure suction mechanism can promptly suction and remove the fragmented tissue from the eye, preventing accidental damage to surrounding intraocular tissues. This invention's intraocular tissue disruptor is very convenient and safe for clinical use.

Description

Technical Field

[0001] This invention relates to an intraocular tissue disruptor for breaking up intraocular tissue, particularly an intraocular tissue disruptor with breaking and suction functions. Background Technology

[0002] A rupture or excessive laxity of the lens suspensory ligaments can cause the lens to completely dislocate from its physiological position, a condition known as total lens dislocation. Total lens dislocation can lead to serious complications such as refractive errors, uveitis, and secondary glaucoma. If the lens dislocates completely into the vitreous cavity, it can also cause traction on the vitreous base, leading to serious complications such as retinal detachment. Therefore, patients with total lens dislocation require evaluation of their visual function, the degree of lens opacity, age, and any related complications; surgical treatment may be necessary in some cases.

[0003] For moderate to severe complete lens dislocation extending into the vitreous cavity, if a hard nucleus is suspected, the current standard surgical method is to remove it via pars plana cycloplegic resection. However, when the lens nucleus is highly rigid, the fragments generated by cycloplegia, under the impact of the fluid flow, may damage the retina, potentially leading to retinal tears, hemorrhage, or even retinal detachment, causing significant harm to vision. Furthermore, extremely hard lens fragments are difficult to remove precisely using cycloplegia. Therefore, further improvements to existing tissue fragmentation instruments are necessary. Summary of the Invention

[0004] The intraocular tissue disruptor of this invention, through the combined design of the clamping disruptor and the storage sheath, not only generates good clamping and disrupting force, but also allows the storage sheath to be connected to a negative pressure suction device to promptly remove the disrupted tissue, achieving precise disruption and timely removal, effectively preventing the fragments generated after tissue disruption from causing accidental damage to the retina or surrounding tissues.

[0005] The intraocular tissue disruptor of the present invention is characterized in that: the intraocular tissue disruptor 100 includes a clamping and disrupting part 1, a connecting mechanism 2, a storage sheath 3, a control mechanism 4, and a handle 5;

[0006] A. The connecting mechanism 2 is disposed inside the storage sheath 3, and the distal end of the connecting mechanism 2 is provided with the clamping and breaking part 1; the proximal end of the connecting mechanism 2 is provided with the handle 5;

[0007] B. The control mechanism 4 controls the opening or closing of the clamping and crushing part 1;

[0008] C. The clamping and crushing part 1 contains at least two working parts 11.

[0009] When the control mechanism 4 controls the clamping and crushing part 1 to close, the connecting mechanism 2 drives the clamping and crushing part 1 to move into the storage sheath 3, and the working part 11 of the clamping and crushing part 1 closes to crush the tissue. In particular, when a cutting edge 32 is provided at the end of the storage sheath 3, the intraocular tissue can be easily crushed under the combined action of the cutting edge 32 and the working part 11.

[0010] The clamping and crushing section 1 includes three working sections 11. The clamping section 1 may have multiple working sections 11, such as three or more, which can better ensure the stability of the tissue clamping and achieve a better crushing effect.

[0011] The working part 11 includes a clamping surface 11-1 and a crushing end 11-2. The clamping surface 11-1 can effectively fix the tissue to be crushed, and the crushing end 11-2 can apply crushing force to the clamped tissue. The crushing end 11-2 can be designed as a linear cutting edge, a serrated cutting edge, or an arc-shaped cutting edge.

[0012] The clamping surface 11-1 is arc-shaped and encircling. The arc-shaped encircling shape can apply better clamping force to tissues, especially semi-rigid or elastic tissues.

[0013] The intraocular tissue disruptor 100 includes a negative pressure suction mechanism 6. The negative pressure suction mechanism 6 can promptly suction and remove the fragmented tissue from the eye, preventing accidental damage to surrounding intraocular tissues caused by the fragmented tissue.

[0014] The negative pressure suction mechanism 6 includes a suction inlet 61, a suction channel 62, and a discharge outlet 63; the suction inlet 61 is located at the distal end of the storage sheath 3, and the discharge outlet 63 is located on the handle 5. The discharge outlet 63 is connected to the negative pressure source 200.

[0015] The control mechanism 4 is a linkage-type control mechanism, a hinge-type control mechanism, or a rotary control mechanism. The applicant has only listed the above three types of control mechanisms. Those skilled in the art can design control mechanisms 4 with different functions as needed, without departing from the protection scope of this application.

