Sound wave and low-frequency pulse cooperative sleep-aiding oxygen cabin control method

Abstract

The invention provides a sound wave and low-frequency pulse collaborative sleep-aiding oxygen cabin control method, which comprises the following steps of: setting a basic period stage of an oxygen cabin on the basis of a superdaily rhythm of a human body sleep period, and determining a core physiological target of the basic period stage; respectively carrying out time sequence matching of low-frequency pulse, sound wave stimulation and oxygen supply pressurization; carrying out nerve conduction path matching and organ response space-time synchronization based on the corresponding organ nerve induction partitions; establishing a fuzzy control model, performing multi-parameter real-time detection, and dynamically adjusting modal parameters according to monitoring data of human body physiological indexes; setting a pulse intensity upper limit, a sound wave decibel upper limit, an oxygen concentration upper limit and a single pressurization duration, and executing oxygen cabin operation control according to the modal parameters. The method has the advantages in physiological adaptation, collaborative accuracy, individual adaptation and safety guarantee, fits the electroencephalogram and physiological law of natural sleep, and improves the sleep continuity.

Description

Technical Field

[0001] This invention belongs to the field of intelligent oxygen chamber technology, specifically relating to an oxygen chamber control method that uses sound waves and low-frequency pulses to synergistically promote sleep. Background Technology

[0002] With the fast pace of modern life and rising stress levels, sleep disorders have become a common problem affecting people's health. According to relevant studies, about one-third of the world's population suffers from varying degrees of difficulty falling asleep, shallow sleep, frequent awakenings, or sleep maintenance disorders.

[0003] In the process of conceiving and researching this technical solution, the applicant has identified at least the following problems: While current sleep aid technologies are diverse, they generally have shortcomings. Single-technique sleep aids are often effective in the short term, but long-term use may lead to dependence, disrupt normal sleep rhythms, and cause various physical discomforts. Summary of the Invention

[0004] To alleviate the above problems, the main objective of this application is to propose a method for controlling an oxygen chamber that uses sound waves and low-frequency pulses in synergistic sleep aid, including:

[0005] Based on the super-diurnal rhythm of the human sleep cycle, the basic cycle phase of the oxygen chamber is set, and the core physiological objectives of the basic cycle phase are determined.

[0006] Based on the core physiological goals of the aforementioned basic cycle phase, the timing of low-frequency pulses, sound wave stimulation, and oxygen supply pressurization is matched accordingly.

[0007] Based on the organ nerve sensory partitions corresponding to the low-frequency pulse, sound wave stimulation and oxygen supply pressurization, nerve conduction pathway matching and organ response spatiotemporal synchronization are performed.

[0008] A fuzzy control model is established, and multiple parameters are detected in real time. The modal parameters are dynamically adjusted based on the monitoring data of human physiological indicators.

[0009] Set the upper limit of pulse intensity, upper limit of sound decibel, upper limit of oxygen concentration, and duration of single pressurization, and execute oxygen chamber operation control according to the modal parameters.

[0010] Optionally, the process of setting the basic cycle phase of the oxygen chamber based on the hyperrheic rhythm of the human sleep cycle and determining the core physiological goals of the basic cycle phase includes:

[0011] Using the super-diurnal rhythm of the human sleep cycle as the underlying time reference, the core time frame and stage dimensions are determined.

[0012] Based on the aforementioned core time frame and stage dimensions, the core physiological goals for each stage are defined according to the characteristics of human sleep.

[0013] Optionally, the process of defining the core physiological goals for each stage based on the core time frame and stage dimensions, according to the characteristics of human sleep, and preceding this process, includes:

[0014] The basic cycle phases of the oxygen chamber are set as the sleep induction period, the deep sleep period, and the sleep maintenance period;

[0015] During the sleep induction period, the brain's arousal signals are suppressed, the parasympathetic nervous system is activated, and blood oxygen saturation is increased.

[0016] During the deep sleep period, the brainstem's inhibition of the brain's arousal signals is enhanced, promoting the generation of deep sleep characteristic waves and stabilizing cerebral blood flow.

[0017] During the sleep maintenance period, mild neural inhibition is maintained to prevent sudden awakening from deep sleep and to simulate the mechanical stimulation of natural breathing.

[0018] Optionally, the process of timing-matching low-frequency pulses, sound wave stimulation, and oxygen pressurization according to the core physiological goals of the basal cycle phase includes:

[0019] Based on the core physiological goals of each stage, the key physiological characteristics corresponding to each stage are determined.

[0020] Based on the aforementioned key physiological characteristics, the control timing of the low-frequency pulse, sound wave stimulation, and oxygen supply pressurization is set respectively.

[0021] Optionally, the process of setting the control timing of the low-frequency pulse, sound wave stimulation, and oxygen supply pressurization according to the key physiological characteristics, and the preceding steps, include:

[0022] The key physiological characteristics of the sleep induction period are set as follows: transitioning from a waking state to a light sleep state, with a gradual decrease in heart rate, and achieving a target blood oxygen saturation of 95%. During the sleep induction period, 0.5-2Hz pulse stimulation is initiated to reduce the heart rate. Simultaneously, sound waves of the same frequency as the low-frequency pulse are released, and the fundamental frequencies of the low-frequency pulse and the sound wave stimulation are strictly aligned to form phase lock, establishing synergy between the auditory cortex and the pulse signal. At the same time, the pressure inside the oxygen chamber is gradually increased from atmospheric pressure to 1.2 ATA, and the oxygen concentration is simultaneously increased to 30%-40%.

[0023] Optionally, the key physiological characteristics of the deep sleep period are set to simulate the brainwave transition process from shallow to deep sleep in natural sleep, while controlling the risk of oxygen exposure; during the deep sleep period, the pulse frequency is increased to 2-3Hz, the main frequency of the sound wave is adjusted to be the same as the pulse frequency, and a 4-7Hz theta wave sound wave is inserted during the pulse interval, while the cabin pressure is stabilized at 1.3ATA, and the oxygen concentration is dynamically adjusted through blood oxygen monitoring feedback.

[0024] Optionally, the key physiological characteristics of the sleep maintenance period are set to match the EEG characteristics of REM sleep, with air pressure and blood oxygen gradually decreasing to near normal pressure levels, and chest and abdominal fluctuations simulated by micro-pressure difference changes; during the sleep maintenance period, the pulse is switched to 0.5-1Hz, while the intensity is reduced to half of the initial value, 1-4Hz mixed sound waves are released, the cabin pressure gradually decreases to 1.1ATA, the oxygen concentration decreases to 25%, and a brief negative pressure is introduced during the pulse interval.

[0025] Optionally, the process of matching neural conduction pathways and synchronizing organ responses in spatiotemporal mode based on the organ's neural sensory partitions corresponding to the low-frequency pulse, sound wave stimulation, and oxygen supply pressurization includes:

[0026] Based on the corresponding organ nerve sensory zones of low-frequency pulses, sound wave stimulation, and oxygen supply pressurization, conduction pathways are designed and targeted conduction pathways are planned so that the signals from multiple pathways can ultimately work synergistically to the relevant nerve centers.

[0027] Based on the neural conduction pathway matching and combined with the signal conduction speed of each stimulation mode, the output rhythm of each stimulus is synchronously regulated during the peak pulse period and the inter-pulse period, so that the organ response is coordinated in time and space.

