Mongolian medicine tea drink capable of relieving respiratory system abnormality and preparation method and application of Mongolian medicine tea drink

Abstract

The invention relates to the technical field of traditional Chinese medicine, in particular to a Mongolian medicine tea drink for relieving respiratory system abnormality and a preparation method and application thereof.The Mongolian medicine tea drink is prepared from rhododendron micranthum, pomegranate, amomum cardamomum, long pepper, cinnamon, safflower, agilawood, clove, myristica fragrans, fructus choerospondiatis, bistort rhizome, liquorice, tabasheer, radix aucklandiae, white raisin and crabs. The processed products of the Mongolian medicine components are weighed according to the formula dosage, then ground into coarse powder and bagged to prepare tea bags. The Mongolian medicine tea drink provided by the invention has the effects of moving Heli, relieving cough, reducing phlegm and diminishing swelling; the traditional Chinese medicine composition can be used for treating the symptoms such as' Bada dry Hiri 'dizziness, asthma, chronic bronchitis, indigestion disease, edema, edema and water tympanites.

Description

Technical Field

[0001] The invention belongs to the technical field of Mongolian medicine application, and particularly relates to a Mongolian medicine tea drink for relieving respiratory system abnormalities and an application thereof. Background Art

[0002] Respiratory diseases are a complex, common and frequently occurring disease, mainly caused by bacteria or viruses that lead to lesions in the human trachea, bronchi, lungs, chest cavity and other parts of the body. There are many types of respiratory diseases, the most common of which are asthma, pneumonia, tuberculosis, cor pulmonale, chronic obstructive pulmonary disease (COPD), acute upper respiratory tract infection and chronic bronchitis.

[0003] Current medications for treating respiratory diseases include bronchodilators (such as aminophylline, tiotropium, and other β2 receptor stimulants. Corticosteroids can be used as needed), antibiotics, and home oxygen therapy. However, these medications only treat the symptoms and not the root cause. Long-term use of these medications can also have serious side effects. Long-term use of bronchodilators can easily lead to lung expansion, which can lead to cor pulmonale (lung compression of the heart). Therefore, a daily beverage that can alleviate respiratory abnormalities without toxic side effects was developed. Summary of the Invention

[0004] In view of the deficiencies in the prior art, the present invention provides a Mongolian medicinal tea for relieving respiratory system abnormalities, and a preparation method and application thereof.

[0005] Specifically, the first aspect of the present invention provides a Mongolian medicinal tea for relieving respiratory system abnormalities, which is composed of the following Mongolian medicinal ingredients: Zhaoshanbai, pomegranate, white cardamom, long pepper, cinnamon, safflower, agarwood, cloves, nutmeg, jujube, polygonum multiflorum, licorice, rhizoma jasminoides, costus root, white raisins, and crab.

[0006] As a further illustration of the present invention, the Mongolian medicine tea is composed of the following Mongolian medicine ingredients in parts by weight: 20g of Rhizoma Zangbolis, 40g of pomegranate, 2.5g of white cardamom, 5g of Piper longum, 2.5g of cinnamon, 25g of safflower, 25g of agarwood, 25g of cloves, 20g of nutmeg, 30g of Ziziphus jujuba, 24g of Polygonum multiflorum, 16.5g of licorice, 29g of Radix Glycyrrhizae, 20g of costus root, 15g of white raisins, and 10g of crab.

[0007] The second aspect of the present invention provides the use of any one of the Mongolian medicinal teas described above in the preparation of a medicine for alleviating respiratory system abnormalities.

[0008] The third aspect of the present invention provides a method for preparing a Mongolian medicinal tea for relieving respiratory system abnormalities, comprising: weighing each of the above-mentioned single Mongolian medicinal herbs according to the dosage of the formula, then grinding them into coarse powder, and bagging them to make tea bags.

[0009] As a further illustration of the present invention, the dosage of the tea bag is 30-50 g / bag.

[0010] The following is an overview of the individual herbs used in the above teas: 1. Zhaoshanbai The Mongolian name "Altan-Kharabul" is the dried young branches and leaves of the evergreen shrub Rhododendron micranthum Turcz. or Rhododendron dauricum L. of the Ericaceae family.

[0011] 1.1 Chemical composition study Zhaoshanbai contains terpenes (lupeol ethyl ester, germacron, lupeol, lupeol, 3-hydroxy-30-norlupan-20-one, 3-hydroxy-11-ene-11,12-dehydro-28,13-ursolic acid lactone, ursolic acid, betulinic acid, 3,11-dihydroxy-12-ene-12,13-dehydro-ursane, oleanolic acid), fatty acids, sterols (6β-hydroxy-4-ene-3-stigmasterone, 3β-hydroxy-5-ene-7-stigmasterone, β-sitosterol), benzene ring derivatives (p-hydroxybenzaldehyde, 2-(hydroxymethyl)phenol), 5-hydroxy-7-methoxy-2-(4-methoxyphenyl)-6-methyl-4H-chromene-4-one and umbelliferone and other chemical components. Yang Xiuling et al. studied the flavonoid components and isolated six monomer compounds from it, including quercetin, kaempferol, quercetin-3-O-β-D-glucoside, hyperoside, quercetin and myricetin. Among them, the compounds quercetin-3-O-β-D-glucoside, quercetin and myricetin were isolated from this plant for the first time.

[0012] 1.2 Pharmacological studies Flavonoids from the herb Dioscorea altissima have significant anti-inflammatory, analgesic, and anti-prostatic hyperplasia effects. Research by Yang Xiuling et al. showed that total flavonoids from Dioscorea altissima significantly inhibited testosterone propionate-induced prostatic hyperplasia in normal male mice. It also significantly inhibited the increase in peritoneal capillary permeability induced by glacial acetic acid, reduced xylene-induced ear swelling in mice, and significantly inhibited acetic acid-induced writhing reactions in mice, reducing the number of writhing episodes. Furthermore, research by Liu Yang et al. found that an ethanol extract from Dioscorea altissima altered the physicochemical parameters of ischemic myocardium in rats, demonstrating a significant protective effect.

[0013] 2. Pomegranate Pomegranate, called "Anar" in Mongolia, is the fruit of Punica granatum L., a plant of the Pomegranate family.

[0014] 2.1 Chemical composition study Pomegranate contains chemical components such as tannins, polyphenols, flavonoids, alkaloids, organic acids, triterpenoids and phytosterols. Among them, granatin A and B, pomegranate tannin, 2,3-O-digalloyl pomegranate tannin and ellagitannins are tannin components; punicalagin, punicalagin, anthocyanidin, pomegranate ellagitannin, punicalagin A, punicalagin B, gallic acid, punicalagin, punicalagin, tannin and ellagic acid are polyphenol components; rutin, pelargonidins, delphinidins, luteolin, quercetin, punicine, isopunicine, pseudopunicine, N-methylisopunicine, camphor, 5,7,4'-trihydroxyisoflavone and 7,4'- Dihydroxyisoflavones, luteolin, flavonoids, etc. are flavonoid components, arachidic acid, arachidic acid, 11-arachidic acid, palmitic acid, stearic acid, behenic acid and other fatty acid components and cyanidin-3-glucoside, cyanidin-3,5-diglucoside and other components.

