Muscle relaxation device and related methods of use

Abstract

A muscle relaxation device comprising a driving mechanism (3) to induce muscle relaxation in a subject, and a support section (4) attached to said driving mechanism (3) that holds at least one body part of said subject such as the heel, characterized in that the driving mechanism (3) is configured to move at least said body part and optionally at least another body part; said driving mechanism (3) being furthermore configured to induce an oscillatory motion applied to the subject, and wherein said oscillatory motion is applied at a speed of 5.0 m / min or less and / or at 63 rpm or less. The invention also relates to a combination using the said device with a resting surface (1, 2, 7, 8, 9) and to methods using the said device.

Description

[0001] P4308PC00 / 1547-001 dpt

[0002] Muscle relaxation device and related methods of use Field of the Invention

[0003] The present invention relates to a muscle relaxation device and to different methods using such a device. More specifically, it concerns a device that directly and / or indirectly may move tense muscles through passive methods, inducing activity in the afferent and efferent nerves of the targeted muscles to facilitate muscle relaxation.

[0004] Background of Invention

[0005] In modern society, prolonged sedentary work, improper biomechanical use of the body, bad habits, stress, and accumulated fatigue are common. These factors contribute to chronic muscle tension, which in turn can exert pressure on nerves, hinder blood and lymphatic circulation, and lead to postural imbalances. Over time, such conditions may cause muscular pain and chronic disorders, including persistent fatigue and sleep disturbances.

[0006] Chronic muscle tension accumulates throughout a person’s lifetime. While most acute muscle tension is naturally relieved over time, a small fraction of it develops into chronic tension without being noticed in daily life.

[0007] Chronic muscle tension is fundamentally different from simple muscle fatigue or transient muscle contraction. The “chronically stiff muscles” and “loss of tissue elasticity” observed in many clinical conditions do not represent an actively contracting muscle state. Rather, they arise as a result of maladaptive learning within the body’s overall control system, in which muscle tension is persistently maintained and progressively fixed over time.

[0008] From a physiological perspective, chronic muscle tension constitutes a complex condition composed of multiple interacting layers. First, at the level of the brain and spinal cord, neural circuits related to postural control and protective reflexes become re-learned, leading to the automatic and continuous transmission of tension commands to specificP4308PC00 / 1547-001 dpt

[0009] muscle groups. As a consequence, muscle spindle thresholds are reset toward a shortened state, making voluntary relaxation difficult or impossible.

[0010] Second, when neurogenic tension persists over prolonged periods, intracellular calcium ions (Ca2+) are no longer fully re-sequestered within muscle fibers, accompanied by ATP depletion and impaired function of the sarcoplasmic reticulum (SR). This results in partial maintenance of actin-myosin cross-bridges, shifting the muscle into a semi-contracted state that closely resembles physiological contracture.

[0011] Third, sustained contraction compresses capillary networks, inducing local ischemia and hypoxia. This impairs metabolic recovery and promotes the accumulation of metabolic byproducts and energy deficiency, thereby establishing a self-reinforcing vicious cycle that further perpetuates muscle tension.

[0012] With further progression, portions of muscle fibers undergo degeneration or loss, triggering fibroblast activation during the repair process and excessive deposition of collagen-rich connective tissue. This process, known as muscular fibrosis, replaces contractile tissue with structurally rigid material, resulting in a marked loss of elasticity. In severe cases, dystrophic calcification may occur, characterized by calcium salt deposition within degenerated tissue, producing palpably “bone-like” hardness. At this stage, tissue reversibility is significantly diminished.

[0013] As the volume of accumulated chronic muscle tension increases over time, it can compress nerves and disrupt blood circulation within and around muscles, potentially leading to common chronic conditions such as lower back pain, knee pain, neck pain, and headaches.

[0014] Chronic lower back pain is one of the most prevalent musculoskeletal disorders worldwide, affecting approximately one in five adults. It is particularly common in individuals over the age of 60.

[0015] Chronic knee pain and osteoarthritis (OA) are increasing globally due to aging populations, with an estimated 250 million people affected by OA. This condition is more prevalent in individuals over 40 years old, with a higher incidence in women than in men. Chronic knee pain significantly impacts mobility and daily life and often requires surgery or prolonged rehabilitation.P4308PC00 / 1547-001 dpt

[0016] Chronic neck pain is also widespread, with approximately 30% of adults experiencing it. With the increasing use of computers and smartphones in modern society, neck pain is becoming more common among younger individuals.

[0017] Chronic headaches affect about 15% of the global population, including migraines and tension-type headaches. According to the WHO, migraines are more common in women, particularly among working-age individuals between 20 and 50 years old, contributing to reduced work productivity and absenteeism.

[0018] Overall, chronic pain conditions, including lower back pain, knee pain, neck pain, and headaches, affect hundreds of millions of people worldwide. Although not directly lifethreatening, they significantly reduce quality of life, impair work productivity, and increase medical expenses. With aging populations and increasing digitalization, the burden of these conditions is expected to rise, emphasizing the need for effective medical treatments and preventive health management.

[0019] One major contributing factor to these chronic conditions is the accumulation of muscle tension, which affects overall blood circulation and triggers pain.

[0020] It is known that chronic muscle tension stimulates the sympathetic nervous system, exacerbating poor circulation and creating a vicious cycle of chronic pain. Muscle tension reduces blood flow, leading to oxygen deficiency and the accumulation of metabolic waste products, which cause pain. This pain further activates the sympathetic nervous system, leading to vasoconstriction and worsening circulation, perpetuating the pain cycle.

[0021] Normally, sympathetic activation prioritizes blood flow to skeletal muscles. However, prolonged muscle tension and excessive sympathetic stimulation can cause localized ischemia. This issue is particularly problematic in areas prone to chronic tension, such as the neck, shoulders, and back.

[0022] To break this cycle, muscle relaxation is a fundamental solution, and various methods have been proposed.P4308PC00 / 1547-001 dpt

[0023] Physiotherapy and massage are commonly used to relax muscles, providing some degree of relief. However, they still face challenges in effectively addressing chronic muscle tension.

[0024] US patent 5,107,822 describes a device that applies a twisting and swaying motion to the abdomen while the user lies on their back. By swinging the ankles, the device transmits movement to the abdomen, aiming to stimulate internal organ function, enhance oxygen intake, and promote overall health. Specifically, it consists of an ankle rest that moves laterally with an amplitude of 20-40 mm at a speed of 100-200 cycles per minute(rpm) via a motor. This motion induces a twisting and swaying effect on the user's abdomen and legs.

[0025] This device is advantageous as it does not require intense physical activity like jogging or aerobics, making it accessible to individuals with low physical fitness. However, while it temporarily improves circulation, it does not effectively relieve chronic muscle tension and does not provide a fundamental solution.

[0026] US patent 11,207,238 describes a method and system for muscle relaxation. This method involves applying pressure to specific skin areas using fingers or tools while the muscle is contracting and extending. The applied pressure ranges from 1 kgf to 100 gf over an area of 2 cm2to 0.1 cm2.