[0028] Optionally, the process of designing conduction pathways and planning targeted conduction pathways based on the corresponding organ nerve sensory zones of low-frequency pulses, sound wave stimulation, and oxygen supply pressurization, so that the multi-path signals ultimately work synergistically to the relevant nerve centers, includes:

[0029] The conductive pads on the armrests of the oxygen chamber seats correspond to the median and ulnar nerves of the arm. The low-frequency pulses released by the conductive pads stimulate the median and ulnar nerves, and the signals are transmitted along the spinothalamic tract to the brainstem reticular formation, directly inhibiting the ascending activating system and achieving targeted transmission from the peripheral nerves to the brainstem.

[0030] Optionally, air conduction is configured to correspond to the external auditory canal to the cochlear hair cells, and bone conduction is configured to correspond to the skull in contact with the occiput to the cochlea. Air conduction allows sound waves to activate the cochlear hair cells via the external auditory canal, and the signal is transmitted from the auditory center of the brainstem to the thalamus. Bone conduction allows sound waves to directly stimulate the cochlea through skull vibration, and the signal is transmitted along the same path. After the two signals are integrated in the auditory cortex, they are transmitted through the thalamus and cortical pathways, and resonate with the slow waves induced by low-frequency pulses in the brainstem reticular formation, thereby achieving the coordinated transmission of sound wave signal stimulation to the brainstem.

[0031] Optionally, the increased oxygen partial pressure corresponds to stimulation of the carotid body chemoreceptors, causing the stimulation signal to be transmitted through the sinus nerve to the glossopharyngeal nerve to the medullary respiratory center, reflexively reducing the excitability of the medullary respiratory center, and indirectly inhibiting the arousal drive of the brainstem reticular formation, forming a supplementary transmission from peripheral chemoreception to the brainstem with the transmission pathway of low-frequency pulses and sound waves.

[0032] Optionally, the process of synchronously regulating the output rhythm of each stimulus during the peak and interval periods of the pulse, based on the matching of the neural conduction pathways and combined with the signal conduction velocity of each stimulus modality, to enable the organ response to coordinate in time and space, includes:

[0033] When the low-frequency pulse is at its peak, the sound wave generator releases the sound wave peak to synchronously trigger the oxygen supply system to instantly increase the flow rate, forming an instantaneous synergistic effect to enhance the inhibition of the locus coeruleus and achieve spatiotemporal synchronization of organ response during the pulse peak period.

[0034] When the low-frequency pulse is in a resting state, the sound wave stimulation is switched to the 4-7Hz theta wave frequency band, and the oxygen supply mode is simultaneously adjusted to intermittent pulse oxygen supply. This stimulates the lung stretch receptors through the micro-pressure difference in the chamber, enhances vagal nerve tone, and works with the theta wave sound wave to maintain the stable response of the organs, achieving spatiotemporal synchronization of organ response during the pulse interval.

[0035] Optionally, the process of establishing a fuzzy control model and performing real-time multi-parameter detection, and dynamically adjusting modal parameters based on monitoring data of human physiological indicators, includes:

[0036] Establish a physiological indicator collection system to conduct real-time monitoring of multiple parameters and obtain the data foundation for dynamic regulation;

[0037] Construct the correlation logic between physiological indicators and multimodal parameters, and establish a fuzzy control model;

[0038] The stimulation parameters are optimized in real time based on the output of the fuzzy control model.

[0039] Optionally, the process of constructing the correlation logic between physiological indicators and multimodal parameters, and establishing the fuzzy control model, includes the following prior steps:

[0040] Collect data on the proportion of alpha, delta, and theta waves in the brain to determine the current sleep stage of the target user;

[0041] Analyze the low-frequency and high-frequency components in heart rate variability and calculate the ratio of low-frequency to high-frequency components to determine the level of sympathetic nerve activity.

[0042] Real-time tracking of blood oxygen saturation levels ensures that blood oxygen saturation monitoring remains within a safe and effective range;

[0043] Acquire skin conductance monitoring data, and use changes in skin resistance to reflect the user's stress response and help indicate the activity of the sympathetic nervous system;

[0044] The real-time monitored brainwave proportions, low-frequency to high-frequency component ratios, blood oxygen saturation values, and skin conductance values ​​are converted into fuzzy linguistic variables and divided into fuzzy subsets.

[0045] Based on the physiological mechanisms of sleep, targeted regulation rules are set, and a fuzzy rule base is constructed.

[0046] Output layer mapping is performed to transform the fuzzy rule inference results into specific trimodal parameter adjustment values, ensuring that the adjustment instructions can directly drive hardware execution.

[0047] This application provides a method for controlling an oxygen chamber that uses sound waves and low-frequency pulses to promote sleep. Based on the trans-diurnal rhythm of the human sleep cycle, a basic cycle phase of the oxygen chamber is established, and the core physiological goals of this basic cycle phase are determined. According to the core physiological goals of the basic cycle phase, the timing of low-frequency pulses, sound wave stimulation, and oxygen pressurization is matched. Based on the organ nerve sensory partitions corresponding to the low-frequency pulses, sound wave stimulation, and oxygen pressurization, neural conduction pathway matching and spatiotemporal synchronization of organ responses are performed. A fuzzy control model is established, and multiple parameters are monitored in real time. Modal parameters are dynamically adjusted based on monitoring data of human physiological indicators. Upper limits for pulse intensity, sound wave decibels, oxygen concentration, and single pressurization duration are set, and oxygen chamber operation control is executed according to the modal parameters. This method has significant advantages in physiological adaptation, precise coordination, individual adaptation, and safety assurance. The oxygen chamber regulation is designed to perfectly match the EEG characteristics and physiological transition patterns of natural sleep, laying a physiological foundation for efficient sleep aid, significantly improving sleep aid efficiency and sleep continuity, greatly enhancing the targeted inhibition effect on the arousal center, achieving personalized and refined sleep aid regulation, and providing users with a safe and reliable user experience. Attached Figure Description

[0048] The accompanying drawings, which are incorporated in and form part of this specification, illustrate embodiments consistent with this application and, together with the description, serve to explain the principles of this application. To more clearly illustrate the technical solutions of the embodiments of this application, the drawings used in the description of the embodiments will be briefly introduced below. Obviously, those skilled in the art can obtain other drawings based on these drawings without any creative effort.

[0049] Figure 1 This is an embodiment of the present application.

[0050] The realization of the objectives, functional features, and advantages of this application will be further explained in conjunction with the embodiments and with reference to the accompanying drawings. The accompanying drawings have illustrated specific embodiments of this application, which will be described in more detail below. These drawings and textual descriptions are not intended to limit the scope of the concept in any way, but rather to illustrate the concepts of this application to those skilled in the art through reference to specific embodiments. Detailed Implementation

[0051] It should be understood that the specific embodiments described herein are merely illustrative of this application and are not intended to limit this application.

[0052] Various embodiments of the present application will now be described with reference to the accompanying drawings. In the following description, suffixes such as “module,” “part,” or “unit” used to denote elements are used only for the convenience of the description and have no specific meaning in themselves.

[0053] First Embodiment

[0054] To alleviate the above problems, the main objective of this application is to propose a method for controlling an oxygen chamber that uses sound waves and low-frequency pulses in synergistic sleep aid. Figure 1 This is a flowchart of an oxygen chamber control method for synergistic sleep aid using sound waves and low-frequency pulses, according to an embodiment of this application.

[0055] like Figure 1 As shown, in one embodiment, an oxygen chamber control method for synergistic sleep aid using sound waves and low-frequency pulses includes:

[0056] S10: Based on the super-diurnal rhythm of the human sleep cycle, set the basic cycle stage of the oxygen chamber and determine the core physiological goals of the basic cycle stage.