[0016] The control mechanism 4 is a linkage control mechanism 4-1; the linkage control mechanism 4-1 includes a control switch 41. When the control switch 41 is pressed, the connecting mechanism 2 moves backward and the clamping crushing part 1 closes. When the control switch 41 is released, the connecting mechanism 2 resets and the clamping crushing part 1 opens.

[0017] The linkage control mechanism 4-1 includes a control switch 41, a control rod 42, a connecting seat 43, and a return spring 44. The control rod 42 is connected to the connecting seat 43, and the return spring 44 is located on the outside of the connecting seat 43. The control rod 42, the connecting seat 43, and the return spring 44 are housed within the housing 51 of the handle 5. The distal end of the housing 51 is provided with a storage sheath 3, and the connecting mechanism 2 is housed within the storage sheath 3. The proximal end of the connecting mechanism 2 is connected to the connecting seat 43. Pressing the control switch 41 causes the control rod 42 to move backward, which in turn causes the connecting seat 43 to move backward. The return spring 44 is compressed, causing the connecting mechanism 2 to move backward. The clamping and crushing part 1 connected to the distal end of the connecting mechanism 2 retracts and enters the storage sheath 3. The clamping and crushing part 1 closes, and the working part 11 and the cutting edge at the distal end of the storage sheath 3 work together to crush the tissue. When the control switch 41 is released, the connecting seat 43 moves forward under the restoring force of the return spring 44. The connecting mechanism 2 connected to the connecting seat 43 drives the clamping and crushing part 1 to move forward. The clamping and crushing part 1 extends out from the distal end of the storage sheath 3 and opens.

[0018] The through-hole 31 of the storage sheath 3 constitutes the attraction channel 62.

[0019] The connecting mechanism 2 is a hollow tubular structure.

[0020] The central hole 21 of the hollow tubular structure of the connecting mechanism 2 constitutes the suction channel 62. The hollow tubular structure design maximizes the suction channel 62, better ensuring that the fragmented tissue can be effectively suctioned out of the eye.

[0021] The negative pressure suction mechanism 6 includes a suction switch 64. The suction switch 64 can control the opening and closing of the negative pressure suction.

[0022] The suction switch 64 is disposed on the handle 5 or as a foot switch. The suction switch 64 may be disposed at various parts of the intraocular tissue disruptor of the present invention as needed. The applicant will not give examples of each of them here, but none of them depart from the protection scope of this application.

[0023] In clinical use, first close the clamping and breaking section 1, insert the intraocular tissue disruptor of this invention into the eye, release the control mechanism 4, open the clamping and breaking section 1 to hold the tissue to be broken, close the clamping and breaking section 1, and the connecting mechanism 2 moves backward, causing the clamping and breaking section 1 to retract into the storage sheath 3. Under the combined action of the breaking end 11 and the cutting edge 32 of the storage sheath 3, the tissue is broken. When the intraocular tissue disruptor of this invention is connected to the negative pressure source 200, turn on the suction switch 64, and the broken tissue is sucked into the suction channel 62 from the suction inlet 61 and discharged out of the eye through the outlet 63.

[0024] The intraocular tissue disruptor of this invention comprises a clamping and disrupting part 1, a connecting mechanism 2, a storage sheath 3, a control mechanism 4, and a handle 5. The connecting mechanism 2 is disposed within the storage sheath 3, with the clamping and disrupting part 1 located at its distal end and the handle 5 located at its proximal end. The control mechanism 4 controls the opening and closing of the clamping and disrupting part 1. When the control mechanism 4 controls the clamping and disrupting part 1 to close, the connecting mechanism 2 drives the clamping and disrupting part 1 to move into the storage sheath 3, and the working part 11 of the clamping and disrupting part 1 closes. Under the combined action of the cutting edge 32 of the storage sheath 3 and the working part 11, the intraocular tissue is disrupted. The intraocular tissue disruptor 100 includes a negative pressure suction mechanism 6. The negative pressure suction mechanism 6 can promptly suction and remove the disrupted tissue from the eye, preventing accidental damage to surrounding intraocular tissues. The intraocular tissue disruptor of this invention is very convenient and safe for clinical use. Attached Figure Description

[0025] Figure 1 This is a three-dimensional structural diagram of the intraocular tissue disruptor of the present invention when the clamping and disrupting part is open.