[0027] By allowing either the patient or a practitioner to apply pressure, this technique aims to promote muscle relaxation, particularly in the lower back. Properly managing pressure intensity and application area is expected to facilitate the removal of waste products and excess calcium, thereby relieving muscle tension.

[0028] However, this method has limitations: it requires the patient to actively move their body and demands a high level of skill from the practitioner. Additionally, it does not provide specific explanations or proven effects for addressing chronic muscle tension throughout the body.

[0029] US patent application 2004 / 102724 discloses a device that provides passive exercise. This device supports the user's limbs and lower back and applies controlled movements through motor-driven mechanisms to promote blood circulation and assist in muscle relaxation. It is intended for use in rehabilitation and health promotion, particularly forP4308PC00 / 1547-001 dpt

[0030] elderly individuals or users with reduced physical ability, providing a form of exercise with minimal physical strain.

[0031] However, in the technical field related to the present invention, while it is important to alleviate chronic muscle tension, the technology described in this prior art, although expected to temporarily improve blood flow by oscillation, does not propose a fundamental solution to chronic muscle tension. Additionally, there is no specific description regarding the operating speed of the device, and the conditions necessary to optimize the effectiveness of the exercise are not explicitly stated. Consequently, if the operating speed is not appropriately set, the desired effects may not be adequately achieved.

[0032] Also, muscle relaxants and botulinum toxin (Botox), which are widely used in existing medical approaches, are both methods that induce temporary muscle relaxation by suppressing or blocking the neural signals responsible for generating muscle tension.

[0033] Muscle relaxants primarily reduce neural activity at the level of the central nervous system or spinal reflex pathways, whereas botulinum toxin inhibits the release of acetylcholine at the neuromuscular junction, thereby rendering muscle contraction physically impossible. Through these interventions, further release of calcium ions (Ca2+) is suppressed, and existing actin-myosin cross-bridges are temporarily released, resulting in muscle relaxation.

[0034] However, both approaches are fundamentally focused on interrupting the neural commands that produce muscle tension and do not address the underlying mechanisms by which chronic tension is formed and maintained. These mechanisms include maladaptive learning within neural circuits, altered muscle spindle threshold settings, structural changes in fascia and connective tissue, and persistent alterations in local blood flow patterns. Consequently, once the pharmacological effects diminish, the original tension patterns are likely to re-emerge, limiting the potential for sustained, long-term improvement.

[0035] In this sense, these interventions should be understood as techniques that temporarily suppress muscle tension rather than methods that reconstruct a physiological state in which chronic muscle tension does not arise.P4308PC00 / 1547-001 dpt

[0036] As described above, chronic muscle tension represents a state that is memorized and stabilized across multiple layers, including neural control, physiological processes, circulation, fascial systems, and structural tissue adaptations. For this reason, single-mode interventions such as pharmacological treatment, neural signal blockade, or force-based stretching are insufficient to achieve a fundamental resolution.

[0037] Considering the nature of chronic muscle tension, previous studies (Non-Patent Literature 1 and Non-Patent Literature 2) have discussed a cross-bridge-based mechanism in which a small number of attached cross-bridges can persist in relaxed muscle. The formation of such cross-bridges has been reported to depend on intracellular calcium levels and prior mechanical history. The extent of residual cross-bridges influences the resistance encountered during slow passive muscle stretching. However, these studies primarily focus on short-range mechanical behavior observed during transient passive stretching and do not address persistent, layered muscle tension maintained by long-term postural and neuromuscular adaptation.

[0038] Non-Patent Literature 1: Campbell, K. S., Lakie, M., " A cross-bridge mechanism can explain the thixotropic short-range elastic component of relaxed frog skeletal muscle", Journal of Physiology, 1998.

[0039] Non-Patent Literature 2: Proske, U., Morgan, D. L., " Do cross-bridges contribute to the tension during stretch of passive muscle?", Journal of Muscle Research and Cell Motility, Vol. 20, pp. 433-442, 1999.

[0040] Description of the Invention

[0041] In light of the aforementioned issues the present invention proposes a muscle relaxation device and related methods that effectively alleviate chronic muscle tension.

[0042] The inventor has identified that muscle tension can be categorized into two primary phases: one is acute muscle tension, which typically recovers within a few days, and the other is chronic muscle tension, which develops when minor acute muscle tensions accumulate due to repetitive movements or prolonged static postures. Additionally, chronic muscle tension follows a specific neuromuscular feedback loop. When a muscle experiences excessive tension or a painful stimulus, the brain perceives it as a threat, triggering a neuromuscular response that stiffens the muscle as a protective mechanism. This creates a vicious cycle, further exacerbating the condition over time.P4308PC00 / 1547-001 dpt

[0043] Since chronic muscle tension is ultimately maintained under neural control by the brain and spinal cord, the invention proposes a process in which the release of this neural control shifts the muscle state from chronic to acute, allowing the muscle tension to gradually diminish. To simplify the evaluation of chronic muscle relaxation, this invention utilizes changes in height before and after the passive movement of a body part. For effective relaxation of chronic muscle tension, it is crucial to trigger the spinal cord’s inhibitory functions, which facilitate muscle relaxation, without provoking defensive reflexes from external stimuli regulated by the brain and spinal cord. This aspect plays a key role in evaluating the effectiveness of the proposed method.

[0044] Specifically, chronic muscle tension not only affects the muscle itself but also leads to a shortening of specific muscles, while their antagonistic muscles remain in an extended state. This imbalance influences tendons, fascia, and even the skin at the muscle-skeletal junctions. Chronic muscle tension forms a network throughout the body, which aligns with the concept of myofascial connections, such as those described in Anatomy Trains. These interconnected fascial structures influence skeletal alignment and ultimately affect posture.

[0045] Given that chronic muscle tension impacts the entire body, it is essential to relieve muscle tension on a global scale. By passively moving a specific body part, the resistance encountered reflects the level of tension across the interconnected muscle network. This principle provides a way to evaluate the severity of chronic muscle tension. The measured resistance value includes frictional and shear forces that occur between the moving body part and the surface where the subject is lying.

[0046] In the present invention, it is preferable to select conditions where the resistance value generated by passively moving a part of the body is high, within a range that does not cause pain or discomfort. When a body part is moved passively, the body weight of the lying subject generates a reaction force from the flat space, linear, and / or point-shaped support structures, which in turn applies pressure to the muscles, resulting in resistance.

[0047] Furthermore, to relieve chronic muscle tension throughout the body, the power generated by passively moving a part of a limb, such as the heel, must be transmitted across the entire body to the head as well as the arms and hands. Therefore, except for the portion of one or several limbs being passively moved, the entire body weight should be rested on the resting surface.P4308PC00 / 1547-001 dpt

[0048] The present invention therefore concerns a muscle relaxation device comprising a driving mechanism to induce muscle relaxation in a subject, and a support section attached to said driving mechanism that holds at least one body part of said subject such as the heel, characterized in that the driving mechanism is configured to move at least said body part and optionally at least another body part; said driving mechanism being furthermore configured to induce an oscillatory motion applied to the subject, and wherein said oscillatory motion is applied at a speed of 5.0 m / min or less and / or at 63 rpm or less.

