Dual-epitope car and dual-epitope car-t cell targeting CLL1, and use thereof
By designing a dual-epitope chimeric antigen receptor (CAR) targeting CLL1 and combining the co-stimulatory signal transduction domain and the chimeric switch receptor, the problems of bone marrow failure and immunosuppression of CAR-T cells in the treatment of AML are solved, and efficient killing and inhibition of CLL1-positive tumor cells are achieved, which has broad clinical application prospects.
Patent Information
- Application Number
- PCT/CN2025/091697
- Authority / Receiving Office
- WO · WO
- Patent Type
- Applications
- Current Assignee / Owner
- Priority Date
- 2024-03-14
- Filing Date
- 2025-04-28
- Publication Date
- 2025-09-18
AI Technical Summary
Existing CAR-T cell therapy in the treatment of AML has problems such as differences in the properties of myeloid antigens leading to bone marrow failure and a lack of alternative treatments. In addition, CAR-T cells that single-target tumor antigens have limited efficacy in AML and are difficult to effectively eradicate tumor cells.
A dual-epitope chimeric antigen receptor (CAR) was designed, which contains two single-domain antibodies and a transmembrane domain targeting CLL1, combined with a co-stimulatory signaling domain, and connected to the chimeric switch receptor PD-1 & CD27 through a self-cleaving peptide to form a dual-epitope CAR-T cell, which enhances the killing activity against CLL1-positive tumor cells and resists the immunosuppressive microenvironment.
Dual-epitope CAR-T cells significantly enhanced their ability to kill CLL1-positive tumor cells, secreted IFN-γ, inhibited tumor growth, and prolonged survival in mouse models. They had good in vivo tumoricidal activity and durability, and could effectively treat CLL1-positive cancers such as AML, MDS, and CML.
Abstract
Citation Information
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