Method for producing non-human primate model of cerebral infarction and pharmaceutical composition for treating cerebral infarction

JPWO2024005124A5Pending Publication Date: 2026-07-06
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Patent Information

Authority / Receiving Office
JP · JP
Patent Type
Applications
Filing Date
2023-06-29
Publication Date
2026-07-06

AI Technical Summary

Technical Problem

Current cerebral infarction models, particularly in non-human primates, are inadequate for evaluating therapeutic agents in subacute to chronic stages due to limited durability of motor dysfunction and difficulty in inducing consistent damage in perforator-innervated regions, leading to a lack of effective treatments for these stages.

Method used

A non-human primate animal model is created by administering endothelin to the basal ganglia and thalamic regions to induce basal ganglia, thalamic, and internal capsule damage, which is maintained over a long period, and NeuroD1 is used as a therapeutic agent to promote nerve regeneration.

Benefits of technology

The model allows for the evaluation of therapeutic agents in subacute to chronic cerebral infarction and perforator infarction, demonstrating prolonged motor dysfunction and potential for functional recovery through NeuroD1-induced nerve regeneration.

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Abstract

The present invention relates to: a method for producing a non-human primate model of cerebral infarction, said method including administering an endothelin to the cerebral basal ganglia and thalamic area of a non-human primate and thus inducing cerebral basal ganglia damage, thalamic damage and internal capsule damage; and a pharmaceutical composition for treating subacute to chronic cerebral infarction, perforating branch infarction, or cerebral infarction with brain damage in the perforating branch dominant area, said pharmaceutical composition containing NeuroD1 protein or a polynucleotide encoding NeuroD1 protein.
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