[0015] 2.2 Pharmacological studies Pomegranate has antioxidant, hypoglycemic, anti-inflammatory, anti-atherosclerotic, anti-tumor, and antibacterial pharmacological effects.

[72] In addition, it also has inhibitory and therapeutic effects on the nervous system, gastrointestinal digestive system, osteoarthritis, nephritis, asthma and acne.

[0016] 2.2.1 Anti-inflammatory effect Pomegranate extract, ellagitannins, and ellagic acid are promising agents for targeting the SARS-CoV-2 virus, limiting the host's inflammatory response to viral infection, and replenishing depleted host antioxidant levels during the COVID-19 recovery phase. Wang Ying et al.'s research suggests that punicalagin inhibits ASMC proliferation and induces apoptosis, effectively suppressing the malignant proliferation of ASMCs. Punicalagin alleviates airway remodeling in young asthmatic rats, inhibiting ASMC proliferation and inducing apoptosis. Xie Dongjie's research found that pomegranate peel and its active ingredient, tannic acid, significantly blocked the binding of multiple GII / GLNoV strains to HBGA receptors. This broad-spectrum blocking effect on HBGA receptor binding may be the primary mechanism of action of the traditional Chinese medicine "astringent bowel and antidiarrheal" for treating viral gastroenteritis. Wei Lu's research suggests that pomegranate peel polyphenol cream inhibits inflammatory responses by suppressing the HIF-1α / IL-17 signaling pathway, thereby exerting its therapeutic effects on acne. Pomegranate peel tannins have a significant therapeutic effect on rats with passive Heymann nephritis.

[0017] 2.2.2 Other functions In addition to its anti-inflammatory effects, pomegranate also has pharmacological effects such as anti-tumor, antihypertensive, and antidepressant. Chen Chen et al. proposed that punicic acid can inhibit the viability of endometrial adenocarcinoma RL-952 cells and promote apoptosis of RL-952 cells, and its effect is stronger than that of another conjugated linolenic acid isomer, α-eleostearic acid.

[79] . Among the 24 pomegranate derivatives, pedunculagin is especially important, which may help lower blood pressure through the dual mechanisms of increasing NO levels and inhibiting ACE. Pang Zhenzhen's research found that pomegranate peel extract (punicalagin) can improve the depressive symptoms of CUMS model mice. Its mechanism of action may be related to improving intestinal flora and reducing intestinal wall inflammation and damage. Wang Cheng et al. found that punicalagin has a significant improvement effect on ovariectomized female osteoarthritis rats. Pomegranate seed oil can increase bone volume fraction, reduce structural model index, and improve subchondral osteoporosis; pomegranate seed oil can downregulate the expression of MMP-1 in chondrocytes, inhibit CoⅡ degradation, restore cartilage elasticity, and delay articular cartilage degeneration.

[83] .

[0018] 3. White cardamom White cardamom, called "Sugemul" in Mongolian, is the fruit of the ginger family plants Amomum kravanh Pierre exGagnep. and Javanese white cardamom Amomum compactum Soland ex Maton (A.cardamomum auct.nonL.).

[0019] 3.1 Chemical composition study: Xiao Shuhua et al. found that white cardamom contains volatile oils (monoterpenes and sesquiterpenes such as 1,8-cineole, α-pinene, α-terpinene, and β-pinene), flavonoids, diarylheptanes, and other chemical components. Badalahu et al. found that the aqueous extract of white cardamom contains organic acids (47 compounds, including aromatic acids and fatty acids), flavonoids (aromatic acids and fatty acids), amino acids, alkaloids, and lactones. Hao-Ming Xiong, Hui-Ying Li et al. isolated four new sesquiterpenoids, three new monoterpene derivatives, two new neolignans, and a new nosesquiterpenoid from white cardamom.

[0020] 3.2 Pharmacological studies: Modern research indicates that different active ingredients in white cardamom have protective effects against renal lesions. Cheng Xiaoling, Feng Zhuzhu, Zhang Ke, and others found that white cardamom volatile oil can improve gentamicin-induced acute kidney injury in rats. Liu Xiaoqin and others found that white cardamom ethanol extract has a protective effect against adenine-induced renal fibrosis in rats. Furthermore, white cardamom can alleviate renal damage, inhibit renal cell apoptosis, oxidative stress, and inflammation, and inhibit the STAT1-P53-P21 pathway, thereby alleviating doxorubicin-induced nephropathy, reducing renal inflammation and oxidative stress, and delaying the progression of renal fibrosis, thereby protecting against chronic renal failure.

[0021] 4. Piper longum Piper longum, also known as "bibiling" in Mongolia, is the dried mature fruit cluster of Piper longum L., a perennial climbing vine of the Piperaceae family.

[0022] 4.1 Chemical composition study Piper longum contains a variety of chemical components, including alkaloids, flavonoids, lignans, volatile oils, and fatty acids. The most important chemical components are amide alkaloids, of which piperine is a representative alkaloid compound. Wang Rui et al. found that the volatile oil of Piper longum contains low levels of monoterpenes, moderate levels of sesquiterpenes, and high levels of aliphatic hydrocarbons. Hui Yuyu et al. found that piperine is the main component of Piper longum root and its chemical structure is a cinnamamide. Liao Cuiping et al. identified alkaloids such as piperine, piperine and pellitorine, flavonoids such as rutin and isoquercetin, amino acids such as glutamic acid and aspartic acid, and glycosides such as adenosine from the alcohol extract of Piper longum. They also identified seven compounds, including safrole, protocatechuic aldehyde, 6-hydroxykaempferol-3,6-diglucoside, adenosine, polygalic acid, kaempferol-3-rutinoside and narcissin, from Piper longum for the first time.