[0049] The device is preferably configured to be used with a resting surface that supports a subject in a rest position. To have the entire body in contact with the resting surface the distance between the resting surface and the support section should be as small as possible.

[0050] As used herein, a "resting surface" refers to an area configured to support a subject in a rest position, and may include substantially any surface or structure on which the subject can lie or recline, including, but not limited to, a mattress, a chair, a bench, or a floor of a room.

[0051] The present invention is based on the hypothesis that muscles and tendons inherently contain sensors, known as muscle spindles and Golgi tendon organs, which prevent excessive stretching and contraction. This invention utilizes these natural biological mechanisms for muscle relaxation. Muscle spindles, which are located within the muscle fibers, detect changes in muscle length and regulate tension through the stretch reflex. When a muscle is rapidly stretched, the muscle spindle reacts strongly by triggering a reflexive contraction to prevent overstretching. In contrast, when a muscle is slowly and passively stretched, the spindle’s response is more subdued, allowing the muscle to gradually relax.

[0052] Golgi tendon organs, which are located in tendons, detect changes in muscle tension and regulate force through the inverse stretch reflex. When excessive tension is detected, these sensors send inhibitory signals to relax the muscle, reducing the risk of injury. They also help distribute muscular force more evenly to prevent localized overstrain. Since these mechanisms are complementary, they help regulate muscle tone and maintain a balance between contraction and relaxation.

[0053] Additionally, human physiology includes spinal reflex mechanisms that facilitate muscle relaxation. Reciprocal inhibition occurs when the contraction of one muscle automatically relaxes its antagonist muscle, ensuring smooth motion. Autogenic inhibition occurs whenP4308PC00 / 1547-001 dpt

[0054] excessive tension within a muscle itself triggers signals to suppress contraction, reducing strain and protecting against injury. By appropriately utilizing these reflexes, the present invention facilitates gradual and controlled relaxation of chronic muscle tension.

[0055] The invention achieves muscle relaxation preferably by passively oscillating both legs. The speed of the left and right feet moved passively does not need to be the same. It can be adjusted based on resistance values measured in real-time. Resistance to this movement serves as an indicator of muscle tension. However, if the oscillation is too rapid, the stretch reflex is triggered, leading to an increase in resistance due to reflexive contractions. The stretch reflex is generally said to occur when moving at speeds faster than 18 m / min, but a much slower speed is preferable. To avoid this, the movements must be slow and controlled. The inventor has discovered that the preferred speed should be set at 5.0 m / min or lower, with a more desirable range being 2.0 m / min or lower, and the optimal speed being approximately 0.5 m / min or lower. When converted to revolutions per minute (rpm), assuming an oscillation amplitude of 6 cm, the corresponding values are 41.7 rpm or lower, with a preferred range of 16.7 rpm or lower, and an optimal value of 4.2 rpm or lower. If the oscillation amplitude is 4 cm, the corresponding values are 62.5 rpm or lower, with a preferred range of 25 rpm or lower, and an optimal value of 6.3 rpm or lower.

[0056] When the movement speed exceeds 5.0 m / min, muscle spindles might react defensively, triggering reflexive contractions. Since the stretch reflex response depends on the velocity and intensity of stimulation, it is desirable to minimize these reflexes when passively moving the body. By measuring resistance during repetitive leg oscillations, the degree of chronic muscle tension can be assessed.

[0057] The passive oscillation is not limited to the legs; it can also be applied to the arms, head, torso, or rotational movements. The preferred amplitude range for oscillatory movement is 10 cm or less, with a more desirable range being 8 cm or less, and the optimal range being 6 cm or less. The amplitude does not need to be constant. It can be adjusted based on resistance values measured in real-time. While directly moving the target muscle is preferable, indirect stimulation is also effective due to the interconnected muscle tension network, which propagates relaxation effects throughout the body. The design of the device should consider which part of the body to oscillate, with options including the pelvis or knees. Also, Passive oscillation can be generated not only by mechanical systems but also through other means, such as air-based mechanisms.P4308PC00 / 1547-001 dpt

[0058] For more details of the device, a notable whole-body muscle relaxation effect is observed despite applying only gentle, passive oscillatory movement to the heels. This phenomenon is not attributable to a localized effect on a single muscle group, but is instead understood as the result of interactions across multiple bodily layers, including myofascial continuity, biomechanics, and neural reflex regulation.

[0059] Muscles do not function as isolated units; rather, they are interconnected through the fascial system, forming a continuous tension network throughout the body. The plantar fascia, Achilles tendon, and lower-leg musculature constitute terminal structures within this network, transmitting mechanical tension toward the trunk and spinal column. The heel, positioned at the most distal and load-bearing point of this system, serves as an effective mechanical input site from which distributed forces can propagate through the body.

[0060] In the device according to the invention, linear translational motion may advantageously be applied horizontally to the heel. However, because the foot rests on the resting surface, the foot does not move purely in translation. Instead, the linear displacement of the heel is transformed into a subtle rolling and rotational motion of the foot. This conversion from linear to rotational movement induces small, distributed torsional stresses that propagate through the ankle, lower leg, pelvis, and spinal structures.

[0061] Importantly, this input does not forcibly expand joint range of motion. Rather, it delivers low-threshold mechanical fluctuations to the layered structures of muscles, fasciae, joint capsules, and ligaments. As a result, the intervention functions as an indirect, whole-body passive movement rather than a localized stretching stimulus.

[0062] A critical feature of heel oscillation is that it is entirely passive, extremely slow, and of very low intensity. Under these conditions, protective reflexes mediated by muscle spindles and nociceptors are unlikely to be triggered. Consequently, excessive excitability within spinal reflex circuits and the autonomic nervous system — mechanisms that sustain chronic muscle tension — may be attenuated. Over time, this process is hypothesized to recalibrate the neural thresholds that govern baseline muscle tone.

[0063] Rather than actively attempting to “relax” muscles, this approach allows the nervous system to relearn a state in which sustained muscular contraction is no longer required. Through repeated exposure, chronically maintained muscle-tension patterns may therefore become progressively less stable and more amenable to release.P4308PC00 / 1547-001 dpt

[0064] As mechanical displacement travels farther from the heel, both movement amplitude and tensile variation diminish. Accordingly, relaxation effects in regions such as the shoulders, arms, and neck are expected to be attenuated compared to more proximal structures. Nevertheless, owing to the continuity of the myofascial network and central regulation of global muscle tone, the effect is not eliminated and can still manifest throughout the body.