[0026] Figure 1-1 yes Figure 1 The main view.

[0027] Figure 1-2 yes Figure 1 Enlarged view of point A.

[0028] Figure 1-3 yes Figure 1-1 BB cross-sectional view.

[0029] Figure 1-4 yes Figure 1-3 Enlarged view of point C.

[0030] Figure 1-5 yes Figure 1 Exploded view.

[0031] Figure 2 yes Figure 1A three-dimensional structural diagram of the clamping and breaking part when it is closed.

[0032] Figure 2-1 yes Figure 2 The main view.

[0033] Figure 2-2 yes Figure 2 Enlarged view of point D.

[0034] Figure 2-3 yes Figure 2-1 EE sectional view.

[0035] Figure 3 This is a schematic diagram of the intraocular tissue disruptor of the present invention, which includes three working parts.

[0036] Figure 3-1 yes Figure 3 Enlarged view of point F.

[0037] Figure 4 This is a schematic diagram illustrating the working principle of the intraocular tissue disruptor of the present invention, which includes a negative pressure suction mechanism.

[0038] In the above figure:

[0039] 100 is the intraocular tissue disruptor of the present invention, and 200 is the negative pressure source.

[0040] 1 is the clamping and crushing part, 2 is the connecting mechanism, 3 is the storage sheath, 4 is the control mechanism, 5 is the handle, and 6 is the negative pressure suction mechanism.

[0041] 11 is the working part, 11-1 is the clamping surface, 11-2 is the crushing end; 21 is the center hole; 31 is the through hole, 32 is the cutting edge; 41 is the control switch, 42 is the control core rod, 43 is the connecting seat, 44 is the return spring; 61 is the suction inlet, 62 is the suction channel, 63 is the discharge outlet, and 64 is the suction switch. Detailed Implementation

[0042] Example 1: Intraocular Tissue Disruptor of the Present Invention

[0043] refer to Figures 1 to 2-3 The intraocular tissue disruptor of this embodiment includes a clamping and disrupting part 1, a connecting mechanism 2, a storage sheath 3, a control mechanism 4, and a handle 5.

[0044] The connecting mechanism 2 is disposed inside the storage sheath 3, and the distal end of the connecting mechanism 2 is provided with the clamping and breaking part 1; the proximal end of the connecting mechanism 2 is provided with the handle 5.

[0045] The control mechanism 4 controls the opening or closing of the clamping and crushing part 1.

[0046] The clamping and crushing section 1 includes at least two working sections 11. The clamping section 1 may also have multiple working sections 11, such as three or more, to better ensure the stability of tissue clamping and achieve better crushing results. (Refer to...) Figure 1 and Figure 3 .

[0047] refer to Figure 1-2 The working part 11 includes a clamping surface 11-1 and a crushing end 11-2. The clamping surface 11-1 can effectively fix the tissue to be crushed, and the crushing end 11-2 can apply crushing force to the clamped tissue. The crushing end 11-2 can be designed as a linear cutting edge, a serrated cutting edge, or an arc-shaped cutting edge.

[0048] refer to Figure 1-2 In this embodiment, the clamping surface 11-1 is arc-shaped and encircling. The arc-shaped encircling shape can apply better clamping force to tissues, especially semi-rigid or elastic tissues.

[0049] The control mechanism 4 is a linkage-type control mechanism, a hinge-type control mechanism, or a rotary control mechanism. The applicant has only listed the above three types of control mechanisms. Those skilled in the art can design control mechanisms 4 with different functions as needed, without departing from the protection scope of this application.

[0050] refer to Figure 1-3 and Figure 2-3 In this embodiment, the control mechanism 4 is a linkage control mechanism 4-1; the linkage control mechanism 4-1 includes a control switch 41. When the control switch 41 is pressed, the connecting mechanism 2 moves backward and the clamping crushing part 1 closes. When the control switch 41 is released, the connecting mechanism 2 resets and the clamping crushing part 1 opens.