[0023] 4.2 Pharmacological studies Piper longum extract has anti-inflammatory, cancer cell growth inhibition, and anti-tumor pharmacological effects. Piper longum amide can inhibit cancer cell proliferation in vitro and in vivo, promote cancer cell ferroptosis, and exert anti-tumor effects. Piper longum derivative C12 activates the MAPK signaling pathway in H1299 cells by upregulating the expression of p-JNK, p-Erk1 / 2, and p-p38, thereby exerting its anti-tumor activity. Studies by Tang Shi et al. have shown that the Piper longum extract, piperonine, can significantly inhibit AVIC calcification induced by high calcium and high phosphorus. Its anti-calcification effect is mainly through inhibiting the activation of the BMP2 pathway, inhibiting the osteogenic differentiation of valvular interstitial cells. ]Yang Yanmin's research found that piperine and its derivatives can inhibit the expression and release of inflammatory factors IL-1β, IL-6, and TNF-α during the foaming process of macrophages stimulated by PA, suggesting that piperine and its derivatives have anti-inflammatory effects. Yang Yanmin et al. also found that piperine and piperine have a significant effect on improving glucose and lipid metabolism disorders caused by insulin resistance.

[0024] 5. Cinnamon Cinnamon, called "Gabilayin-Halisu" in Mongolia, is the dry bark and branch bark of the cinnamon plant (Cinnamomum cassiaPresl) of the Lauraceae family.

[0025] 5.1 Chemical composition study The chemical composition of cinnamon is complex, mainly containing volatile components such as cinnamaldehyde, α-copaene, δ-cuberene, α-ylangolene, 1,4-Cadinadiene, α-muurolene, α-cuberol, o-methoxycinnamaldehyde, (+)-lucerne, polysaccharides, polyphenols, and flavonoids. It also contains other components such as cinnamic acid, cinnamic acid, syringic acid and choline.

[0026] 5.2 Pharmacological studies Cinnamaldehyde, an active ingredient in cinnamon, has multiple benefits, including anti-inflammatory, anti-tumor, wound healing, and antihypertensive and hypoglycemic properties. Studies have shown that cinnamaldehyde, trans-cinnamaldehyde, 2'-hydroxycinnamaldehyde, and 2'-benzoyloxycinnamaldehyde exhibit significant anti-inflammatory activity and are effective in treating inflammatory diseases such as neuritis, arthritis, sepsis, and atherosclerosis. The polysaccharides, flavonoids, and polyphenols in cinnamon are the primary components that exert antioxidant activity, and cinnamaldehyde, polysaccharides, and polyphenols also have hypoglycemic effects.

[0027] 5.2.1 Anti-tumor Cinnamaldehyde and cinnamon extracts can inhibit tumor cell proliferation, induce apoptosis, interfere with the tumor cell cycle, and inhibit epithelial-mesenchymal transition through multiple pathways, demonstrating promising anti-tumor activity against a variety of malignancies. Han Fuxin's research has shown that cinnamaldehyde reduces inflammation and apoptosis by inhibiting the IRAK4 / TAK1 signaling pathway, thereby alleviating neurological deficits in rats with traumatic brain injury. Liao Zhenggen et al. found that cinnamon volatile oil exhibited a significant inhibitory effect on breast cancer cell proliferation.

[0028] 5.2.2 Other functions In addition to inhibiting tumor cells, cinnamon's active ingredients also exhibit multiple pharmacological effects, including anti-inflammatory, hepatoprotective, and wound healing. Cinnamaldehyde promotes wound healing in diabetic rats by increasing wound healing rates, reducing levels of inflammatory factors such as IL-6 and TNF-α, increasing levels of VEGF and collagen, inhibiting inflammatory responses, and increasing angiogenesis and collagen synthesis. Cinnamaldehyde also alleviates high-glucose-induced ferroptosis, possibly by regulating iron metabolism and inhibiting iron-dependent lipid peroxidation. Cinnamaldehyde alleviates airway inflammation in COPD rats, possibly by inhibiting the SDF·1 / CXCR4 axis. It also reduces gastric mucosal damage in rats with Helicobacter pylori-induced gastritis and effectively reduces liver damage in NASH mice. This mechanism may be related to regulating the AMPK / SREBP1c pathway, inhibiting oxidative stress, and improving hepatic steatosis. Furthermore, cinnamon ethanol extract effectively improves benign prostatic hyperplasia.

[0029] 6. Safflower Safflower is called "Gurigumu" in Mongolian. It is the flower of the safflower (Carthamus tinctorius L.) of the Asteraceae family.

[61] .

[0030] 6.1 Chemical composition study Safflower contains flavonoids, alkaloids, polyacetylenes, lignans, sterols, organic acids and alkyl glycols. Ma Xinyi et al. separated and obtained benzyl-β-D-glucoside, benzyl-O-rutinoside, phenylethanol rutinoside, 3-(4-O-β-D-glucopyranosylphenyl) propionic acid methyl ester, carthamoside B5, junipediol A-8-O-β-D-glucopyranoside, 7-O-β-benzoylrutinoside, syringin, 4-allyl-2,6-dimethoxyphenyl glucoside, 2,6-dimethoxy-4-[(1E)-prop-2-enyl]phenyl-O-rutinoside, 4-O-β-D-glucopyranosyl trans-phenylacrylic acid, 4-O-β-D-glucopyranosyl cis-phenylacrylic acid, 1-(4-O-β-D-glucopyranosyl)benzoic acid, 4-(2-aminoethyl)phenyl-1-O-β-D-glucopyranosyl, (+) Fifteen aromatic glycosides were identified, including 1-candelilla resin phenol-3α-O-β-D-pyranoside. Alkaloids such as N1, N5, and N10-tri-p-coumaroylspermidine and N-cis-feruloyltyramine, terpenoids such as (-)-methyldihydrophaseate, plucheol A, and myrtin, and phenylpropanoids such as 4-ethoxycinnamic acid, ethyl cis-p-hydroxycinnamate, and ethyl trans-p-hydroxycinnamate were also identified in safflower. It also contains seven sesquiterpenoids, including (+)-4a,5-dimethyl-3-(prop-2-enyl)-octahydronaphthalene-2β,8a-diol [(+)-4a,5-dimethyl-3-(prop-1-en-2yl)-octahydronaphthalene-2β,8a-diol, 1], baimuxinic acid (2), baimuxinol (3), vetaspira-2(11),6-dien-14-al (4), baimuxinal (5), 10-epi-γ-eudesmol [(−)-10-epi-γ-eudesmol, 6], and 9β-hydroxyl-α-agarofuran (7).

[0031] 6.2 Pharmacological studies 6.2.1 Hepatoprotective Effect Safflower, a liver medicine, has hepatoprotective properties. Safflower water extract has preventive and therapeutic effects on alcoholic liver injury. Hydroxysafflor yellow A can alleviate inflammation and oxidative stress, inhibit pyroptosis, and thus improve non-alcoholic steatohepatitis. It also reduces hepatic ischemia-reperfusion injury by activating the Sirt1 / FOXO1 signaling pathway. Safflower extract can also exert antioxidant and anti-inflammatory effects by regulating the Keap1Nrf2 and STAT3 / NF-κB signaling pathways, alleviating alcoholic liver injury in mice.