[0065] Conversely, when pronounced chronic tension is localized to a specific region, applying a similar passive movement closer to the affected area may yield a more efficient and localized relaxation response.

[0066] In summary, in the above-cited embodiment the device according to the invention does not target the heel itself as a relaxation endpoint. Instead, the heel is utilized as a safe, stable, and highly reproducible input site through which low-intensity passive motion can engage myofascial continuity and neural reflex mechanisms, thereby indirectly facilitating the reduction of chronic, whole-body muscle tension.

[0067] The present invention is designed to reset this multi-layered condition — referred to as Layered Muscle Memory — by targeting the mechanisms that sustain chronic muscle tension across neural, physiological, and structural levels. Rather than suppressing muscle tension, this method aims to enable the body to re-learn a state in which chronic tension does not arise.

[0068] Through stepwise modulation of reflex thresholds at the neural level, passive and low-intensity movement inputs that facilitate reorganization of spinal circuits, restoration of local circulation and metabolic conditions, and gradual improvement of fascial glide, this method directly intervenes in the memory structures that maintain chronic muscle tension.

[0069] Unlike pharmacological treatments or temporary physical interventions, this approach leverages the body’s inherent plasticity to establish a state in which the recurrence of excessive muscle tension is less likely.

[0070] In other words, the present invention identifies an effective approach to relieving chronic muscle tension by leveraging natural physiological reflexes. By passively inducing slow, controlled oscillatory movements, the invention minimizes stretch reflex resistance while stimulating natural inhibitory mechanisms in the spinal cord. This method provides a systematic and effective means of evaluating and alleviating chronic muscle tension throughout the body.P4308PC00 / 1547-001 dpt

[0071] The present invention provides a device for relaxing tense muscles by moving them passively, either directly or indirectly, at a speed of 5.0 m / min or less and / or at a reciprocal motion speed of 63 rpm or less. This controlled movement helps to alleviate chronic muscle tension effectively.

[0072] Brief description of figures

[0073] Figure 1 shows a muscle relaxation device according to the invention, together with a resting surface and a subject.

[0074] Figure 2 shows the muscle relaxation device of figure 1, but without the subject.

[0075] Figure 3 presents the experimental results of Example 2, indicating that changes in height are significant at low speed.

[0076] Figure 4 details a drive mechanism, a driving transmission component, a support section, and a cushioning layer.

[0077] Figure 5 illustrates the internal structure of a driving transmission component.

[0078] Figure 6 shows the structure of an aluminum bar and a EVA form used in Example 3.

[0079] Figure 7 presents the muscle relaxation device used in Example 4, which is equipped with a wave foam.

[0080] Figure 8 presents the experimental results of Example 4, showing that height changes are even more pronounced at low speed.

[0081] Figure 9 shows an example of a graph of resistance values measured over 30 seconds. From this graph, both the integral and range of the average right and left resistance values were calculated.

[0082] Figure 10 shows the results of confirming long-term postural changes.

[0083] Figure 11 presents the experimental results of Example 7, indicating that changes in height are in the different hardness of wave foam.

[0084] Figure 12 illustrates the internal structure of a mechanical movement unit.

[0085] Figure 13 Details of a driving transmission component.

[0086] Figure 14 shows schematic representation of wave foam geometry.

[0087] Figure 15 presents the experimental results of Example 8, indicating that changes in height are increased in the lower speed.

[0088] Figure 16 presents the experimental results of Example 9, indicating that changes in height are similar in the amplitude range between 1cm and 8cm.

[0089] Figure 17 presents the integration of example 2, 4 and 8.

[0090] Figure 18 illustrates the usage of a typical goldfish exercise device.P4308PC00 / 1547-001 dpt

[0091] One embodiment of the invention (see figures 1 and 2) includes a muscle relaxation device in combination with a resting surface 1,2 where the subject can lie down and a drive mechanism 3. This device moves a part of the subject's body at a speed of 5.0 m / min or less, guiding the muscles into a relaxed state. The resting surface 1,2 for the subject to lie down ensures a flat area of 190 cm in length and 70 cm in width, allowing the subject to lie on their back. The heels of both feet rest parallel on a support section 4 of the drive mechanism 3, which holds a part of the subject’s body. The drive mechanism 3 can move both feet simultaneously or only one foot at a time. By utilizing a drive mechanism 3 with a 6 cm amplitude range of motion and repeating the movement at a speed of 5.0 m / min or less, the device passively moves a part of the body, promoting muscle relaxation.

[0092] To enhance the efficiency of muscle relaxation (see figure 5), the drive mechanism consists of a driving transmission component 5 and a support section 4 that holds a part of the subject's body. The support section 4 can be equipped with a cushioning layer 6 to absorb slight resistance and / or alleviate discomfort. When resistance occurs due to the material properties or shape of the space where the subject lies, springs 5b are installed in the driving transmission component 5 to buffer this resistance. Specifically, the bottom part of the driving transmission component 5a is designed with a buffer function of approximately 15 mm on each side. Slide components 5d on an upper part of the driving transmission component 5e connect slide rails 5g placed by slide rail fixations 5f in the bottom part of the driving transmission component 5a. The movement of the slide components 5d is measured by a detector with a belt 5c through an interconnection 5h. This buffer function allows the device to detect and measure the resistance value generated during the passive motion.

[0093] The driving transmission component 5 allows relative motion between the driving mechanism and the body part such that the body part is not forcibly constrained to follow a predetermined trajectory. Instead, the body part is permitted to undergo a self-adjusting motion in response to reaction forces generated by the user's musculoskeletal system during displacement.

[0094] The driving transmission component 5 may be implemented by, for example, one or more elastic members such as springs or rubber elements, compliant interfaces, damping elements, slidable or floating couplings providing mechanical play or freedom of movement, torque limiting mechanisms, control-based force modulation including current or torque control of a drive motor, or combinations thereof.P4308PC00 / 1547-001 dpt

[0095] The driving transmission component 5 is not limited to any particular structural configuration and may be implemented independently of a support section or surface on which the user's body part is placed. However, in some embodiments, the buffering function may be at least partially realized by a compliant support section, such as a relatively thick foam member forming as a cushioning layer 6 provided at a location configured to receive the body part.

[0096] In one exemplary implementation used for evaluation, the driving transmission component may exhibit a compliance characteristic such that an applied load of approximately 1 kg results in a displacement of approximately 5 mm.

[0097] For a user having relatively low muscle tension and a body weight of approximately 60 kg, a buffering force on the order of several newtons per side may be sufficient to achieve the desired compliant response. For users having greater body weight or higher muscle tension, or when combined with a resting surface 2 such as a wave foam 9 exhibiting higher resistance, the buffering function may be configured to provide a higher force capacity and / or greater displacement to accommodate increased resistance.