[0051] The linkage control mechanism 4-1 includes a control switch 41, a control rod 42, a connecting seat 43, and a return spring 44. The control rod 42 is connected to the connecting seat 43, and the return spring 44 is located on the outside of the connecting seat 43. The control rod 42, the connecting seat 43, and the return spring 44 are housed within the housing 51 of the handle 5. The distal end of the housing 51 is provided with a storage sheath 3, and the connecting mechanism 2 is housed within the storage sheath 3. The proximal end of the connecting mechanism 2 is connected to the connecting seat 43. Pressing the control switch 41 causes the control rod 42 to move backward, which in turn causes the connecting seat 43 to move backward. The return spring 44 is compressed, causing the connecting mechanism 2 to move backward. The clamping and crushing part 1 connected to the distal end of the connecting mechanism 2 retracts and enters the storage sheath 3. The clamping and crushing part 1 closes, and the working part 11 and the cutting edge at the distal end of the storage sheath 3 work together to crush the tissue. When the control switch 41 is released, the connecting seat 43 moves forward under the restoring force of the return spring 44. The connecting mechanism 2 connected to the connecting seat 43 drives the clamping and crushing part 1 to move forward. The clamping and crushing part 1 extends out from the distal end of the storage sheath 3 and opens.

[0052] In clinical use, first close the clamping and breaking part 1, place the intraocular tissue disruptor of the present invention into the eye, release the control mechanism 4, open the clamping and breaking part 1 to clamp the tissue to be broken, close the clamping and breaking part 1, the connecting mechanism 2 moves backward, driving the clamping and breaking part 1 into the storage sheath 3, and the tissue is broken by the combined action of the breaking end 11 and the cutting edge 32 of the storage sheath 3.

[0053] When the control mechanism 4 controls the clamping and breaking part 1 to close, the connecting mechanism 2 drives the clamping and breaking part 1 to move into the storage sheath 3. The working part 11 of the clamping and breaking part 1 closes, and under the combined action of the cutting edge 32 of the storage sheath 3 and the working part 11, the intraocular tissue is broken up. The intraocular tissue breaker of this embodiment is very convenient for clinical use.

[0054] Example 2: Intraocular Tissue Disruptor of the Present Invention with a Negative Pressure Suction Mechanism

[0055] refer to Figure 1-3 , Figure 2-3 and Figure 4 The difference between this embodiment and embodiment 1 is that, in this embodiment, the intraocular tissue disruptor 100 further includes a negative pressure suction mechanism 6.

[0056] The negative pressure suction mechanism 6 includes a suction inlet 61, a suction channel 62, and a discharge outlet 63; the suction inlet 61 is located at the distal end of the storage sheath 3, and the discharge outlet 63 is located on the handle 5. The discharge outlet 63 is connected to the negative pressure source 200.

[0057] In this embodiment, the connecting mechanism 2 is provided in the through hole 31 of the storage sheath 3, and the connecting mechanism 2 is a hollow tubular structure.

[0058] The central hole 21 of the hollow tubular structure of the connecting mechanism 2 constitutes the suction channel 62. The hollow tubular structure design maximizes the suction channel, better ensuring that the fragmented tissue can be effectively suctioned out of the eye.

[0059] The negative pressure suction mechanism 6 includes a suction switch 64. The suction switch 64 can control the opening and closing of the negative pressure suction.

[0060] The suction switch 64 is disposed on the handle 5 or as a foot switch. The suction switch 64 may be disposed at various parts of the intraocular tissue disruptor of the present invention as needed, or it may be disposed on the negative pressure source 200. The applicant will not give examples here, but none of them depart from the protection scope of this application.

[0061] In clinical use, first close the clamping and breaking part 1, insert the intraocular tissue disruptor of this invention into the eye, release the control mechanism 4, open the clamping and breaking part 1 to hold the tissue to be broken, close the clamping and breaking part 1, and the connecting mechanism 2 moves backward, causing the clamping and breaking part 1 to retract into the storage sheath 3. Under the combined action of the breaking end 11 and the cutting edge 32 of the storage sheath 3, the tissue is broken. When it is necessary to remove the broken tissue from the eye, turn on the suction switch 64, and the broken tissue is sucked into the suction channel 62 from the suction inlet 61 and discharged from the eye through the outlet 63.

[0062] Compared with Example 1, the presence of the negative pressure suction mechanism 6 allows for timely suction and discharge of fragmented tissue from the eye, preventing accidental damage to surrounding tissues and making it safer for clinical use.

[0063] It should be noted that the structures disclosed and described herein can be replaced by other structures with the same effect, and the embodiments described herein are not the only structures for implementing the present invention. Although preferred embodiments of the present invention have been described and illustrated herein, those skilled in the art will understand that these embodiments are merely illustrative, and those skilled in the art can make numerous variations, modifications, and substitutions without departing from the present invention. Therefore, the scope of protection of the present invention should be defined in accordance with the spirit and scope of the appended claims.