[0032] 6.2.2 Cardiovascular system With the continuous deepening of research, safflower is increasingly widely used clinically in cardiovascular, gynecological, orthopedic, and dermatological fields, with significant therapeutic effects. Hydroxysafflor yellow A exerts cardioprotective effects through multiple signaling pathways and different therapeutic targets, such as reducing inflammatory activity, inhibiting apoptosis, regulating autophagy, improving vascular activity, and regulating angiogenesis and remodeling. Hydroxysafflor yellow A inhibits the oxidative stress response of hypoxia / reoxygenation cardiomyocytes by regulating miR-499 protein expression, inhibits cell apoptosis, promotes cell proliferation, and reduces myocardial damage. Safflower yellow also reduces pyroptosis and inflammatory damage in cardiomyocytes in rats with myocardial infarction by inhibiting the NF-κB / NLRP3 signaling pathway.

[0033] Safflower has a protective and therapeutic effect on acute blood stasis in rats by restoring platelet aggregation activity, improving coagulation, hemorheology, endothelial function, and inhibiting thrombosis. Hydroxysafflor yellow A significantly relaxes rat coronary arteries pre-contracted by KCl and U46619. Safflower polysaccharide can improve early brain damage in rats with subarachnoid hemorrhage by inhibiting the expression of NLRP3 and cleaved caspase 1. Safflower yellow can reduce hypoxic-ischemic brain injury and oxidative stress in neonatal rats, protecting brain tissue. Furthermore, safflower yellow can reduce Aβ deposition and microglial activation in the brain tissue of TREM2-silenced APP / PS1 mice. By inhibiting the TLR4-mediated NF-κB signaling pathway and reducing the release of inflammatory mediators, it can improve learning and memory in these TREM2-silenced APP / PS1 mice. Safflower yellow can alleviate neurological damage after spinal cord injury, and its mechanism may be related to inhibiting the caspase-3 signaling pathway. Safflower yellow pigment upregulates miR-140-5p, promotes IL-1β-induced autophagy in osteoarthritis chondrocytes, and reduces cell apoptosis and inflammatory factor secretion.

[0034] 6.2.3 Anti-tumor Studies have shown that NK cells and safflower polysaccharide have a synergistic effect on killing colon cancer cells in vitro. This mechanism of action may be related to safflower polysaccharide's regulation of NK cell TNF-α and IFN-γ secretion and the expression of activating receptors and activating ligands. Hydroxysafflor yellow A and safflower yellow inhibit TGF-β1-induced proliferation, invasion, and migration of ovarian cancer SKOV-3 cells. This mechanism of action may be related to suppressing intracellular reactive oxygen species (ROS) levels and attenuating epithelial-mesenchymal transition (EMT). Hydroxysafflor yellow A inhibits liver cancer cell proliferation, invasion, migration, and angiogenesis by regulating the p38MAPK and PI3K / Akt signaling pathways. It also inhibits the PI3K / AKT / mTOR signaling pathway, inducing autophagy and autophagosome formation in liver cancer cells. It also promotes liver cancer cell apoptosis by impairing lysosomal activity and blocking autophagic flux. Safflor yellow B (SYB) has a significant inhibitory effect on the proliferation of the liver cancer cell line HepG2, demonstrating its ability to inhibit liver cancer cell proliferation. SPS may inhibit the invasion and migration of melanoma cells by activating the Notch1 signaling pathway in macrophages, inducing their polarization toward the M1 type. Hydroxysafflor yellow A inhibits the proliferation and migration of colorectal cancer cells and improves their resistance to 5-FU chemotherapy.

[0035] 6.2.4 Lung protection Safflower yellow significantly improves lung function in rats with COPD and reduces myocardial damage in rats with polystyrene microsphere-induced pulmonary embolism. Hydroxysafflor yellow A (HSYA) may protect the lungs by inhibiting programmed necrosis. Hydroxysafflor yellow A reduces airway inflammation in asthmatic mice through the AMPK / NF-κB / NLRP3 signaling pathway. Hydroxysafflor yellow A effectively reduces airway inflammation in asthmatic guinea pigs, possibly by regulating the HMGB1 / TLR4 / NF-κB signaling pathway. Safflower extract has a potent antifibrotic effect in mice with bleomycin-induced systemic sclerosis and reduces COLI and α-SMA expression in mice. HSYA significantly improves right heart failure caused by pulmonary hypertension, significantly reducing pulmonary artery pressure, improving hemodynamics and right ventricular function, and inhibiting right ventricular remodeling, thereby improving right heart failure induced by pulmonary hypertension. Safflower yellow has the effect of alleviating paraquat-induced pulmonary fibrosis, providing a possible therapeutic drug for the clinical treatment of paraquat poisoning-induced pulmonary fibrosis. Safflower yellow pigment may inhibit PQ-induced pulmonary fibrosis by inhibiting the expression of TGF-β1 protein and α-SMA.

[0036] 6.2.5 Other functions Safflower not only protects against complications of diabetes, such as kidney damage and podocyte damage, but also prevents diseases such as tinnitus and Parkinson's disease. Safflower yellow has a protective effect on the kidney tissue of mice with streptozotocin-induced diabetic nephropathy and can also attenuate inflammatory damage and oxidative stress damage in diabetic nephropathy. Its mechanism may be related to the regulation of the PKC / Ras-RafMEK-ERK pathway. High-dose safflower yellow intervention can promote the healing of foot ulcers in diabetic mice by increasing angiogenesis, collagen formation, and cell proliferation, and reducing insulin resistance, inflammatory response, and cell apoptosis, thereby reducing high-glucose-induced podocyte damage in mice. In addition, hydroxysafflower yellow A can effectively prevent the occurrence of tinnitus in mice, improve motor dysfunction in the LPS-induced zebrafish Parkinson's disease model, and reduce dopaminergic neuron damage. Safflower glycosides have a protective effect on hydrogen peroxide-induced osteoblast damage.

[0037] 7. Agarwood Agarwood is called "Agaru" in Mongolia. It is the resinous wood of Aquilaria agallocha Lour.Roxb. or Aquilaria sinensis of the Thymelaeaceae family.

[61] .

[0038] 7.1 Chemical composition study The chemical components isolated from agarwood include monoterpenes, sesquiterpenes, diterpenes, triterpenes, sterols, flavonoids, chromones, phenolic acids, and aliphatic compounds. The chemical composition of agarwood is primarily composed of volatile oils, the main active components of which are sesquiterpenes and aromatic compounds (agarfuran-type, agarhelicane-type, eudesane-type, erimopane-type, guaiacane-type, cadinane-type, and prevanillane-type). Sesquiterpenes and chromones, the main components of agarwood, account for 25.6% and 52.0%, respectively. Agarwood tetraol is the main characteristic component of agarwood. LC / MS / MS analysis of agarwood also includes compounds such as sesquiterpenes, aromatic compounds, aliphatic alkanes, and fatty acid esters.