[0098] To further improve the effectiveness of muscle relaxation, the invention may incorporate a process that deforms specific parts of the muscle. This is prepared by modifying the surface of the resting surface 2 where the subject lies into a linear or point-like pattern. The muscle deformation occurs simultaneously with the movement process, enhancing relaxation efficiency. A linear deformation structure can be prepared by arranging aluminum bars 7, each 11 mm wide, in parallel along the short side of the space at 19 mm intervals. To provide cushioning, an 8 mm thick, 10 mm wide EVA (Ethylene- Vinyl Acetate) form 8 is attached to the aluminum bars 7 using double-sided tape, creating a linear deformation function for the muscles. The preferred hardness of the EVA foam 8 is around 45 degrees. The relaxation area should ideally be in contact with the slitted surface. For point-like deformation, a commercially available a wave foam 9 is also used to provide localized deformation of the muscles. A wave foam 9 is designed by balancing support-point density and local pressure, with the profile pitch set at approximately 5 cm. Because the mechanical properties of the foam directly influence the formation of point contact under a supine posture, the primary material specification is defined as the 40% compression hardness (N), measured with a compression platen area of 314 cm2. To minimize excessive local pressure and avoid the induction of protective muscularP4308PC00 / 1547-001 dpt

[0099] contraction, foam materials within a compression hardness range of approximately 250 N or lower are preferentially selected. The wave foam 9 may be constructed using a uniform foam material across the entire surface; alternatively, foam materials with different mechanical properties can be selectively applied to specific anatomical regions, enabling individualized or region-specific customization.

[0100] By applying slight pressure to the muscle area, the muscle spindle and Golgi tendon organ sense changes in muscle length and force. This triggers stretch reflexes and autogenic inhibition signals, which help to level the muscle’s length and tension. Once the pressure is released, the muscle tension in the targeted area is expected to improve. This process is assumed to promote the activity of afferent and efferent nerves in the muscles, reducing resistance when the muscles are passively moved. Consequently, the residual calcium ions in the affected area may be released, and the cross-bridges within the muscle fibers gradually break down.

[0101] The pressure exerted by modifying the resting surface 2 into linear or point-like structures varies depending on the subject’s weight, the degree of muscle tension, posture, and the material or shape used for deformation. However, optimization can be performed as needed. Although a flat surface also has a muscle relaxation effect, it is preferable to modify the surface to create linear or point-like structures that contact the body. These linear or point-like structures do not move on their own like a massage function. To efficiently facilitate contact with the body, an additional layer of the same or different material can be inserted beneath and / or above the modified surface, ensuring a better fit to the curvature of the lower legs, waist and / or neck. To further efficiently facilitate contact with the body, air can be injected into segmented areas in the lower layer beneath the modified surface, inflating specific areas as needed to partially improve contact between the modified surface and the body. The same or similar material of linear or point-like structures of the surface 2 of the resting surface 1, 2 can be used to additionally contact and deform the open space of the body lying on the device.

[0102] The muscle deformation process can also be achieved by wearing a full-body suit with internal slits or point-like structures. Additionally, using a hammock-like linear support structure to support the body can serve as a means of muscle deformation. However, careful consideration is needed regarding how to move a part of the subject’s body externally. Another possible approach involves utilizing water pressure from inside a waterbed to modify specific muscles.P4308PC00 / 1547-001 dpt

[0103] The recommended operation time for the device is approximately 10 - 15 minutes per session per a side. Using the device for longer than 15 minutes may not be efficient, as the relaxation curve stabilizes. If a total of 20 - 30 minutes of operation is required, it is preferable to divide the session into two separate uses. Chronic muscle tension, unless caused by specific conditions such as accidents or strokes, generally accumulates over a long period due to individual movement patterns and postural habits. As a result, chronic muscle tension tends to increase with age, making it increasingly difficult to maintain proper posture. Since muscle tension does not immediately release throughout the entire body, but rather gradually relaxes from the surface, long-term observation over several hours or more may be necessary for optimal use of this device. The relaxation of chronic muscle tension is effective not only for preventive purposes but also for long-term health when practiced daily over many years.

[0104] In addition to relaxing the muscles along the back, it is also beneficial to relieve tension on the chest side. This can be achieved by having the subject lie face down on the resting surface 2 provided by the device.

[0105] To confirm the effectiveness of this invention, several evaluation methods were employed, including changes in height, variations in resistance, resistance value when passively moving both feet, muscle hardness measurements, and posture changes.

[0106] Height changes were measured using the Charder Model HM200P. The subject’s height was measured before the session, followed by the experimental procedure, and measured again within one minute after the session. Since height fluctuates throughout the day, typically being tallest in the morning and decreasing gradually into the evening due to spinal disc compression, all measurements were conducted at a consistent time to ensure accuracy.

[0107] Height changes was adopted as one of the outcome measures in this study. Previous research has shown that acute postural or stretching-based exercises can induce immediate changes in measured stature, reflecting a combined effect of spinal decompression, postural realignment, and alterations in muscle activity (Ramos- Jimenez et al., 2024).

[0108] Although height changes represent an integrated outcome influenced by multiple biomechanical and neuromuscular factors, we hypothesize that the primary initiating factor underlying these changes is the reduction of chronic muscle tension. In particular, long-standing tonic muscle activity and co-contraction patterns are assumed to maintainP4308PC00 / 1547-001 dpt

[0109] spinal compression and postural misalignment. Their gradual relaxation may allow spinal structures to recover toward their unloaded configuration, resulting in measurable increases in hight.

[0110] Therefore, in the present study, changes in hight are not interpreted as isolated structural elongation, but as a global indicator reflecting the cumulative effects of muscle tension release, spinal decompression, and postural reorganization.

[0111] Ramos-Jimenez, A., Garcia-Hernandez, I. A., Chavez-Guevara, I. A., et al. (2024).

[0112] Enhanced stature in the elderly: The immediate impact of acute postural exercises.

[0113] The variation and value in resistance when passively moving both feet was assessed using a driving transmission component 5 where the driving force was transmitted through the bottom part of the driving transmission component 5a. An additional plate with the same shape, as the upper part of the driving transmission component 5e was placed on top of the bottom part of the driving transmission component 5a, designed to slide when resistance was detected between the two. Two slide rails 5g were installed in the bottom part of the driving transmission component 5a, with springs 5b (k-value of 0.348 N / mm) placed on each slide rail. By measuring the displacement of the slide components 5d, the resistance value was calculated by the detector with a belt 5c through an interconnection 5h connected to the upper part of the driving transmission component 5e.

[0114] Muscle hardness was measured using the TDM-NA1 muscle hardness meter manufactured by Neutone. This device is designed to non-invasively assess muscle stiffness by applying a fixed amount of pressure and notifying the user with an alarm (LED light and buzzer) when the pressure threshold is reached. Measurements were conducted on the central quadriceps region of the subjects. The device probe was placed perpendicularly on the muscle, and pressure was gradually applied until the internal alarm was activated. Three measurements were taken, and the average value was used as the muscle hardness index.

