Low-frequency treatment circuit and sleep instrument

Abstract

The utility model discloses a low-frequency therapeutic circuit, which comprises a current output unit, the current output unit comprises a resistor R39, a resistor R33, a triode V1, a field effect transistor Q1, a field effect transistor Q2, a field effect transistor Q7, a field effect transistor Q8 and a current output interface J1, one end of the resistor R33 is connected with an MCU, the other end of the resistor R33 is connected with the base electrode of the triode V1, and the other end of the resistor R33 is connected with the base electrode of the triode V1. The collector electrode of the triode V1 is connected with the drain electrodes of the field effect transistors Q7 and Q8, the drain electrode of the field effect transistor Q1 is connected with the source electrode of the field effect transistor Q7, the drain electrode of the field effect transistor Q2 is connected with the source electrode of the field effect transistor Q8, and the drain electrodes of the field effect transistor Q1 and the field effect transistor Q2 are respectively connected with the current output interface J1. The sleep instrument comprising the low-frequency treatment circuit can improve the sleep quality through a micro-current stimulation therapy.

Description

Technical Field

[0001] The utility model relates to the technical field of sleep equipment, in particular to a low-frequency treatment circuit and a sleep instrument. Background Art

[0002] Modern life is fast-paced, and young people face daily pressures from society, family, and work, leading to insomnia becoming a common problem for many. In recent years, research on the mechanisms of insomnia has focused on the following main trends. Studies have shown that insomnia is often associated with dysfunction of the hypothalamic-pituitary-adrenal axis. Studies of healthy subjects have also linked insomnia to vagal tone, potentially contributing to its development. Decreased melatonin system function, the effects of inflammatory factors, disturbances in central neurotransmitters, and abnormalities in limbic-cortical circuitry may all contribute to insomnia. In recent years, cognitive-behavioral therapy for chronic insomnia has also gained increasing recognition. Research has demonstrated that the pathological mechanisms of chronic insomnia involve limbic-cortical circuitry, which is closely linked to the generation of negative emotions. This suggests that the cognitive mechanisms underlying negative emotions may be relevant to chronic insomnia.

[0003] Cranial electrotherapy stimulation (CES) is a non-invasive, non-pharmacological treatment for depression, anxiety, insomnia, and other disorders. CES involves delivering microscopic bioelectric currents with a uniquely designed waveform and frequency directly into the brain via the temporal region of the skull, commonly known as a brain reflex. CES therapy is a non-invasive, non-pharmacological treatment for anxiety, depression, and insomnia. It has been accepted by the US Food and Drug Administration as a proven and effective treatment, but it has not yet been included in treatment guidelines. CES therapy uses a stimulation frequency of 0.5 to 100 Hz and a current of 1 to 500 μA. It is recommended to use the therapy twice daily for 20 or 60 minutes for the first two weeks, and every two days thereafter, or as directed by a physician. This therapy was introduced to China in 2002 and is currently primarily promoted in psychiatric and psychological clinics, but has not yet been applied in sleep therapy.

[0004] Therefore, the inventor of the present invention urgently needs to conceive a new technology to improve the problem. Utility Model Content

[0005] The utility model aims to provide a low-frequency therapeutic circuit and a sleep instrument.

[0006] In order to solve the above technical problems, the technical solution of the utility model is:

[0007] A low-frequency treatment circuit includes: a current output unit, the current output unit including a resistor R29, a resistor R37, a transistor V5, a field-effect transistor Q1, a field-effect transistor Q2, a field-effect transistor Q5, a field-effect transistor Q7, a field-effect transistor Q8, and a current output interface J1, wherein one end of the resistor R37 is connected to the MCU, and the other end is connected to the base of the transistor V5, the collector of the transistor V5 is connected to the gate of the field-effect transistor Q5 after passing through the resistor R29, the source of the field-effect transistor Q5 is connected to the source of the field-effect transistor Q1 and the source of the field-effect transistor Q2, respectively, and the drains of the field-effect transistor Q1 and the field-effect transistor Q2 are connected to the field-effect transistor Q7, the field-effect transistor Q8, and the current output interface J1, respectively.