[0039] 7.2 Pharmacological studies 7.2.1 Cardiovascular system The active ingredients in agarwood have a protective effect on the cardiovascular system. Zingiberone, a characteristic terpenoid in mountain agarwood, has anti-myocardial ischemia effects. Agarwood volatile oil can treat angina pectoris through multiple biological processes, including targets such as ALB and CYP3A4, drug metabolism-mediated signaling pathways such as cytochrome P450 and calcium signaling, receptor signaling pathways, and metabolic processes.

[0040] 7.2.2 Nervous system In addition to its protective effects on the cardiovascular system, agarwood's active ingredients also have therapeutic effects on neurological disorders. Agarwood extracts ameliorate corticosterone-induced apoptosis in PC12 cells by regulating the Caspase-3 / PARP pathway, thereby protecting neurons. Agarwood volatile oil, agarwood powder, and agarwood thread incense all have significant anxiolytic and antidepressant effects. Agarwood powder and extracts also promote sleep.

[0041] 7.2.3 Other functions Total lignans from Aquilaria sinensis can significantly improve cerebral ischemia-reperfusion injury in mice and reduce oxidative stress. Brass from Aquilaria sinensis leaves has a protective effect against H2O2-induced oxidative stress injury in HepG2 cells. Whole Aquilaria sinensis has significant anti-inflammatory and anti-asthmatic effects, and its mechanism of action may be related to its anti-inflammatory and anti-apoptotic effects. Aquilaria sinensis extract inhibits taurocholic acid-induced apoptosis of gastric epithelial cells by regulating endoplasmic reticulum stress mediated by the PERK / eIF2α / CHOP signaling pathway, providing a potential drug for the treatment of bile reflux gastritis. The 2-(2-phenylethyl) chromone fraction of Aquilaria sinensis inhibits taurocholic acid-induced excessive apoptosis of gastric epithelial cells and has a good protective effect against bile acid-induced gastric mucosal damage. Aquilaria sinensis leaf ethanol extract has anti-inflammatory effects in mice. Aquilaria sinensis ethanol extract can significantly inhibit cisplatin-induced vomiting in pigeons.

[0042] 8. Clove Clove, called "Gao Le Du-Bao Ru" in Mongolia, is the flower bud of Syzygium aromaticum L.Merr.et Perry, a plant of the Myrtaceae family.

[0043] 8.1 Chemical composition study The chemical components of cloves mainly fall into two categories: volatile and non-volatile. Volatile components include sesquiterpenes, monoterpenes, diterpenes, and aromatic compounds, primarily including clove oil and eugenol. Non-volatile components include flavonoids, steroids, triterpenes, and tannins. Clove oil contains phenols (eugenol, eugenyl acetate, β-caryophyllene, α-humulene, eucalyptol, caryophyllene, and α-pyrantel), esters, olefins, monoterpenes, sesquiterpenes, aromatic compounds, and a small amount of small-molecule aliphatic compounds. Non-volatile components include flavonoids such as quercetin, kaempferol, isorhamnetin, gallic acid, kaempferol, luteolin, syringin, and myricetin; sterols such as oleanolic acid, ursolic acid, corosolic acid, and maslinic acid; and terpenes. The main active ingredients of cloves are eugenol (2-methoxy-4-(2-propenyl)phenol, β-caryophyllene, eugenol acetate and α-humulene), chavicol, α-ylanene, methyl salicylate and other ingredients.

[0044] 8.2 Pharmacological Studies 8.2.1 Anti-inflammatory and antibacterial Eugenol inhibits both the free growth of Candida albicans and the early stages of biofilm formation, demonstrating efficacy against vaginitis in ICR mice caused by C. albicans. Clove essential oil exhibits strong antibacterial activity, showing significant inhibitory effects against Staphylococcus aureus and Pseudomonas aeruginosa. Eugenol exhibits significant antibacterial activity against both standard and resistant strains of S. aureus, disrupting bacterial cell structure, altering cell membrane permeability and integrity, inhibiting BF formation, clearing mature BF, and suppressing the transcription and expression of bacterial BF and adhesion-invasion-related virulence factors. Syringin can suppress inflammation and pyroptosis by downregulating the NLRP3 / Caspase-1 signaling pathway, alleviating pulmonary edema and lung tissue damage in rats with sepsis.

[0045] 8.2.2 Anti-tumor Existing research indicates that clove active ingredients have therapeutic effects on the nervous system. Clove active components activate the caspase cascade and induce apoptosis in human colon cancer HCT116 cells, which is associated with inhibition of the PI3K / Akt / mTOR signaling pathway. Clove extracts exert anti-tumor effects against radioresistant esophageal cancer cells through mechanisms including induction of autophagy and promotion of apoptosis, cell cycle arrest, inhibition of cellular energy metabolism, and migration inhibition. Eugenol inhibits NF-κB, which in turn inhibits AGR2 expression, reducing the growth and proliferation of pancreatic cancer cells and making it a potential therapeutic agent for pancreatic cancer. Clove extracts induce apoptosis in pancreatic cancer cells Panc-1 and Panc-28, and inhibit their proliferation, colony formation, migration, and invasion. Eugenol may induce apoptosis in PC-12 cells by upregulating Bax expression, downregulating Bcl-2 expression, and activating Caspase-3. Clove active components exhibit strong anti-colon cancer activity, with HCT116 cells being the most sensitive.

[0046] 8.2.3 Hepatoprotective Effect Eugenol regulates hepatic lipid metabolism by activating the GLP-1-based gut-brain-liver axis, providing a new strategy for the treatment of NAFLD. Clove leaf extract may improve liver tissue damage in rats with acute pancreatitis by activating the SIRT1 / PGC-1α / NRF1 signaling pathway, reducing oxidative stress and inflammation. Eugenol improves non-alcoholic fatty liver disease by activating the AMPK / ACC / mTOR / p70S6k signaling pathway and inhibiting the expression of the lipogenesis transcription factor SREBP1 and its target genes. Syringic acid significantly improves intestinal barrier damage in cholestatic mice and promotes bile acid metabolism. The ethyl acetate fraction of Eugenia syringae has demonstrated efficacy in preventing and treating alcohol-induced gastric damage.

[0047] 8.2.4 Nervous system Modern research indicates that the active ingredients of clove have protective effects on the nervous system. Eugenol and methyl eugenol exhibit potent anxiolytic activity, acting by regulating the SIRT1-MAO-A signaling pathway, thereby affecting the metabolism of the monoamine neurotransmitter serotonin (5-HT) and the activity and gene expression of COMT, ALDH, and AR enzymes in the catecholamine metabolic pathway. Methyl eugenol can mitigate damage in HK-2 cells induced by H / R. Treatment with eugenol significantly reduces injury after middle cerebral artery occlusion in rats and improves neurological deficits.