[0057] "Multimodal" refers to the various core stimulation / regulation methods that oxygen chambers can use to achieve synergistic sleep-aiding functions. For example, the three modalities work synergistically through temporal synchronization, spatial coupling, and closed-loop feedback to jointly achieve the goal of inhibiting arousal and promoting sleep. For instance, using the super-diurnal rhythm of the human sleep cycle as the underlying time benchmark, the overall regulation cycle of the oxygen chamber is clearly divided into three function-oriented core stages. Each stage corresponds to key physiological changes in the sleep process, providing a time framework for setting parameters for subsequent multimodal stimulation (low-frequency pulses, sound waves, and oxygen supply). For example, based on the super-diurnal rhythm of the human sleep cycle (approximately 90 minutes), the oxygen chamber regulation cycle is divided into three stages: the sleep induction period (0-30 minutes), the deep sleep period (30-90 minutes), and the sleep maintenance period (after 90 minutes). Synergistic parameters for sound waves, pulses, and oxygen supply are set for each stage.

[0058] After dividing the basic cycle into stages, the characteristics of human sleep and core physiological goals such as EEG waveform, heart rate, and blood oxygen demand for each stage are further clarified, providing a physiological basis for the subsequent timing matching of "low-frequency pulse, sound wave stimulation, and oxygen supply pressurization".

[0059] S20: Based on the core physiological goals of the basic cycle phase, perform timing matching of low-frequency pulses, sound wave stimulation, and oxygen supply pressurization.

[0060] For example, during the sleep induction period, the main focus is on matching the transition characteristics from wakefulness to light sleep, with the core objective of suppressing arousal signals and activating the parasympathetic nervous system. During the deep sleep period, the focus is on matching the transition characteristics from light sleep to deep sleep, with the core objective of promoting delta wave generation and preventing neural adaptation. During the sleep maintenance period, the focus is on matching the characteristics from deep sleep to REM sleep cycles, with the core objective of stabilizing the sleep state and preventing sudden awakenings.

[0061] S30: Based on the organ nerve sensing partitions corresponding to the low-frequency pulse, sound wave stimulation and oxygen supply pressurization, perform nerve conduction path matching and organ response spatiotemporal synchronization.

[0062] For example, during the neural conduction pathway matching process, targeted conduction pathways can be planned according to the organ neural sensing zones of each stimulus modality. During the spatiotemporal synchronization of organ response, the instantaneous coordination of multimodal stimuli and organ responses can be achieved according to the temporal characteristics of neural conduction.

[0063] S40: Establish a fuzzy control model and perform real-time detection of multiple parameters, dynamically adjusting modal parameters based on monitoring data of human physiological indicators.

[0064] For example, a physiological indicator acquisition system is built to achieve real-time monitoring of multiple parameters, thereby obtaining the data foundation for dynamic regulation; a fuzzy control model is established to construct the correlation logic between physiological indicators and three-modal parameters. Finally, based on the output of the fuzzy control model, the stimulation parameters are optimized in real time to achieve dynamic adjustment of multimodal parameters.

[0065] S50: Set the upper limit of pulse intensity, upper limit of sound decibels, upper limit of oxygen concentration and single pressurization duration, and execute oxygen chamber operation control according to the modal parameters.

[0066] For example, upper limits for pulse intensity (<5mA), sound wave decibels (<60dB, to avoid hearing damage), oxygen concentration (40%), and single pressurization duration (<120 minutes) can be set. Through hardware redundancy circuits and software watchdog programs, physiological risks caused by exceeding the limits of multimodal parameters can be prevented.

[0067] This embodiment boasts significant advantages in physiological adaptation, precise coordination, individualized fit, and safety assurance. It ensures that the oxygen chamber regulation perfectly aligns with the EEG characteristics and physiological transition patterns of natural sleep, laying a physiological foundation for efficient sleep aid, significantly improving sleep efficiency and sleep continuity, greatly enhancing the targeted inhibition effect on the arousal center, achieving personalized and refined sleep aid regulation, and providing users with a safe and reliable experience.

[0068] Optionally, the process of setting the basic cycle phase of the oxygen chamber based on the hyperrheic rhythm of the human sleep cycle and determining the core physiological goals of the basic cycle phase includes:

[0069] Using the super-diurnal rhythm of the human sleep cycle as the underlying time reference, the core time frame and stage dimensions are determined.

[0070] Based on the aforementioned core time frame and stage dimensions, the core physiological goals for each stage are defined according to the characteristics of human sleep.

[0071] For example, using the super-diurnal rhythm of the human sleep cycle (approximately 90 minutes) as the underlying time benchmark, the overall regulation cycle of the oxygen chamber is clearly divided into three function-oriented core stages. Each stage corresponds to key physiological changes in the sleep process, providing a time framework for setting parameters for subsequent multimodal stimulation (low-frequency pulses, sound waves, and oxygen supply). After dividing the basic cycle stages, the corresponding human sleep characteristics (such as EEG waveforms, heart rate, and blood oxygen demand) for each stage are further clarified, providing a physiological basis for the timing matching of subsequent "low-frequency pulses, sound wave stimulation, and oxygen supply pressurization".

[0072] Optionally, the process of defining the core physiological goals for each stage based on the core time frame and stage dimensions, according to the characteristics of human sleep, and preceding this process, includes:

[0073] The basic cycle phases of the oxygen chamber are set as the sleep induction period, the deep sleep period, and the sleep maintenance period;

[0074] During the sleep induction period, the brain's arousal signals are suppressed, the parasympathetic nervous system is activated, and blood oxygen saturation is increased.

[0075] During the deep sleep period, the brainstem's inhibition of the brain's arousal signals is enhanced, promoting the generation of deep sleep characteristic waves and stabilizing cerebral blood flow.

[0076] During the sleep maintenance period, mild neural inhibition is maintained to prevent sudden awakening from deep sleep and to simulate the mechanical stimulation of natural breathing.

[0077] For example, the sleep induction period can last from 0 to 30 minutes, with the core objective of helping users transition from wakefulness to light sleep, focusing on matching the physiological needs of "wakefulness inhibition → initial sleep onset". The core physiological objectives during this period can be set as inhibiting brain arousal signals (reducing the proportion of alpha waves), activating the parasympathetic nervous system (reducing heart rate), and increasing blood oxygen saturation (creating a physiological basis for sleep onset).

[0078] The time interval of deep sleep can be set to 30-90 minutes. The core goal is to help users transition from light sleep to deep sleep, focusing on matching the EEG characteristics and blood oxygenation requirements of "light sleep → deep sleep". The core physiological goals can be to enhance the brainstem's inhibition of arousal signals, promote the generation of delta waves (0.5-4Hz, characteristic waves of deep sleep), and stabilize cerebral blood flow (which needs to be reduced by about 20%).

[0079] The sleep maintenance period can be set to 90 minutes, with the core objectives being to stabilize deep sleep, prevent sudden awakenings, and simulate the EEG characteristics of REM sleep (rapid eye movement sleep), focusing on matching the physiological cycle of "deep sleep maintenance → natural sleep cycle." Core physiological goals include maintaining mild neural inhibition, preventing sudden awakenings from deep sleep, and simulating the mechanical stimulation of natural breathing.

[0080] Optionally, the process of timing-matching low-frequency pulses, sound wave stimulation, and oxygen pressurization according to the core physiological goals of the basal cycle phase includes:

[0081] Based on the core physiological goals of each stage, the key physiological characteristics corresponding to each stage are determined.

[0082] Based on the aforementioned key physiological characteristics, the control timing of the low-frequency pulse, sound wave stimulation, and oxygen supply pressurization is set respectively.