[0115] Postural changes were analyzed using the APECS (Al Posture Evaluation and Correction System) developed by Saneftec. This software uses non-invasive photometric technology to evaluate posture and detect distortions. The evaluation process involved taking fullbody photographs from the subject's right side, marking anatomical landmarks according to the software’s guide, and analyzing pre- and post-experimental posture changes quantitatively and objectively.P4308PC00 / 1547-001 dpt

[0116] The present invention provides a device that effectively alleviates chronic muscle tension through passive movement at controlled speeds. By moving a part of the body at a speed of 5.0 m / min or less, and / or at a reciprocating motion speed of 63 rpm or less, the device minimizes the activation of stretch reflexes while facilitating the natural relaxation of muscles. The mechanism incorporates both direct and indirect movement of tense muscles, allowing for gradual muscle relaxation through neuromuscular inhibition and sensory feedback processes. Additionally, the invention proposes an evaluation method based on changes in height before and after use, resistance value during passive movement, muscle hardness, and posture changes, ensuring an objective assessment of the device’s effectiveness.

[0117] Example 1: Evaluation of the Cushioning Layer in the Support Section

[0118] For this experiment, the muscle relaxation device was tested using a resting surface 1,2 measuring 190 cm in length and 60 cm in width. The resting surface 2 was prepared using a 7 cm-thick polyester foam cushion (Folding Mattress 60x190x7cm HYLLA BF15) manufactured by JYSK A / S. The drive mechanism 3 was based on the MRM device produced by MRM Leg Arm Fit Sari, with modifications implemented for this study.

[0119] A subject (referred to as Subject A) lay on his back, placing both heels on the support section 4 of the drive mechanism 3. The inner surface of the support section was lined with a 2.54 cm-thick Memory Foam Standard from FormOnline.com as a cushioning layer 6 to provide cushioning. The device was operated at a speed of 0.39 m / min (3.25 rpm) with an amplitude movement range of 6 cm, and passive motion was applied to the subject’s body for 10 minutes as shown in Fig 1.

[0120] Before and after the session, the subject’s height was measured using the Charder Model HM200P. A height increase of 4 mm was observed after the session, suggesting a reduction in muscle tension and spinal decompression following the passive movement process.P4308PC00 / 1547-001 dpt

[0121] Example 2: Evaluation of the Relationship Between Passive Movement Speed and Height Changes

[0122] For this experiment, the muscle relaxation device was tested using the same resting surface 2 setup as in Example 1, with a 190 cm * 60 cm area covered by a 7 cm -thick polyester foam cushion (Folding Mattress 60x190x7cm HYLLA BF15) manufactured by JYSK A / S. The drive mechanism 3, based on the MRM device by MRM Leg Arm Fit Sari, was further modified to include a driving transmission component 5 to have buffering function and measure resistance value as shown in Fig 4 and 5.

[0123] A subject (Subject A) lay on their back, placing both heels on the support section 4 of the drive mechanism 3. The inner surface of the support section was lined with a 7 mm-thick EVA form manufactured by Kappa as the cushioning layer 6. The subject’s legs were passively moved at predetermined speeds, with an amplitude movement range of 6 cm, for 10 minutes per trial as shown in Fig 2.

[0124] The experiment was conducted with movement speeds ranging from 0.39 m / min (3.25 rpm) to 7.92 m / min (66 rpm). Resistance values during movement were recorded starting 30 seconds after initiating motion, and resistance profiles were analysed over a 30-second measurement period. The recorded data included the average integrated resistance value for both legs over 30 seconds, as well as the average maximum and minimum resistance range during this period.

[0125] Additionally, the subject’s height was measured before and after each session to determine any correlation between passive movement speed and spinal decompression. The collected data provided insights into the optimal movement speed for maximizing muscle relaxation.

[0126] Table 1

[0127] rpm Speed (m / min) Integral Ave. Right & Left { N ) Range Ave. Right & Left (N) Height difference (mm) 3.25 0.39 120.39 1.24 4.5 10 1.2 118.24 1.12 3.7

[0128] 20 2.4 2.8

[0129] 30 3.6 108.95 1.10 2.5 36 4.32 2.0

[0130] 41 4.92 78.81 0.81 2.0

[0131] 48 5.76 66.50 0.74 1.0

[0132]

[0133] 66 7.92 0.8P4308PC00 / 1547-001 dpt

[0134] As the speed decreases, the height difference increases, indicating a strong negative correlation between these variables as shown in Fig 3. Conversely, both the integral and range of the average right and left resistance values show a strong positive correlation with the height difference, meaning that greater resistance values correspond to a larger height difference. This suggests that higher speeds reduce the applied and / or reacted force, leading to a smaller height difference.

[0135] Example 3: Evaluation of Muscle Hardness Using an Aluminum Bar Configuration

[0136] Instead of the polyester foam cushion as the resting surface 2 used in Example 2, the surface of the resting surface Iwas modified into a linear structure by arranging parallel

[0137] (e.g. aluminum) bars 7, each 11 mm in width, with 19 mm spacing along the short side of the space. To provide cushioning, an 8 mm thick and approximately 10 mm wide EVA form 8 was attached to the 11 mm wide surface of each aluminum bar 7 using doublesided tape, allowing for a linear deformation of specific muscle areas as shown in Fig 6. The EVA foam 8 used had a hardness of 45 degrees.

[0138] A subject (Subject B) lay on their back and placed both heels on the support section 4 of the drive mechanism 3 used in Example 2. The inner surface of the support section was lined with a 7 mm-thick EVA form manufactured by Kappa as the cushioning layer 6. Passive movement was applied for 5 minutes at a speed of 1.2 m / min (10 rpm). Before and after the session, muscle hardness in the central part of the right calf was measured using a muscle hardness meter, with three measurements taken (n=3), and the average value was calculated.

[0139] Before the session, the muscle hardness was recorded at 17 (100%). After the session, it had decreased to 14 (82.4%), indicating a reduction in muscle stiffness following the passive movement process.

[0140] Following this, Subject B lay face down and placed the tops of their feet on the support section 4 of the drive mechanism 3. The same passive movement was applied for 5 minutes at a speed of 1.2 m / min (10 rpm). Muscle hardness in the central part of the right quadriceps was measured before and after the session using the same method (n=3, with the average value calculated).P4308PC00 / 1547-001 dpt

[0141] Before the session, the muscle hardness of the right quadriceps was recorded at 15.3 (100%). After the session, it had decreased to 12.7 (82.6%), demonstrating a reduction in muscle stiffness.

[0142] Example 4: Evaluation of the Relationship Between Passive Movement Speed and Height Changes Using a Wave Foam

[0143] For this experiment, the same 190 cm * 60 cm polyester foam cushion as the resting surface 2(7 cm thick, Folding Mattress 60x190x7cm HYLLABF15) from JYSK A / S used in Example 2 was modified by placing a wave foam 9, a Softpur GmbH Noppenplatte (500mm x 500mm x 50mm) on top, with the protruding surface facing upwards as shown in Fig 7.