[0048] 8.2.5 Cardioprotective Effects Eugenol can regulate myocardial energy metabolism disorders caused by myocardial ischemia-reperfusion injury through the AMPK / PGC-1α signaling pathway, thereby playing a protective role against myocardial ischemia-reperfusion injury. Syringaresinol may alleviate the arthritis symptoms of rats with type II collagen-induced arthritis by inhibiting the TLR4 / NF-κB signaling pathway. Eugenol can alleviate myocardial ischemia-reperfusion injury. Eugenol can improve isoproterenol-induced acute myocardial infarction in rats, and its mechanism may be related to the regulation of iNOS, inflammation and oxidative stress. Syringin can improve the structure and function of the intervertebral disc by inhibiting the expression of matrix metalloproteinases 3, 13 and increasing the expression of type II collagen and aggrecan, thereby preventing and slowing down the process of intervertebral disc degeneration.

[202] Eugenol can inhibit MIRI in rats, and its mechanism may be related to the regulation of HIF-1 signaling pathway.

[0049] 9. Nutmeg Nutmeg is called "Zadi" in Mongolian. It is the seed of Myristica fragrans Houtt., a plant of the Myristicaceae family.

[0050] 9.1 Chemical composition study Nutmeg contains a variety of chemical constituents, including lignans (dibenzylbutanes, arylnaphthalenes, tetrahydrofurans, benzofurans and 8-O-4'-type neolignans, diarylpropanes, non-cyclopropanol neolignans and others), volatile oils (phenylpropanoids, monoterpenes and sesquiterpenes), polyphenols (phenolic acids and flavonoids), and other components (diphenylalkanes and steroidal). The chemical constituents of the ethyl acetate fraction of a 70% acetone extract of nutmeg fruit were studied. Five diarylpropane compounds, including 1-(3',4'-dihydroxyphenyl)-3-(4''-methoxyphenyl)-propane, 1-(4'-hydroxy-3'-methoxyphenyl) -3-(2"-hy-droxy-4''-methoxyphenyl) -propane, horsfielenidine A, 1-(2'-hydroxy-4'-methoxyphenyl) -3-(3'',4''-methyl-enedioxyphenyl) -propan-2-ol and virolanol B, were isolated and identified.

[205] Nutmeg contains about 250 chemical components, including lignans, neolignans, diphenylalkanes, phenylpropanoids, and terpenes. Among them, camphene, elemene, myristicin, eugenol, isoelemene, isoeugenol, methoxyeugenol, pinene, sabinene, safrole, myristic acid, myristicin, and caffeic acid have biological activities.

[0051] 9.2 Pharmacological Studies Myristyl glutamate can inhibit neuronal damage by suppressing endogenous neurotoxins. Nutmeg volatile oil exhibits excellent antibacterial activity, with the inhibitory effect against five test bacteria ranked in the order of Staphylococcus aureus > Escherichia coli > Bacillus subtilis > Saccharomyces cerevisiae > Aspergillus flavus. Studies on brain tissue from rats with chronic cerebral hypoperfusion demonstrated that nutmeg extract inhibits neuronal damage by reducing phosphorylation of the PI3K / AKT / FoxO3a signaling pathway and enhancing the expression and content of clenbuterol in brain tissue, thereby improving learning and memory in rats. Nutmeg also exhibits anti-tumor effects, with myristicin inhibiting the growth of colon cancer cells in vitro. Myristyl glutamate promotes apoptosis in gastric cancer cells by inhibiting the PI3K / AKT signaling pathway and inducing the expression of BAX, Caspase-3, and Caspase-9 proteins.

[0052] 10. Ziziphus jujuba The Mongolian name of the Chinese jujube is "Zhu Ruhen-Shaosha". It is the fruit of the Anacardiaceae plant Choerospondias axillaris (Roxb. Burtt et Hill).

[0053] 10.1 Chemical composition study The medicinal part of the fruit of the jujube tree mainly contains phenolic acid components such as organic acids and flavonoids, as well as polysaccharides, multiple amino acids (including lysine, histidine and arginine), vitamins and inorganic elements such as potassium, sodium, iron, zinc, phosphorus, selenium, silicon, and trace elements.

[0054] 10.2 Pharmacological Studies Modern pharmacology suggests that Choerospondias australis has therapeutic effects on cardiovascular diseases. Choerospondias australis total flavonoids, the main active ingredient in Choerospondias australis, can counteract various experimental ventricular and atrial arrhythmias induced by calcium chloride, acetylcholine, aconitine, and valine, and improve arrhythmias caused by myocardial ischemia. Choerospondias australis extract and injection can significantly alleviate myocardial ischemia. Its main component, TFC, can significantly improve coronary circulation in dogs, increase myocardial blood flow and oxygen supply, reduce the size of myocardial infarction, and restore normal myocardial metabolism. Pharmacological studies have confirmed that TFC can significantly inhibit platelet aggregation, reduce various hemorheological parameters, increase blood flow velocity, and improve blood circulation and microcirculation. The presence of bioactive compounds in Choerospondias australis fruit may play an important role in inhibiting breast cancer cell proliferation. Choerospondias australis polysaccharides promote the growth of beneficial bacteria and restrict the development of harmful bacteria. Among them, acetic acid, n-butyric acid, isovaleric acid, and n-valeric acid are the main fatty acid components that significantly enhance CAP fermentation. Choerospondias australis polysaccharides can significantly regulate intestinal microbial metabolism. Choerospondias australis fruit can be used to reduce inflammation in inflammatory diseases such as rheumatoid arthritis.

[0055] 11. Bistort Bistort is the rhizome of Polygonum bistorta L., a plant of the Polygonaceae family.

[0056] 11.1 Chemical Composition Study Bistort contains volatile oils, organic acids, phenols, flavonoids, glycosides, terpenes, steroids, saponins, phenylpropanoids, alkaloids, and other compounds. Comprehensive research shows that bistort primarily contains volatile oils, organic acids, and phenols. Five of the most studied active ingredients are gallic acid, chlorogenic acid, catechins, rutin, and β-sitosterol.

[0057] 11.2 Pharmacological Studies Aqueous extracts of Polygonum multiflorum may treat ulcerative colitis by increasing the levels of butyric acid, isobutyric acid, valeric acid, and isovaleric acid in the intestine. HPLC fingerprint analysis of Polygonum multiflorum, combined with spectrum-activity analysis of its antioxidant activity, revealed that phenolic acids (6-O-galloylglucose, digalloylglucose, and chlorogenic acid) and flavonoids (catechin and epicatechin) are the main potential antioxidant components of Polygonum multiflorum. Both crude and refined Polygonum multiflorum total flavonoids exhibited inhibitory effects against Staphylococcus aureus and Escherichia coli.