[0083] For example, during the sleep induction period, the main focus is on matching the transition characteristics from wakefulness to light sleep, with the core objective of suppressing arousal signals and activating the parasympathetic nervous system. During the deep sleep period, the focus is on matching the transition characteristics from light sleep to deep sleep, with the core objective of promoting delta wave generation and preventing neural adaptation. During the sleep maintenance period, the focus is on matching the characteristics from deep sleep to REM sleep cycles, with the core objective of stabilizing the sleep state and preventing sudden awakenings.

[0084] Optionally, the process of setting the control timing of the low-frequency pulse, sound wave stimulation, and oxygen supply pressurization according to the key physiological characteristics, and the preceding steps, include:

[0085] The key physiological characteristics of the sleep induction period are set as follows: transitioning from a waking state to a light sleep state, with a gradual decrease in heart rate, and achieving a target blood oxygen saturation of 95%. During the sleep induction period, 0.5-2Hz pulse stimulation is initiated to reduce the heart rate. Simultaneously, sound waves of the same frequency as the low-frequency pulse are released, and the fundamental frequencies of the low-frequency pulse and the sound wave stimulation are strictly aligned to form phase lock, establishing synergy between the auditory cortex and the pulse signal. At the same time, the pressure inside the oxygen chamber is gradually increased from atmospheric pressure to 1.2 ATA, and the oxygen concentration is simultaneously increased to 30%-40%.

[0086] For example, the key physiological characteristics of the sleep induction period need to transition from a waking state (primarily alpha waves at 8-12Hz) to a light sleep state (with theta waves initially appearing at 4-7Hz), with a gradual decrease in heart rate and a target blood oxygen saturation of ≥95%. A 0.5-2Hz pulse (e.g., 1Hz) is initiated and applied to the sensory nerves of the arm through conductive pads on the armrest, directly stimulating the parasympathetic nervous system, reducing heart rate, and achieving timing matching of the low-frequency pulse, thus establishing the physiological basis for sleep. A sound wave generator at the occipital region synchronously releases a 1Hz sound wave (including bone conduction components), forming a phase lock with the low-frequency pulse. Through the synergy between the auditory cortex and the pulse signal, it enhances the inhibition of alpha waves (8-12Hz, the dominant wave in the waking state), weakens the arousal signal, and achieves timing matching of sound wave stimulation. The cabin pressure is gradually increased from atmospheric pressure to 1.2 ATA (simulating the oxygen partial pressure at an altitude of 1500 meters), while the oxygen concentration is simultaneously increased to 30%-40%. By increasing blood oxygen saturation (target ≥95%), the brain's glucose metabolism rate is reduced, creating a low-metabolic, high-oxygen physiological environment for falling asleep, and achieving timing matching of oxygen supply and pressurization.

[0087] Optionally, the key physiological characteristics of the deep sleep period are set to simulate the brainwave transition process from shallow to deep sleep in natural sleep, while controlling the risk of oxygen exposure; during the deep sleep period, the pulse frequency is increased to 2-3Hz, the main frequency of the sound wave is adjusted to be the same as the pulse frequency, and a 4-7Hz theta wave sound wave is inserted during the pulse interval, while the cabin pressure is stabilized at 1.3ATA, and the oxygen concentration is dynamically adjusted through blood oxygen monitoring feedback.

[0088] For example, during deep sleep, neural adaptation to a single stimulus should be avoided, mimicking the EEG transition from light sleep to deep sleep in natural sleep, while controlling oxygen exposure risk (pressurization duration should be <60 minutes). The pulse frequency is increased to 2-3 Hz (e.g., 2.5 Hz) and strictly aligned with the fundamental frequency of the sound waves. This enhances the inhibition of arousal signals by the brainstem reticular formation through phase synchronization, directly promoting the generation of delta waves (0.5-4 Hz, the dominant wave in deep sleep), achieving low-frequency pulse timing matching. During the timing matching of sound wave stimulation, the dominant frequency is maintained at 2.5 Hz (the same frequency as the pulse), while 4-7 Hz theta waves are inserted during pulse intervals (e.g., every 50 seconds) to simulate the EEG transition from light sleep to deep sleep in natural sleep, avoiding neural adaptive fatigue caused by single-frequency stimulation. During the oxygen supply and pressurization timing matching process, the cabin pressure can be stabilized at 1.3 ATA, and the oxygen concentration can be maintained at 30%. The oxygen supply is dynamically adjusted through blood oxygen monitoring feedback: on the one hand, it ensures that cerebral blood flow is reduced by about 20% (which is in line with the physiological characteristics of deep sleep), and on the other hand, it controls the pressurization time to <60 minutes to avoid the risk of oxygen poisoning.

[0089] Optionally, the key physiological characteristics of the sleep maintenance period are set to match the EEG characteristics of REM sleep, with air pressure and blood oxygen gradually decreasing to near normal pressure levels, and chest and abdominal fluctuations simulated by micro-pressure difference changes; during the sleep maintenance period, the pulse is switched to 0.5-1Hz, while the intensity is reduced to half of the initial value, 1-4Hz mixed sound waves are released, the cabin pressure gradually decreases to 1.1ATA, the oxygen concentration decreases to 25%, and a brief negative pressure is introduced during the pulse interval.

[0090] For example, during the sleep maintenance phase, the EEG characteristics of REM sleep (containing 20% ​​alpha wave component) can be matched. Air pressure and blood oxygen levels need to gradually decrease to near-normal levels, simulating chest and abdominal fluctuations through micro-pressure differential changes. At this time, the pulses switch to 0.5-1Hz low-frequency oscillations, while the intensity is reduced to 50% of the initial value, maintaining only mild neural inhibition to avoid overstimulation interfering with the sleep cycle, thus achieving low-frequency pulse timing matching. During the sound wave stimulation timing matching process, 1-4Hz mixed sound waves (containing 20% ​​alpha wave component) are released to simulate the EEG characteristics of REM sleep, preventing the user from suddenly waking from deep sleep and maintaining sleep continuity. During the oxygen supply and pressurization timing matching process, the cabin pressure can gradually decrease to 1.1 ATA, and the oxygen concentration drops to 25%; simultaneously, a brief negative pressure (-0.05 ATA) is introduced during the pulse interval, simulating the chest and abdominal fluctuations of natural breathing through air pressure changes, forming a synergy of "mechanical stimulation + neural regulation" to further stabilize the sleep state.

[0091] Optionally, the process of matching neural conduction pathways and synchronizing organ responses in spatiotemporal mode based on the organ's neural sensory partitions corresponding to the low-frequency pulse, sound wave stimulation, and oxygen supply pressurization includes:

[0092] Based on the corresponding organ nerve sensory zones of low-frequency pulses, sound wave stimulation, and oxygen supply pressurization, conduction pathways are designed and targeted conduction pathways are planned so that the signals from multiple pathways can ultimately work synergistically to the relevant nerve centers.

[0093] Based on the neural conduction pathway matching and combined with the signal conduction speed of each stimulation mode, the output rhythm of each stimulus is synchronously regulated during the peak pulse period and the inter-pulse period, so that the organ response is coordinated in time and space.