Claims

1. An intraocular tissue disruptor, characterized in that: The intraocular tissue disruptor (100) includes a clamping and disrupting part (1), a connecting mechanism (2), a storage sheath (3), a control mechanism (4), and a handle (5). A. The connecting mechanism (2) is disposed inside the storage sheath (3), and the distal end of the connecting mechanism (2) is provided with the clamping and breaking part (1); the proximal end of the connecting mechanism (2) is provided with the handle (5). B. The control mechanism (4) controls the opening or closing of the clamping and crushing part (1); C. The clamping and crushing part (1) contains at least two working parts (11); D. The end of the storage sheath (3) is provided with a cutting edge (32). When the control mechanism (4) controls the clamping and breaking part (1) to close, the connecting mechanism (2) drives the clamping and breaking part (1) to move into the storage sheath (3). The working part (11) of the clamping and breaking part (1) closes. Under the combined action of the cutting edge (32) and the working part (11), the intraocular tissue is broken.

2. The intraocular tissue disruptor according to claim 1, characterized in that: The clamping and crushing part (1) includes three working parts (11).

3. The intraocular tissue disruptor according to claim 2, characterized in that: The working part (11) includes a clamping surface (11-1) and a crushing end (11-2).

4. The intraocular tissue disruptor according to claim 3, characterized in that: The clamping surface (11-1) is arc-shaped and encircling.

5. The intraocular tissue disruptor according to claim 1, characterized in that: The intraocular tissue disruptor (100) includes a negative pressure suction mechanism (6).

6. The intraocular tissue disruptor according to claim 5, characterized in that: The negative pressure suction mechanism (6) includes a suction inlet (61), a suction channel (62), and a discharge outlet (63); the suction inlet (61) is located at the far end of the storage sheath (3), and the discharge outlet (63) is located on the handle (5).

7. The intraocular tissue disruptor according to claim 1, characterized in that: The control mechanism (4) is a linkage control mechanism, a hinge control mechanism, or a rotary control mechanism.

8. The intraocular tissue disruptor according to claim 7, characterized in that: The control mechanism (4) is a linkage control mechanism (4-1); the linkage control mechanism (4-1) includes a control switch (41). When the control switch (41) is pressed, the connecting mechanism (2) moves backward and the clamping crushing part (1) closes. When the control switch (41) is released, the connecting mechanism (2) resets and the clamping crushing part (1) opens.

9. The intraocular tissue disruptor according to claim 8, characterized in that: The linkage control mechanism (4-1) includes a control switch (41), a control core rod (42), a connecting seat (43), and a return spring (44); the control core rod (42) and the connecting seat (43) are connected, and the return spring (44) is located on the outside of the connecting seat (43); the control core rod (42), the connecting seat (43), and the return spring (44) are located inside the housing (51) of the handle (5), and the distal end of the housing (51) is provided with a storage sheath (3), the connecting mechanism (2) is located inside the storage sheath (3), and the proximal end of the connecting mechanism (2) is connected to the connecting seat (43); when the control switch (41) is pressed, the control core rod (42)... The rod (42) moves backward, causing the connecting seat (43) to move backward. The return spring (44) is compressed, causing the connecting mechanism (2) to move backward. The clamping and breaking part (1) connected to the far end of the connecting mechanism (2) retracts and enters the storage sheath (3), and the clamping and breaking part closes. When the control switch (41) is released, under the restoring force of the return spring (44), the connecting seat (43) moves forward. The connecting mechanism (2) connected to the connecting seat (43) causes the clamping and breaking part (1) to move forward. The clamping and breaking part (1) extends out from the far end of the storage sheath (3), and the clamping and breaking part (1) opens.

10. The intraocular tissue disruptor according to claim 6, characterized in that: The through-hole (31) of the storage sheath (3) constitutes the attraction channel (62).

11. The intraocular tissue disruptor according to claim 9, characterized in that: The connecting mechanism (2) is a hollow tubular structure.

12. The intraocular tissue disruptor according to claim 11, characterized in that: The central hole (21) of the hollow tubular structure of the connecting mechanism (2) forms an attraction channel (62).

13. The intraocular tissue disruptor according to claim 5, characterized in that: The negative pressure suction mechanism (6) includes a suction switch (64).

14. The intraocular tissue disruptor according to claim 13, characterized in that: The attraction switch (64) is located on the handle (5) or is a foot switch.

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