[0144] A subject (Subject A) lay on their back, positioning both heels on the support section 4 of the drive mechanism 3. The inner surface of the support section was lined with a 7 mm-thick EVA form manufactured by Kappa as the cushioning layer 6. Passive movement was applied for 10 minutes at predetermined speeds, moving a part of the body passively. The drive mechanism had an amplitude movement range of 6 cm.

[0145] The experiment was conducted at movement speeds ranging from 0.39 m / min to 7.92 m / min to examine the effect of passive oscillation on height changes.

[0146] Table 2

[0147] rpm Speed (m / min) Integral Ave. Right & Left (N) Range Ave. Right & Left (N) Height difference (mm) 3.25 0.39 297.35 3.11 5.3 8 0.96 5.0 20 2.4 3.7 27 3.24 285.85 2.84 3.5 35 4.2 2.5 41 4.92 281.60 2.78 2.0 48 5.76 256.47 2.70 1.0

[0148]

[0149] 66 7.92 0.5

[0150] As the speed decreases, the difference in height variation further increases, indicating a strong negative correlation between these variables as shown in Fig 8. Conversely, both the integral and range of the average right and left values show a strong positive correlation with the height difference, meaning that greater resistance values correspond to a larger height difference. This suggests that higher speeds reduce the applied and / or reacted force, leading to a smaller height difference.P4308PC00 / 1547-001 dpt

[0151] Example 5: Long-Term Evaluation and Postural Changes

[0152] Based on the experimental conditions established in Example 3, a long-term evaluation was conducted to observe changes in posture. The experiment was carried out over a period of seven weeks, with a total of eight sessions.

[0153] In each session, Subject B lay on their back and placed both heels on the support section 4 of the drive mechanism 3. The inner surface of the support section was lined with a 7 mm-thick EVA form manufactured by Kappa as the cushioning layer 6. Passive movement was applied at a speed of 1.2 m / min (10 rpm) for 10 minutes. After this, the subject lay face down, placing the tops of their feet on the support section 4 of the drive mechanism 3, and the same passive movement was applied at 1.2 m / min (10 rpm) for another 10 minutes.

[0154] Over the course of the experiment, the total duration of passive movement amounted to 85 minutes in the face up position and 85 minutes in the face down position. The changes in posture observed throughout the experiment are shown in Fig 10. Most angles have improved.

[0155] Example 6: Additional evaluation in the face down position

[0156] Under the same conditions as in Example 4, Subject A lay on their back, placing both heels on the support section 4 of the drive mechanism 3. Passive movement was applied at a speed of 0.39 m / min for 10 minutes. The recorded change in height was +4.5 mm.

[0157] Following this, the subject turned over into a face down position and underwent an additional 10 minutes of passive movement at 0.39 m / min. After this second session, the height increased by an additional +4.0 mm.

[0158] In total, the subject’s height changed by +8.5 mm after completing both face up and face down sessions.

[0159] Example 7: Evaluation of the Relationship Between Passive Movement and Height Changes Using 2 kinds of a Wave Form

[0160] For this experiment, the muscle relaxation device was tested using a resting surface 1 measuring 187 cm in length and 68 cm in width from Robusta 83691 Top Flex prestige VI KK. The resting surface 2,9 was prepared using 2 different kinds of hardness of waveP4308PC00 / 1547-001 dpt

[0161] foam (70cm x 195cm) on a resting surface 1, with the protruding surface facing upwards made by Polyurethane foam, 8 cm thickness, 5cm profile pitch, 4cm valley depth, manufactured by Toyo Quality One Corporation as shown in Fig 14 as a reference. The drive mechanism 3 was based on the MRM device produced by MRM Leg Arm Fit Sari, including further modification with a driving transmission component 5 to have buffering function.

[0162] A subject (Subject A or E) lay on their back, positioning both heels on the support section 4 of the drive mechanism 3. The inner surface of the support section was lined with a 7 mm-thick EVA form manufactured by Kappa as the cushioning layer 6. Passive movement was applied for 10 minutes at 0.39 m / min speeds, moving a part of the body passively. The drive mechanism had an amplitude movement range of 6 cm.

[0163] The experiment was conducted with 2 different kinds of hardness 145N (product code: 30R) and 185N (product code: 4319R) of wave foam to examine the effect of passive oscillation on height changes as shown in Fig 11. Values are reported as the mean of two measurements (n = 2).

[0164] Table 3

[0165] Height change(mm)

[0166] Hardness(N)

[0167] Subject A Subject E

[0168] 30R 145 5.7 4.8

[0169]

[0170] 4319R 185 5.7 6.0

[0171] Example 8: Evaluation of the Relationship Between Passive Movement speed (m / min) and Height Changes

[0172] For this experiment, the muscle relaxation device was tested using a resting surface 1 measuring 187 cm in length and 68 cm in width from Robusta 83691 Top Flex prestige VI KK. The resting surface 2,9 was prepared by a wave foam (70cm x 195cm) called Classe+ Shiatsu type on a resting surface 1, with the protruding surface facing upwards made by Polyurethane foam, 8 cm thickness, 4.5cm profile pitch, 3.5cm valley depth, manufactured by EMOOR Co., Ltd as shown in Fig 14 as a reference. The drive mechanism 3, as part of a mechanical movement unit 14, was fabricated with a linear actuator (11E13S1004HD5-180RS-E22-300), including further modification with a driving transmission component 10 to have buffering function as shown in Fig 12 and 13.P4308PC00 / 1547-001 dpt

[0173] A subject (Subject C) lay on their back, positioning both heels on the support section 4 of the drive mechanism 3. The inner surface of the support section was lined with a 7 mm-thick EVA form manufactured by Kappa as the cushioning layer 6. Passive movement was applied for 10 minutes at a different speed, moving both heels as part of the body passively. The drive mechanism 3 had an amplitude movement range of 6 cm.

[0174] The experiment was conducted at movement speeds ranging from 0.03 m / min to 0.24 m / min to examine the effect of passive oscillation on height changes as shown in Fig 15.

[0175] Table 4

[0176] Speed(m / min) Subject C

[0177] 0.03 5.0

[0178] 0.05 7.5

[0179] 0.1 6.0

[0180] 0.15 6.0

[0181]

[0182] 0.24 5.0

[0183] Example 9: Evaluation of the Relationship Between Passive Amplitude Movement range(cm) and Height Changes

[0184] For this experiment, the muscle relaxation device was tested using a resting surface 1 measuring 187 cm in length and 68 cm in width from Robusta 83691 Top Flex prestige VI KK. The resting surface 2,9 was prepared by a wave foam (70cm x 195cm) called Classe+ Shiatsu type on top, with the protruding surface facing upwards made by Polyurethane foam, 8 cm thickness, 4.5cm profile pitch, 3.5cm valley depth, manufactured by EMOOR Co., Ltd. The drive mechanism 3, as part of a mechanical movement unit 14, was fabricated with a linear actuator(11E13S1004HD5-180RS-E22-300), including further modification with a driving transmission component 10 to have buffering function.