[0094] For example, during the neural conduction pathway matching process, targeted conduction pathways can be planned according to the organ neural sensory regions of each stimulus modality. Based on the corresponding organ neural sensory regions (such as peripheral nerves of the arm, auditory system, and cardiovascular chemoreceptors) for low-frequency pulses, sound wave stimulation, and oxygen supply pressurization, conduction pathways are designed separately to ensure that the multi-path signals ultimately synergistically act on the nerve centers related to "inhibition of arousal." During the spatiotemporal synchronization of organ responses, the temporal characteristics of neural conduction are used to achieve instantaneous synergy between multimodal stimulation and organ responses. Based on the completed neural conduction pathway matching, and combined with the signal conduction speed of each stimulus modality (such as the time difference between pulse electrical signals, sound wave vibration signals, and blood oxygen change signals), the output rhythm of each stimulus is synchronously controlled at the two key time nodes of "pulse peak period" and "pulse interval period" to ensure a high degree of spatiotemporal synergy in organ responses (such as neural excitation, blood oxygen fluctuations, and respiratory adjustments).

[0095] Optionally, the process of designing conduction pathways and planning targeted conduction pathways based on the corresponding organ nerve sensory zones of low-frequency pulses, sound wave stimulation, and oxygen supply pressurization, so that the multi-path signals ultimately work synergistically to the relevant nerve centers, includes:

[0096] The conductive pads on the armrests of the oxygen chamber seats correspond to the median and ulnar nerves of the arm. The low-frequency pulses released by the conductive pads stimulate the median and ulnar nerves, and the signals are transmitted along the spinothalamic tract to the brainstem reticular formation, directly inhibiting the ascending activating system and achieving targeted transmission from the peripheral nerves to the brainstem.

[0097] For example, after the low-frequency pulses released by the conductive pads on the armrests of the oxygen chamber seats stimulate the median and ulnar nerves, the signals are transmitted along the spinothalamic tract (the conduction pathway from peripheral nerves to the spinal cord to the thalamus) to the brainstem reticular formation, directly inhibiting the ascending activating system (ARAS, the core center for arousal signal transmission), thus achieving targeted transmission from peripheral nerves to the brainstem.

[0098] Optionally, air conduction is configured to correspond to the external auditory canal to the cochlear hair cells, and bone conduction is configured to correspond to the skull in contact with the occiput to the cochlea. Air conduction allows sound waves to activate the cochlear hair cells via the external auditory canal, and the signal is transmitted from the auditory center of the brainstem to the thalamus. Bone conduction allows sound waves to directly stimulate the cochlea through skull vibration, and the signal is transmitted along the same path. After the two signals are integrated in the auditory cortex, they are transmitted through the thalamus and cortical pathways, and resonate with the slow waves induced by low-frequency pulses in the brainstem reticular formation, thereby achieving the coordinated transmission of sound wave signal stimulation to the brainstem.

[0099] For example, air-conducted sound waves activate cochlear hair cells via the external auditory canal, and the signal is transmitted along the auditory nerve → brainstem auditory center → thalamus. Bone-conducted sound waves directly stimulate the cochlea through skull vibration, and the signal is transmitted along the same path. After the two signals are integrated in the auditory cortex, they are transmitted through the thalamus and cortical pathways, resonating with slow waves induced by low-frequency pulses (such as delta waves and theta waves) in the brainstem reticular formation, achieving coordinated transmission of auditory-somatosensory → cortex → brainstem.

[0100] Optionally, the increased oxygen partial pressure corresponds to stimulation of the carotid body chemoreceptors, causing the stimulation signal to be transmitted through the sinus nerve to the glossopharyngeal nerve to the medullary respiratory center, reflexively reducing the excitability of the medullary respiratory center, and indirectly inhibiting the arousal drive of the brainstem reticular formation, forming a supplementary transmission from peripheral chemoreception to the brainstem with the transmission pathway of low-frequency pulses and sound waves.

[0101] For example, after high oxygen partial pressure stimulates the carotid body chemoreceptors, the signal is transmitted to the medullary respiratory center via the sinus nerve to the glossopharyngeal nerve, reflexively reducing the excitability of the medullary respiratory center, while indirectly inhibiting the arousal drive of the brainstem reticular formation. This, together with the conduction pathway of low-frequency pulses and sound waves, forms a supplementary conduction pathway of "peripheral chemoreception → medulla oblongata → brainstem", which together enhances the arousal inhibition effect.

[0102] Optionally, the process of synchronously regulating the output rhythm of each stimulus during the peak and interval periods of the pulse, based on the matching of the neural conduction pathways and combined with the signal conduction velocity of each stimulus modality, to enable the organ response to coordinate in time and space, includes:

[0103] When the low-frequency pulse is at its peak, the sound wave generator releases the sound wave peak to synchronously trigger the oxygen supply system to instantly increase the flow rate, forming an instantaneous synergistic effect to enhance the inhibition of the locus coeruleus and achieve spatiotemporal synchronization of organ response during the pulse peak period.

[0104] When the low-frequency pulse is in a resting state, the sound wave stimulation is switched to the 4-7Hz theta wave frequency band, and the oxygen supply mode is simultaneously adjusted to intermittent pulse oxygen supply. This stimulates the lung stretch receptors through the micro-pressure difference in the chamber, enhances vagal nerve tone, and works with the theta wave sound wave to maintain the stable response of the organs, achieving spatiotemporal synchronization of organ response during the pulse interval.

[0105] Based on the matching of neural conduction pathways, and combined with the signal conduction speed of each stimulus modality (such as the time difference of pulse electrical signals, sound wave vibration signals, and blood oxygen change signals), the output rhythm of each stimulus is synchronously regulated at the two key time nodes of "pulse peak period" and "pulse interval period" to ensure that organ responses (such as nerve excitation, blood oxygen fluctuations, and respiratory adjustment) are highly coordinated in time and space.

[0106] For example, when the frequency pulse is at its peak (such as the positive current phase of a 2.5Hz pulse, the period of fastest electrical signal conduction), two synergistic actions are triggered simultaneously: A) the sound wave generator releases the sound wave peak (ensuring that the sound wave vibration signal and the pulse electrical signal reach the auditory cortex simultaneously); B) the oxygen supply system instantaneously increases the flow rate (+10% instantaneous flow rate, ensuring that the blood oxygen concentration rises rapidly while the pulse inhibits arousal). This forms an instantaneous synergy of "electrical stimulation (pulse) - acoustic vibration (sound wave) - rapid increase in blood oxygen (oxygen supply)," which jointly strengthens the inhibition of the locus coeruleus (the noradrenergic arousal center) and avoids organ response delays caused by single stimulation.

[0107] For example, when the low-frequency pulse is in a resting state (e.g., during a 50-second interval, when the electrical signal is paused), two parameters are simultaneously adjusted: A) the sound wave stimulation is switched to the theta wave band of 4-7Hz (matching the EEG transition from light sleep to deep sleep in natural sleep, filling the gap in arousal inhibition during pulse pauses); B) the oxygen supply mode is adjusted to intermittent pulse oxygen supply (5 seconds of high flow + 10 seconds of low flow, simulating the fluctuation of respiratory rate from 12 breaths / min to 8 breaths / min in natural sleep). The micro-pressure difference in the chamber (pressure changes caused by intermittent oxygen supply) stimulates the lung stretch receptors, further enhancing vagal tone, and together with the theta wave sound waves, maintains the stable response of organs (lungs and brain), avoiding a rebound of arousal signals during pulse intervals.

[0108] Optionally, the process of establishing a fuzzy control model and performing real-time multi-parameter detection, and dynamically adjusting modal parameters based on monitoring data of human physiological indicators, includes:

[0109] Establish a physiological indicator collection system to conduct real-time monitoring of multiple parameters and obtain the data foundation for dynamic regulation;

[0110] Construct the correlation logic between physiological indicators and multimodal parameters, and establish a fuzzy control model;

[0111] The stimulation parameters are optimized in real time based on the output of the fuzzy control model.

