Construction of a cell model of collagen metabolism abnormality induced by knockout of sparc gene and application thereof
By designing and validating sgRNA sequences and recombinant vectors targeting the human SPARC gene, the problem of low sgRNA editing efficiency in HeLa cells using CRISPR-Cas9 technology was solved, and an efficient SPARC gene knockout model was constructed to study the function of SPARC in tumor cell migration and collagen metabolism.
Patent Information
- Authority / Receiving Office
- CN · China
- Patent Type
- Applications(China)
- Current Assignee / Owner
- GUANGXI UNIV
- Filing Date
- 2026-04-14
- Publication Date
- 2026-07-07
AI Technical Summary
Existing CRISPR-Cas9 technology suffers from problems such as low sgRNA editing efficiency, strong design blindness, low screening efficiency, and lack of efficient and specific sgRNA tools when constructing SPARC gene knockout models, especially in human cervical cancer HeLa cells where efficient knockout is difficult to achieve.
A sgRNA sequence (sgRNA1, sgRNA2, sgRNA3) targeting the human SPARC gene was provided. Combined with the recombinant expression vector PX459 V2.0, a homozygous SPARC gene knockout model was constructed by transfecting HeLa cells, and the knockout effect was verified by PCR and T7EI restriction enzyme digestion.
Efficient and specific SPARC gene knockout was achieved in HeLa cells, significantly shortening the research cycle and providing stable experimental materials for in-depth research on the function of SPARC in tumor cell migration and collagen metabolism.
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