[0058] 12. Licorice Licorice is called "Xiheri-Ubus" in Mongolian. It is the root and rhizome of the legume plants Glycyrrhiza uralensis Fisch., Glycyrrhiza uralensis, and Glycyrrhiza glabra.

[0059] 12.1 Chemical Composition Study Licorice contains a large amount of flavonoids (isoliquiritigenin, liquiritigenin, liquiritin, formononetin, quercetin, isoliquiritigenin, kaempferol, rutin, naringenin, isoliquiritigeninol, glycyrrhizinol, 6,8-diisopentenyl genistein, and luteolin), phenolic acid compounds (2,4-dihydroxybenzoic acid, di(2-ethylhexyl) phthalate, p-hydroxybenzoic acid, and ethyl anisate), triterpenes, coumarins, sterols, stilbenes, and amino acids. Its main chemical components include triterpenoid saponins (glycyrrhizic acid), flavonoids, and polysaccharides. Thirteen compounds were isolated from the alcohol extract of the aerial part of Glycyrrhiza uralensis, three of which were new compounds, including (2S)-6-[(Z)-3-hydroxymethyl-2-butenyl]-5, 7,3′-trihydroxy-4′-methoxy-dihydroflavone, (2S)-8-[(E)-3-hydroxymethyl-2-butenyl]-5, 7, 3′,5′-Tetrahydroxy-flavonoid, 5,7,3′,4′-tetrahydroxy-6-isopentenyl-flavonoid, and naringenin are flavonoids; α,α′-dihydro-5,4′-dihydroxy-3-acetoxy-2-isopentenylstilbene is a stilbene; 6-isopentenylquercetin is a flavonol; 6-isopentenylquercetin-3-methyl ether, quercetin-3,3′-dimethyl ether, euphorbia cerifera, and diosmetin are flavonoids; formononetin is an isoflavone; (10E,12Z,14E)-9,16-dicarbonyl-10,12,14-triene-octadecanoic acid and dibutyl phthalate are fatty acids.

[0060] 12.2 Pharmacological Studies By targeting inflammatory signaling pathways and reducing inflammatory cytokine levels, it has demonstrated protective effects against sepsis-related lung, heart, kidney, and liver damage, demonstrating promising research and application value. Licorice flavonoids significantly improved pulmonary ventilation in rats with acute septic lung injury, reduced systemic inflammatory responses, and reduced 48-hour postoperative mortality. Licorice polysaccharides are unique anti-inflammatory and immune components that can help the body resist disease. Glycyrrhizic acid preparations can counteract inflammatory damage by reducing oxidative stress, thereby reducing TGF-β1 expression, regulating TGF-β1 / Smads and p38 mitogen-activated protein kinase (MAPK) signaling pathways, promoting INF-γ secretion, inhibiting extracellular matrix remodeling, and improving lung function, thereby preventing and treating pulmonary fibrosis. Magnesium isoglycyrrhizate treatment of hepatitis patients has a significantly lower risk of hypokalemia than compound glycyrrhizin, and offers a superior safety profile. Licorice can promote the metabolism and excretion of Tripterygium wilfordii in the body. It may also be related to its ability to reduce the peak value and concentration of Tripterygium wilfordii's chemical components in the body, prolong its existence time in the body, and regulate the tissue distribution of Tripterygium wilfordii's chemical components in the body, thereby achieving the effect of reducing toxicity and increasing efficacy.

[0061] 13. Indian Yellow Tianzhuhuang, also known as "Hulusen-Zhugang" in Mongolia, is a blocky substance formed by the sap stored in the internodes of bamboos such as Bambusa textilisMcClure and Bamboo stalks of the Poaceae family, which condenses after drying.

[61] The main component of dahlia is silicate, along with 14 other amino acids: aspartic acid, threonine, serine, glutamic acid, glycine, alanine, valine, methionine, leucine, phenylalanine, lysine, histidine, arginine, and proline. Its pharmacological effects include improving memory, suppressing cough, removing phlegm, reducing fever, providing anti-inflammatory, sedative, anticonvulsant effects, protecting the cardiovascular and cerebrovascular system, and protecting the nerves.

[0062] 14. Costus Aucklandia lappa Dacne, also known as Ruda in Mongolia, is the root of Aucklandia lappa Dacne, a plant of the Asteraceae family.

[61] The main chemical components of costus root are volatile oils, including costus alcohol and costus acid. It also contains costus alkaloids, inulin, chrysantheminose, and steroids. Its pharmacological effects include protective effects on small intestinal damage, gastric ulcers, and gallbladder damage in rats, as well as on the respiratory and cardiovascular systems.

[0063] 15. White raisins White raisins, Mongolian name "ujum", are the dried mature fruits of grape (Vitis vinifera L.) of the Vitaceae family.

[0064] 16. Crab Crab, called "Naimoleji" in Mongolian, is the entire Chinese mitten crab (Eriocheir sinensis H. Milne-Edwards) of the family Crabidae.

[0065] Compared with the prior art, the present invention has the following beneficial technical effects: The Mongolian medicine tea provided by the invention has the effects of relieving cough, resolving phlegm and reducing swelling; and can be used for symptoms such as dizziness caused by Bada Gan Heyi, asthma, chronic bronchitis, indigestion, edema, dropsy and the like. BRIEF DESCRIPTION OF THE DRAWINGS

[0066] Figure 1 These are pathological sections of the mouse lungs, where A: normal group, B: model group, C: positive group, D: medium-dose group, E: high-dose group, and F: low-dose group.

[0067] Figure 2 This is an analysis chart of the energy metabolism test results of mice.

[0068] Figure 3 These are the results of an expectorant experiment in mice. DETAILED DESCRIPTION

[0069] The following will clearly and completely describe the technical solutions in the embodiments of the present invention in conjunction with the accompanying drawings. Obviously, the described embodiments are only part of the embodiments of the present invention, not all of the embodiments. Based on the embodiments of the present invention, all other embodiments obtained by ordinary technicians in this field without making creative efforts are within the scope of protection of the present invention.

[0070] Example 1 A Mongolian medicinal tea beverage is composed of the following Mongolian medicinal ingredients in parts by weight: 20g of Rhizoma Dioscoreae, 40g of pomegranate, 2.5g of white cardamom, 5g of Piper longum, 2.5g of cinnamon, 25g of safflower, 25g of agarwood, 25g of cloves, 20g of nutmeg, 30g of jujube, 24g of polygonum multiflorum, 16.5g of liquorice, 29g of rhizoma jasminoides, 20g of costus root, 15g of white raisins, and 10g of crab.