[0112] For example, by integrating multiple types of sensors, core indicators directly related to sleep state and physiological safety are collected in real time, covering brain electrical activity, autonomic nervous function, blood oxygen level, and stress response, providing data input for the fuzzy control model. Based on the characteristics of the above-mentioned monitoring indicators (such as the proportion of brain electrical waveforms, the ratio of low-frequency components to high-frequency components, etc., are all fuzzy data without absolutely clear threshold boundaries), a fuzzy control model is established to clarify the corresponding rules of "abnormal physiological indicators → three-modal parameter adjustment strategy". According to the inference results of the fuzzy control model, the parameters of low-frequency pulses, sound wave stimulation, and oxygen supply pressurization are dynamically adjusted to ensure that the three-modal stimulation always matches the user's current physiological state.

[0113] Optionally, the process of constructing the correlation logic between physiological indicators and multimodal parameters, and establishing the fuzzy control model, includes the following prior steps:

[0114] Collect data on the proportion of alpha, delta, and theta waves in the brain to determine the current sleep stage of the target user;

[0115] Analyze the low-frequency and high-frequency components in heart rate variability and calculate the ratio of low-frequency to high-frequency components to determine the level of sympathetic nerve activity.

[0116] Real-time tracking of blood oxygen saturation levels ensures that blood oxygen saturation monitoring remains within a safe and effective range;

[0117] Acquire skin conductance monitoring data, and use changes in skin resistance to reflect the user's stress response and help indicate the activity of the sympathetic nervous system;

[0118] The real-time monitored brainwave proportions, low-frequency to high-frequency component ratios, blood oxygen saturation values, and skin conductance values ​​are converted into fuzzy linguistic variables and divided into fuzzy subsets.

[0119] Based on the physiological mechanisms of sleep, targeted regulation rules are set, and a fuzzy rule base is constructed.

[0120] Output layer mapping is performed to transform the fuzzy rule inference results into specific trimodal parameter adjustment values, ensuring that the adjustment instructions can directly drive hardware execution.

[0121] For example, a physiological indicator collection system can be established to obtain a data foundation for dynamic regulation. In EEG monitoring, the focus can be on collecting data on the proportions of alpha waves (8-12Hz, a marker of wakefulness / light sleep), delta waves (0.5-4Hz, a marker of deep sleep), and theta waves (4-7Hz, a marker of light sleep transition) to determine the target user's current sleep stage (e.g., alpha wave proportion > 50% indicates wakefulness, and increased delta wave proportion indicates deep sleep). In heart rate variability (HRV) monitoring, the low-frequency component (LF, an indicator of sympathetic nerve activity) and high-frequency component (HF, an indicator of parasympathetic nerve activity) in HRV can be analyzed, and the ratio of low-frequency to high-frequency components can be calculated (a ratio > 3 indicates sympathetic nerve activity, possibly accompanied by anxiety and difficulty falling asleep). In blood oxygen saturation (SpO2) monitoring, the level of oxygen saturation in the blood can be tracked in real time to ensure that blood oxygen saturation is maintained within a safe and effective range (target ≥ 95%, avoiding hypoxia or hyperoxygenation). In terms of skin conductance (SC) monitoring, changes in skin resistance can reflect a user's stress response (elevated skin conductance values ​​suggest sympathetic nerve excitation, which may indicate sleep disturbance factors).

[0122] For example, a fuzzy control model is established to construct the correlation logic between physiological indicators and trimodal parameters. During the definition of the model input layer, the real-time monitored EEG proportions, the ratio of low-frequency to high-frequency components, blood oxygen saturation, and skin conductance are converted into fuzzy linguistic variables (such as "low alpha wave proportion," "high ratio of low-frequency to high-frequency components," "normal blood oxygen saturation," and "high skin conductance"), and fuzzy subsets are defined (such as "high / medium / low" and "strong / medium / weak"). During the construction of the fuzzy rule base, targeted control rules are set according to the physiological mechanisms of sleep. For example, if the EEG shows "alpha wave proportion < 50%" (indicating a conscious state with strong arousal signals), the rule "increase pulse intensity + decrease oxygen concentration" is triggered; if the heart rate variability shows "low-frequency to high-frequency component ratio > 3" (indicating sympathetic nerve activity and anxiety), the rule "delay the phase difference between sound waves and pulses + gradually restore synchronous stimulation" is triggered. At the output layer mapping level, the fuzzy rule reasoning results are transformed into specific three-modal parameter adjustment values ​​(such as "pulse intensity + 20% current", "oxygen concentration reduced to 25%", "phase difference shifted from 0° to 30°"), ensuring that the adjustment commands can directly drive the hardware to execute.

[0123] For example, based on the output of the fuzzy control model, stimulation parameters are optimized in real time, and trimodal parameters are dynamically adjusted. During the adjustment process for "awake state (alpha wave proportion < 50%)", the pulse intensity of low-frequency pulses can be automatically increased (current increased by 20%) to enhance stimulation of the parasympathetic nervous system and strengthen arousal inhibition. Simultaneously, the oxygen concentration is reduced to 25%, promoting increased cerebral blood flow through mild hypoxia stimulation (non-pathological level), thus enhancing the resonance effect between sound waves and nerves. During the adjustment process for "sympathetic nervous system activity (ratio of low-frequency components to high-frequency components > 3)", the phase difference between sound wave stimulation and low-frequency pulses can be delayed (shifted from 0° to 30°). This disrupts neural synchronicity and breaks the anxiety cycle. Once the ratio of low-frequency components to high-frequency components returns to the normal range (< 2), the synchronous phase lock is gradually restored. In the process of regulating "sleep state fluctuations (such as a decrease in the proportion of delta waves and an increase in skin conductance)," for low-frequency pulses, the frequency can be appropriately increased (e.g., from 1Hz to 1.5Hz) to maintain mild neural inhibition; for sound wave stimulation, the proportion of theta wave (4-7Hz) components can be increased to stabilize the light sleep state; at the same time, the oxygen concentration (±2%) or the chamber pressure (±0.05ATA) can be finely adjusted to ensure that the blood oxygen saturation is stable at 95%-97% and to avoid fluctuations in physiological indicators from interfering with sleep.