[0185] A subject (Subject C or D) lay on their back, positioning both heels on the support section 4 of the drive mechanism 3. The inner surface of the support section was lined with a 7 mm-thick EVA form manufactured by Kappa as the cushioning layer 6. Passive movement was applied for 10 minutes at a speed of 0.15 m / min with different amplitude movement range from 1cm to 8cm, which is 0.97rpm (8cm amplitude movement range) to 7.5rpm(lcm amplitude movement range), moving both heels as part of the bodyP4308PC00 / 1547-001 dpt

[0186] passively to examine the effect of passive oscillation on height changes as shown in Fig 16.

[0187] Table 5

[0188] Range(cm) Subject C Subject D

[0189] 1 6.0 5.0

[0190] 2 6.0 6.0

[0191] 4 5.5 4.5

[0192] 6 6.0 5.0

[0193]

[0194] 8 5.0 5.0

[0195] Comparative Example 1: Evaluation of Muscle Hardness Changes Using the Chi Vitalizer

[0196] For comparison, a Chi Vitalizer Classic manufactured by Chi-Enterprise was used as a passive movement device designed for "goldfish exercise" motions. The lowest available setting was selected, operating at 80 rpm (6.4 m / min). Both feet were raised approximately 20 cm, and the heels were placed on the device as shown in Fig 18. Passive movement was applied for 10 minutes, with an amplitude motion range of 4 cm.

[0197] The change in height before and after the session was recorded as +1.0 mm.

[0198] To assess muscle relaxation, a muscle hardness meter was used before and after the session. Measurements were taken from the central right quadriceps and central right calf muscles, with three trials per area (n=3), and the average values were recorded.

[0199] For the central right quadriceps, the pre-experiment muscle hardness was recorded at 18.7 (100%), which decreased slightly to 18.0 (96%) after the session.

[0200] For the central right calf muscles, the pre-experiment muscle hardness was recorded at 16.3 (100%), and there was no change (16.3, 100%) after the session.P4308PC00 / 1547-001 dpt

[0201] Comparative Example 2: Evaluation of Aluminum Bar Alone

[0202] Under the same conditions as Example 3, Subject A lay on their back, placing both heels on the support section 4 of the drive mechanism 3 used in Example 3. The inner surface of the support section was lined with a 7 mm-thick EVA form manufactured by Kappa as the cushioning layer 6. However, in this case, the drive mechanism 3 was not activated, and the subject remained lying still for 10 minutes.

[0203] The change in height before and after the session was 0 mm, indicating no measurable effect.

[0204] Comparative Example 3: Evaluation of Wave Foam Alone

[0205] Under the same conditions as Example 4, Subject A lay on their back, placing both heels on the support section 4 of the drive mechanism 3 used in Example 4. The inner surface of the support section was lined with a 7 mm-thick EVA form manufactured by Kappa as the cushioning layer 6. However, in this case, the drive mechanism 3 was not activated, and the subject remained lying still for 10 minutes.

[0206] The change in height before and after the session was 0 mm, indicating no measurable effect.

[0207] Comparative Example 4: Evaluation with Elevated Legs

[0208] Under the same conditions as Example 4, Subject A lay on their back, placing both heels on the support section 4 of the drive mechanism 3 used in Example 4. The inner surface of the support section was lined with a 7 mm-thick EVA form manufactured by Kappa as the cushioning layer 6. However, the support section 4 was elevated by 17 cm, ensuring that the feet did not contact with the wave foam 9, the Softpur GmbH Noppenplatte during reciprocal movement (while the sacrum to the head remained in contact with the wave foam 9. Passive movement was applied at a speed of 0.39 m / min for 10 minutes.

[0209] The change in height before and after the session was 0 mm, indicating no measurable effect.

[0210] The present invention is of course not limited to the examples discussed previously, which refer to a support section configured for receiving the heel. The support section could also be configured for other parts of the body, including the knees, the hands or the pelvis.P4308PC00 / 1547-001 dpt

[0211] It should also be underlined that the oscillatory motion applied to the subject may apply to any part of the body, which is not necessarily in direct contact with the support sections.

Claims

P4308PC00 / 1547-001 dptClaims1. A muscle relaxation device comprising a driving mechanism (3) to induce muscle relaxation in a subject, and a support section (4) attached to said driving mechanism (3) that holds at least one body part of said subject such as the heel, characterized in that the driving mechanism (3) is configured to move at least said body part and optionally at least another body part; said driving mechanism (3) being furthermore configured to induce an oscillatory motion applied to the subject, and wherein said oscillatory motion is applied at a speed of 5,0 m / min or less and / or at 63 rpm or less,2. The device according to Claim 1, wherein the support section (4) is provided with a buffer element (5b, 6, 10c) configured to buffer the pressure generated from the said body part(s) body when said oscillatory motion is applied.

3. The device of claim 2 wherein the buffer element (5b, 6, 10c) is fixed to the support section (4).

4. The device according to anyone of the previous claims wherein said oscillatory motion is applied at a speed of 2.0 m / min or less.

5. The device according to claim 4 wherein said oscillatory motion is applied at a speed of 0.5 m / min or less.

6. Combination of a muscle relaxation device according to anyone of the previous claims and a resting surface (1,2, 7, 8, 9) that is configured for a subject to be in a rest position, and wherein the driving mechanism (3) and the resting surface (1,2, 7, 8, 9) being furthermore configured in a way that when the oscillatory motion is applied to the subject, its body remains supported by, and in contact with, the resting surface (1,2, 7, 8, 9).

7. Combination according to claim 6 wherein the resting surface (1,2, 7, 8, 9) is horizontal and the support section 4 is located in the same plane as the resting surface (1,2, 7, 8, 9), in a way that the complete body of the subject is oriented along a same plane, that is parallel to the resting surface (1,2, 7, 8, 9).

8. Combination according to 6 or 7 wherein the resting space is made of parallel bars (7,8).P4308PC00 / 1547-001 dpt9. Combination according to anyone of claims 6 to 8 wherein the upper face of the resting space forms a point-like pattern.

10. Combination according to the previous claim wherein the upper face is made by a wave foam (9).

11. Method for using the device according to any one of the previous claims comprising a driving step which induces an oscillatory motion to a muscle to be relaxed, wherein said oscillatory motion is induced at a speed of 5.0 m / min or less and / or at 63 rpm or less.

12. Method according to claim 11 further comprising a deforming step which deforms said muscle, wherein the driving step and the deforming step are performed simultaneously.

13. Method for assessing the degree of overall muscle relaxation, when using the device according to anyone of claims 1 to 3, by measuring changes in the subject height before and after evaluation.

14. Method for assessing the degree of chronic muscle tension, when using the device according to anyone of claims 1 to 3, by utilizing the resistance generated in response to the subject network of chronic muscle tension when a part of its body is moved passively.

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