Application of Tricarpellate Wampee Fruit Extract in Preparing External Preparation for Repairing Skin after Sunburn

By using triangular extract to inhibit signal pathways related to skin post-sun damage, the problems that oxidative stress, inflammatory response and pigmentation in the prior art are solved, and significant skin repair effects are achieved.

CN118986830BActive Publication Date: 2025-06-13ZHAOLAI HLDG (SUZHOU) CO LTD +1
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Patent Information

Application Number
CN202411269889.3
Authority / Receiving Office
CN · China
Patent Type
Patents(China)
Current Assignee / Owner
Filing Date
2024-09-11
Publication Date
2025-06-13
Estimated Expiration
2044-09-11

AI Technical Summary

Technical Problem

The prior art is difficult to effectively solve the problem of skin post-sun damage, especially in terms of oxidative stress, inflammatory response and pigmentation.

Method used

Triangular extract is used as the main component to protect the skin from damage from ultraviolet radiation by inhibiting oxidative stress, inflammatory response and pigmentation. Specific methods include scavenging free radicals, inhibiting ROS production, downregulating the expression of AGEs, reducing gene expression of NF-κB and HMGB1, inhibiting the secretion of TNF-α, and reducing the expression of α-MSH and β-catenin.

Benefits of technology

Significantly alleviates symptoms of skin post-sun damage, including reducing oxidative damage, inflammatory response and pigmentation, thereby improving the skin's repair effect.

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Abstract

The present invention belongs to the technical field of cosmetics, and specifically relates to the application of the extract of Sparganium stoloniferum in the preparation of an external preparation for repairing skin after sunburn. The extract of Sparganium stoloniferum prepared by the present invention can scavenge free radicals, inhibit lipid peroxidation, inhibit the production of ROS, down-regulate the expression of AGEs, and can also inhibit the inflammatory response induced by ultraviolet rays, reduce the expression of NF-κB and HMGB1 genes up-regulated by ultraviolet rays, inhibit the secretion of the inflammatory factor TNF-α. At the same time, it can reduce the expression of the melanocyte-stimulating factor α-MSH and β-catenin genes up-regulated by ultraviolet rays, inhibit the production of melanin and the transport of melanosomes, that is, significantly reduce the symptoms of skin damage after sunburn by improving various signal pathways such as skin oxidative damage, inflammatory response, and pigmentation caused by ultraviolet rays. When it is applied to external preparations such as cosmetics for repairing skin damage after sunburn, it will have broad market application prospects.
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