[0071] All the medicinal materials in the above embodiments are prepared from commercially available Chinese medicine slices.

[0072] After processing the above herbs according to their respective conventional methods, grind them into a coarse powder and prepare them into tea bags. Each bag should be 30g. Brew with boiling water. People with respiratory diseases should take one bag three times a day. After the symptoms of respiratory diseases have basically disappeared, the dosage can be reduced as appropriate. It can also be taken once a day during the peak season to prevent respiratory diseases.

[0073] Experimental example: 1. Construction of animal model: 1.1 Experimental Animals: 60 SPF-grade SD male rats were purchased and placed under SPF conditions for 3 days for adaptation before the experiment. The rats weighed 180-220 g.

[0074] 1.2 Animal model construction method The experimental rats were randomly divided into normal group, model group, low-dose group, medium-dose group, high-dose group and positive group, with 10 rats in each group. The rats in the model group, low-dose group, medium-dose group, high-dose group and positive group were intraperitoneally injected with monocrotaline (1% saline ethanol solution) + pyloric ligation to prepare the lung function abnormality disease model.

[0075] The drugs used in the three dose groups (low, medium, and high) were coarse powders made from the Mongolian medicine tea formula provided in Example 1. The dosages administered to rats in each group were as follows: Low-dose group: 2.5 g / kg Mongolian medicine granules dissolved in 0.4 mL normal saline were administered orally once a day for 7 consecutive days.

[0076] Medium-dose group: 5 g / kg Mongolian medicine granules dissolved in 0.4 mL normal saline were administered orally once a day for 7 consecutive days.

[0077] High-dose group: 10 g / kg Mongolian medicine granules dissolved in 0.4 mL normal saline were administered orally once a day for 7 consecutive days.

[0078] Positive group: 10 g / kg Feining granules dissolved in 0.4 mL normal saline were administered orally once a day for 7 consecutive days.

[0079] Normal group and model group: 0.4 mL normal saline was gavaged once a day for 7 consecutive days.

[0080] 1.3 Experimental results: 1.3.1 Lung pathology analysis The lung pathological sections showed that the model group had proliferation of smooth muscle cells in the lung vascular wall and thickening of the vascular wall compared with the normal group (see the results). Figure 1 ).

[0081] 1.3.2 Pulmonary function test Pulmonary function tests showed that there were significant differences in IC, ERV, FEV, PEF, MMEF, FEF and other indicators between the model group and the normal group (see Table 1 for the results).

[0082] Table 1: Results of rat lung function tests

[0083] Note: Compared with the normal group * P <0.05; 1.3.3 Energy metabolism testing The results of energy metabolism test showed that the CO2 emission of rats in the model group was significantly lower than that in the normal group, and the high, medium and low dose groups of the Mongolian medicine tea provided by the present invention were significantly higher than those in the model group (see the results in Figure 2 ).

[0084] 1.3.4 Cough and expectoration test The results of the rat cough test in an ammonia environment within 2 minutes showed that the cough times of the medium-dose and low-dose groups were significantly reduced compared with the normal group (see Table 2 for results). The results of the rat expectoration experiment showed that the absorbance of the alveolar lavage fluid in the positive group was significantly reduced compared with the normal group (P < 0.05), and the absorbance of the alveolar lavage fluid in the medium-dose group was significantly reduced (P < 0.005) (see Figure 3 ).

[0085] Table 2: Asthma Test Results

[0086] In summary, the Mongolian medicine tea of the present invention uses Zhaoshanbai, which has the effects of regulating body elements, nourishing, and reducing swelling, as the main medicine; and uses pomegranate, safflower, white cardamom, cinnamon, and longan as the assistant medicines, which assist "stomach fire" and distribute "essence" throughout the body, to achieve the effects of not causing excessive heat but also dispelling cold, regulating body elements, and reducing swelling. Agarwood, which has the effect of inhibiting "He Yi" and relieving asthma, costus root, which regulates the conflict between "He Yi" and "Qi Su", nutmeg and clove, which inhibit "He Yi", and zizyphus jujube, which dispels heart heat, as the auxiliary medicines, to achieve the effects of treating asthma and reducing swelling. Meanwhile, liquorice, rhizoma jasminoides, scutellariae, and white grape, which relieve asthma, as the auxiliary medicines, have the effect of relieving cough. This Mongolian medicine tea has the effects of relieving "He Yi", relieving cough, reducing phlegm, and reducing swelling; and can be used for symptoms such as dizziness caused by "Bada Gan He Yi", asthma, chronic bronchitis, indigestion, edema, edema, and dropsy.

[0087] It should be noted that, in this document, terms such as "comprises", "includes" or any other variations thereof are intended to cover non-exclusive inclusion, so that a process, method, article or apparatus that includes a series of elements includes not only those elements, but also other elements not explicitly listed, or also includes elements that are inherent to such process, method, article or apparatus.

[0088] While embodiments of the present invention have been shown and described, it will be appreciated by those skilled in the art that various changes, modifications, substitutions, and variations may be made to these embodiments without departing from the principles and spirit of the invention, and that the scope of the invention is defined by the appended claims and their equivalents.

Claims

1. A Mongolian medicinal tea for relieving respiratory system abnormalities, characterized in that: The Mongolian medicine tea is composed of the following Mongolian medicine ingredients: Zhaoshanbai, pomegranate, white cardamom, long pepper, cinnamon, safflower, agarwood, cloves, nutmeg, jujube, polygonum multiflorum, licorice, rhubarb, costus root, white raisins, and crab.

2. The Mongolian medicinal tea for relieving respiratory system abnormalities according to claim 1, characterized in that: The Mongolian medicine tea is composed of the following Mongolian medicine ingredients in parts by weight: 20g of Rhizoma Dioscoreae, 40g of pomegranate, 2.5g of white cardamom, 5g of Piper longum, 2.5g of cinnamon, 25g of safflower, 25g of agarwood, 25g of cloves, 20g of nutmeg, 30g of jujube, 24g of polygonum multiflorum, 16.5g of liquorice, 29g of rhizoma jasminoides, 20g of costus root, 15g of white raisins, and 10g of crab.

3. Use of the Mongolian medicinal tea according to any one of claims 1 or 2 in the preparation of a medicament for alleviating respiratory system abnormalities.

4. A method for preparing a Mongolian medicinal tea for relieving respiratory system abnormalities, characterized in that: include: The single Mongolian medicines provided in claim 2 are weighed according to the dosage of the formula, mixed evenly, ground into coarse powder, and packed into bags to make tea bags.

5. The method for preparing the Mongolian medicinal tea for relieving respiratory system abnormalities according to claim 4, wherein: The dosage of the tea bag is 30-50g / bag.

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