[0124] This application provides a method for controlling an oxygen chamber that uses sound waves and low-frequency pulses to promote sleep. Based on the trans-diurnal rhythm of the human sleep cycle, a basic cycle phase of the oxygen chamber is established, and the core physiological goals of this basic cycle phase are determined. According to the core physiological goals of the basic cycle phase, the timing of low-frequency pulses, sound wave stimulation, and oxygen pressurization is matched. Based on the organ nerve sensory partitions corresponding to the low-frequency pulses, sound wave stimulation, and oxygen pressurization, neural conduction pathway matching and spatiotemporal synchronization of organ responses are performed. A fuzzy control model is established, and multiple parameters are monitored in real time. Modal parameters are dynamically adjusted based on monitoring data of human physiological indicators. Upper limits for pulse intensity, sound wave decibels, oxygen concentration, and single pressurization duration are set, and oxygen chamber operation control is executed according to the modal parameters. The technical solution of this application has significant advantages in physiological adaptation, precise coordination, individual adaptation, and safety assurance. By dividing the sleep into "sleep induction period, deep sleep period, and sleep maintenance period" based on the human sleep-prone circadian rhythm, and clarifying the core physiological goals of each stage, the oxygen chamber regulation is made to perfectly match the EEG characteristics and physiological transition patterns of natural sleep. This avoids the "blind stimulation" problem caused by traditional sleep aids being out of sync with the human sleep cycle, laying a physiologically adapted foundation for efficient sleep aids. Regarding the precision of multimodal synergy, the timing of low-frequency pulses, sound wave stimulation, and oxygen pressurization is matched in stages. For example, during the sleep induction period, a 1Hz pulse and a sound wave of the same frequency are synchronized to suppress alpha waves; during deep sleep, a 2.5Hz pulse and theta wave sound waves complement each other. This not only specifically enhances the arousal inhibition effect at each stage (such as promoting delta wave generation and preventing sudden awakening from deep sleep), but also avoids the neurological adaptive fatigue caused by a single stimulation mode, significantly improving sleep aid efficiency and sleep continuity. In terms of synergistic action pathways, the system achieves neural conduction pathway matching based on organ-neural sensory partitions (e.g., pulses along the spinothalamic tract, sound waves through the auditory-thalamic pathway, and oxygen delivery to the carotid body), and synchronizes organ responses during the peak and interval phases of the pulse (e.g., instantaneous synergy of "electrical stimulation-sound vibration-blood oxygen surge"), forming a cross-level inhibitory network of "peripheral nerve-auditory system-cardiovascular system → brainstem-cortex". This addresses the pain points of traditional techniques, such as dispersed multimodal signal transmission and poor synergy, significantly enhancing the targeted inhibitory effect on the arousal center. Regarding individual adaptability and dynamic adjustment capabilities, the fuzzy control model, combined with real-time monitoring of multiple parameters such as EEG and heart rate variability, can dynamically adjust the three-modal parameters according to the user's physiological indicators (e.g., alpha wave proportion, LF / HF ratio). This adapts to individual differences among users of different ages and sleep disorder types, while avoiding "overstimulation" (e.g., high oxygen concentration, strong pulses) or "insufficient stimulation," achieving personalized and refined sleep-aid regulation.From a safety perspective, safety thresholds are clearly defined for pulse intensity (<5mA), sound decibels (<60dB), oxygen concentration (≤40%), and single pressurization duration (<120 minutes). Combined with hardware redundancy and software watchdog programs, physiological risks such as oxygen poisoning, hearing damage, and nerve overstimulation are effectively avoided, ensuring the sleep aid effect while providing users with a safe and reliable user experience.

[0125] It should be noted that step designations such as S10 and S20 are used in this application for the purpose of more clearly and concisely describing the corresponding content, and do not constitute a substantial limitation on the order. In specific implementation, those skilled in the art may execute S20 first and then S10, etc., but these should all be within the protection scope of this application.

[0126] In the embodiments of the apparatus and storage medium provided in this application, all the technical features of any of the above-described method embodiments may be included. The extended and explanatory content of the specification is basically the same as that of the embodiments of the above methods, and will not be repeated here.

[0127] This application also provides a computer program product, which includes computer program code. When the computer program code is run on a computer, it causes the computer to perform the methods described in the various possible implementations above.

[0128] This application also provides a chip, including a memory and a processor. The memory is used to store a computer program, and the processor is used to call and run the computer program from the memory, so that a device with the chip installed performs the methods described in the various possible implementations above.

[0129] It is understood that the above scenarios are merely examples and do not constitute a limitation on the application scenarios of the technical solutions provided in the embodiments of this application. The technical solutions of this application can also be applied to other scenarios. For example, as those skilled in the art will know, with the evolution of device architecture and the emergence of new business scenarios, the technical solutions provided in the embodiments of this application are also applicable to similar technical problems.

[0130] The sequence numbers of the embodiments in this application are for descriptive purposes only and do not represent the superiority or inferiority of the embodiments.

[0131] The steps in the method of this application embodiment can be adjusted, combined, or deleted according to actual needs.

[0132] The units in the device of this application embodiment can be merged, divided, and deleted according to actual needs.

[0133] In this application, the same or similar terms, concepts, technical solutions and / or application scenario descriptions are generally described in detail only when they appear for the first time. When they appear again, they are generally not repeated for the sake of brevity. When understanding the technical solutions and other contents of this application, the same or similar terms, concepts, technical solutions and / or application scenario descriptions that are not described in detail later can be referred to their previous relevant detailed descriptions.

[0134] In this application, the descriptions of the various embodiments have different focuses. For parts that are not described in detail or recorded in a certain embodiment, please refer to the relevant descriptions of other embodiments.

[0135] The technical features of the present application can be combined in any way. For the sake of brevity, not all possible combinations of the technical features in the above embodiments are described. However, as long as there is no contradiction in the combination of these technical features, they should be considered to be within the scope of the present application.

[0136] The above are merely preferred embodiments of this application and do not limit the scope of this application. Any equivalent structural or procedural transformations made based on the description and drawings of this application, or direct or indirect applications in other related technical fields, are similarly included within the scope of protection of this application.

Claims

1. A method for controlling an oxygen chamber to promote sleep through the synergistic effect of sound waves and low-frequency pulses, characterized in that, include: Based on the super-diurnal rhythm of the human sleep cycle, the basic cycle phase of the oxygen chamber is set, and the core physiological objectives of the basic cycle phase are determined. Based on the core physiological goals of the aforementioned basic cycle phase, the timing of low-frequency pulses, sound wave stimulation, and oxygen supply pressurization is matched accordingly. Based on the organ nerve sensory partitions corresponding to the low-frequency pulse, sound wave stimulation and oxygen supply pressurization, nerve conduction pathway matching and organ response spatiotemporal synchronization are performed. A fuzzy control model is established, and multiple parameters are detected in real time. The modal parameters are dynamically adjusted based on the monitoring data of human physiological indicators. Set the upper limit of pulse intensity, upper limit of sound decibel, upper limit of oxygen concentration, and duration of single pressurization, and execute oxygen chamber operation control according to the modal parameters.

2. The oxygen chamber control method for synergistic sleep aid using sound waves and low-frequency pulses according to claim 1, characterized in that, The process of establishing the basic cycle phase of the oxygen chamber based on the hyperrheic rhythm of the human sleep cycle, and determining the core physiological goals of the basic cycle phase, includes: Using the super-diurnal rhythm of the human sleep cycle as the underlying time reference, the core time frame and stage dimensions are determined. Based on the aforementioned core time frame and stage dimensions, the core physiological goals for each stage are defined according to the characteristics of human sleep.

3. The oxygen chamber control method for synergistic sleep aid using sound waves and low-frequency pulses according to claim 2, characterized in that, The process of defining the core physiological goals for each stage based on the core time frame and stage dimensions, and according to the characteristics of human sleep, includes the following: The basic cycle phases of the oxygen chamber are set as the sleep induction period, the deep sleep period, and the sleep maintenance period; During the sleep induction period, the brain's arousal signals are suppressed, the parasympathetic nervous system is activated, and blood oxygen saturation is increased. During the deep sleep period, the brainstem's inhibition of the brain's arousal signals is enhanced, promoting the generation of deep sleep characteristic waves and stabilizing cerebral blood flow. During the sleep maintenance period, mild neural inhibition is maintained to prevent sudden awakening from deep sleep and to simulate the mechanical stimulation of natural breathing.

4. The oxygen chamber control method for synergistic sleep aid using sound waves and low-frequency pulses according to claim 3, characterized in that, The process of timing-matching low-frequency pulses, sound wave stimulation, and oxygen pressurization according to the core physiological goals of the basic cycle phase includes: Based on the core physiological goals of each stage, the key physiological characteristics corresponding to each stage are determined. Based on the aforementioned key physiological characteristics, the control timing of the low-frequency pulse, sound wave stimulation, and oxygen supply pressurization is set respectively.

5. The oxygen chamber control method for synergistic sleep aid using sound waves and low-frequency pulses according to claim 4, characterized in that, Based on the aforementioned key physiological characteristics, the process of setting the control timing of the low-frequency pulse, sound wave stimulation, and oxygen supply pressurization, and the preceding steps, include: The key physiological characteristics of the sleep induction period are set as follows: transitioning from a waking state to a light sleep state, with a gradual decrease in heart rate, and achieving a target blood oxygen saturation of 95%. During the sleep induction period, 0.5-2Hz pulse stimulation is initiated to reduce the heart rate. Simultaneously, sound waves of the same frequency as the low-frequency pulse are released, and the fundamental frequencies of the low-frequency pulse and the sound wave stimulation are strictly aligned to form phase lock, establishing synergy between the auditory cortex and the pulse signal. At the same time, the pressure inside the oxygen chamber is gradually increased from atmospheric pressure to 1.2 ATA, and the oxygen concentration is simultaneously increased to 30%-40%. And / or, The key physiological characteristics of the deep sleep period are set to simulate the brainwave transition process from shallow to deep sleep in natural sleep, while controlling the risk of oxygen exposure. During the deep sleep period, the pulse frequency is increased to 2-3Hz, the main frequency of the sound wave is adjusted to be the same as the pulse frequency, and 4-7Hz theta wave sound waves are inserted during the pulse interval. The cabin pressure is stabilized at 1.3ATA, and the oxygen concentration is dynamically adjusted through blood oxygen monitoring feedback. And / or, The key physiological characteristics of the sleep maintenance period are set to match the EEG characteristics of REM sleep. The air pressure and blood oxygen gradually drop to near normal pressure levels, and the chest and abdomen rise and fall are simulated by micro-pressure difference changes. During the sleep maintenance period, the pulse is switched to 0.5-1Hz, while the intensity is reduced to half of the initial value. A mixed sound wave of 1-4Hz is released, the cabin pressure gradually drops to 1.1ATA, the oxygen concentration drops to 25%, and a brief negative pressure is introduced during the pulse interval.

6. The oxygen chamber control method for synergistic sleep aid using sound waves and low-frequency pulses according to claim 1, characterized in that, The process of matching neural conduction pathways and synchronizing organ responses in spatiotemporal mode based on the organ nerve sensory regions corresponding to the low-frequency pulses, sound wave stimulation, and oxygen supply pressurization includes: Based on the corresponding organ nerve sensory zones of low-frequency pulses, sound wave stimulation, and oxygen supply pressurization, conduction pathways are designed and targeted conduction pathways are planned so that the signals from multiple pathways can ultimately work synergistically to the relevant nerve centers. Based on the neural conduction pathway matching and combined with the signal conduction speed of each stimulation mode, the output rhythm of each stimulus is synchronously regulated during the peak pulse period and the inter-pulse period, so that the organ response is coordinated in time and space.

7. The oxygen chamber control method for synergistic sleep aid using sound waves and low-frequency pulses according to claim 6, characterized in that, The process of designing conduction pathways and planning targeted conduction pathways based on the corresponding organ nerve sensory regions of low-frequency pulses, sound wave stimulation, and oxygen supply pressurization, so that the multi-path signals ultimately work synergistically to the relevant nerve centers, includes: The conductive pads on the armrests of the oxygen chamber seats correspond to the median and ulnar nerves of the arm. When the low-frequency pulses released by the conductive pads stimulate the median and ulnar nerves, the signals are transmitted along the spinothalamic tract to the brainstem reticular formation, directly inhibiting the ascending activating system and achieving targeted transmission from the peripheral nerves to the brainstem. And / or, Air conduction corresponds to the external auditory canal to the cochlear hair cells, while bone conduction corresponds to the skull in contact with the occiput to the cochlea. Air conduction allows sound waves to activate the cochlear hair cells via the external auditory canal, with the signal transmitted from the brainstem auditory center to the thalamus. Bone conduction allows sound waves to directly stimulate the cochlea through skull vibration, with the signal transmitted along the same path. After the two signals are integrated in the auditory cortex, they are transmitted through the thalamus and cortical pathways, resonating with the slow waves induced by low-frequency pulses in the brainstem reticular formation, thus achieving the coordinated transmission of sound wave signals to the brainstem. And / or, The increased oxygen partial pressure stimulates the chemoreceptors in the carotid body, causing the stimulation signal to be transmitted through the sinus nerve to the glossopharyngeal nerve and then to the medullary respiratory center. This reflexively reduces the excitability of the medullary respiratory center and indirectly inhibits the arousal drive of the brainstem reticular formation. This, along with the conduction pathways of low-frequency pulses and sound waves, forms a supplementary transmission of peripheral chemoreception to the brainstem.

8. The oxygen chamber control method for synergistic sleep aid using sound waves and low-frequency pulses according to claim 7, characterized in that, The process of coordinating organ responses in a spatiotemporal manner by matching neural conduction pathways and combining the signal conduction velocity of each stimulus modality, synchronously regulating the output rhythm of each stimulus during the peak pulse period and the interpulse period, thereby achieving spatiotemporal coordination of organ responses, includes: When the low-frequency pulse is at its peak, the sound wave generator releases the sound wave peak to synchronously trigger the oxygen supply system to instantly increase the flow rate, forming an instantaneous synergistic effect to enhance the inhibition of the locus coeruleus and achieve spatiotemporal synchronization of organ response during the pulse peak period. When the low-frequency pulse is in a resting state, the sound wave stimulation is switched to the 4-7Hz theta wave frequency band, and the oxygen supply mode is simultaneously adjusted to intermittent pulse oxygen supply. This stimulates the lung stretch receptors through the micro-pressure difference in the chamber, enhances vagal nerve tone, and works with the theta wave sound wave to maintain the stable response of the organs, achieving spatiotemporal synchronization of organ response during the pulse interval.

9. The oxygen chamber control method for synergistic sleep aid using sound waves and low-frequency pulses according to claim 1, characterized in that, The process of establishing a fuzzy control model, performing real-time multi-parameter detection, and dynamically adjusting modal parameters based on monitoring data of human physiological indicators includes: Establish a physiological indicator collection system to conduct real-time monitoring of multiple parameters and obtain data foundation for dynamic regulation; Construct the correlation logic between physiological indicators and multimodal parameters, and establish a fuzzy control model; The stimulation parameters are optimized in real time based on the output of the fuzzy control model.

10. The oxygen chamber control method for synergistic sleep aid using sound waves and low-frequency pulses according to claim 9, characterized in that, The process of constructing the correlation logic between physiological indicators and multimodal parameters, and establishing the fuzzy control model, includes the following prior steps: Collect data on the proportion of alpha, delta, and theta waves in the brain to determine the current sleep stage of the target user; Analyze the low-frequency and high-frequency components in heart rate variability and calculate the ratio of low-frequency to high-frequency components to determine the level of sympathetic nerve activity. Real-time tracking of blood oxygen saturation levels ensures that blood oxygen saturation monitoring remains within a safe and effective range; Acquire skin conductance monitoring data, and use changes in skin resistance to reflect the user's stress response and help indicate the activity of the sympathetic nervous system; The real-time monitored brainwave proportions, low-frequency to high-frequency component ratios, blood oxygen saturation values, and skin conductance values ​​are converted into fuzzy linguistic variables and divided into fuzzy subsets. Based on the physiological mechanisms of sleep, targeted regulation rules are set, and a fuzzy rule base is constructed. Output layer mapping is performed to transform the fuzzy rule inference results into specific trimodal parameter adjustment values, ensuring that the adjustment instructions can directly drive hardware execution.

Images