[0008] Preferably, it also includes a human body resistance detection unit, which includes an operational amplifier U1, a resistor R1, a resistor R3, a resistor R9, a capacitor C6, a capacitor C7, a Zener diode ZD1, and a magnetic bead inductor CZ3, wherein the sixth pin of the operational amplifier U1 is connected to the resistor R1, the resistor R3 and one end of the capacitor C7 respectively; the resistor R9, the capacitor C6 and the Zener diode ZD1 are connected in parallel, one end of the three is connected to the magnetic bead inductor CZ3, and the other end is grounded.

[0009] Preferably, the field effect transistor Q1, the field effect transistor Q2, and the field effect transistor Q5 are VB2355 field effect transistors.

[0010] Preferably, the field effect transistor Q7 and the field effect transistor Q8 are VB1330 field effect transistors.

[0011] Preferably, the transistor V5 is a MMBT3904 transistor.

[0012] Preferably, the magnetic bead inductor CZ3 is a CBW160808U301T magnetic bead inductor.

[0013] Preferably, the operational amplifier U1 is one of INA132, INA148, and INA149 operational amplifiers.

[0014] Preferably, the voltage stabilizing diode ZD1 is a BZT52C3V3 voltage stabilizing diode.

[0015] A sleep device comprises the low-frequency treatment circuit described above.

[0016] Preferably, it also includes a power supply module, a pulse group suppression module, a host computer and an input-output component, wherein the power supply module is respectively connected to the pulse group suppression module, the host computer and the low-frequency treatment circuit, and the low-frequency treatment circuit and the input-output component are both connected to the host computer.

[0017] By adopting the above technical solution, the present invention has at least the following beneficial effects:

[0018] The low-frequency therapeutic circuit and sleep instrument described in the utility model output low-intensity current to stimulate the brain, thereby improving sleep quality to a certain extent. BRIEF DESCRIPTION OF THE DRAWINGS

[0019] Figure 1a This is a circuit diagram of the current output unit described in the present utility model;

[0020] Figure 1b This is a circuit diagram of the current output unit described in the present utility model;

[0021] Figure 2a This is a circuit diagram of the human body resistance detection unit described in the present utility model;

[0022] Figure 2b This is a circuit diagram of the human body resistance detection unit described in the present utility model;

[0023] Figure 3 This is a waveform diagram of the stimulation current described in the present invention;

[0024] Figure 4 This is a structural diagram of the sleep instrument described in the present invention. DETAILED DESCRIPTION

[0025] The following will be combined with the drawings in the embodiments of the present invention to clearly and completely describe the technical solutions in the embodiments of the present invention. Obviously, the embodiments described are only part of the embodiments of the present invention, not all of the embodiments. Based on the embodiments of the present invention, all other embodiments obtained by ordinary technicians in this field without making creative efforts are within the scope of protection of the present invention.

[0026] like Figure 1a and Figure 1bAs shown, a low-frequency treatment circuit in accordance with the present invention includes: a current output unit, the current output unit including a resistor R33, a resistor R39, a transistor V1, a field effect transistor Q1, a field effect transistor Q2, a field effect transistor Q7, a field effect transistor Q8, and a current output interface J1, wherein one end of the resistor R33 is connected to the MCU (the model of the MCU is an ARMCortex series microprocessor, such as an ARMCortex-M4F, of course, it can also be other models, the utility model is not limited to this, and those skilled in the art should know it), and the other end is connected to the base of the transistor V1, the collector of the transistor V1 is connected to the drain of the field effect transistor Q7 and the field effect transistor Q8 respectively, and the drain of the field effect transistor Q1 and the field effect transistor Q2 is connected to the source of the field effect transistor Q7, the field effect transistor Q8 and the current output interface J1 respectively. The field effect transistors Q1, Q2, Q7, and Q8 all work in a switching state, and their source-drain voltage drop is almost 0V when turned on. Therefore, the magnitude of the current flowing through the current output interface is determined by the collector current of the transistor V1. The transistor V1 operates in the current amplification region, and its current is determined by the base current. Figure 1b As shown, the voltage output by the MCU to the resistor R33 determines the base current of the transistor, that is, determines the current flowing through the load. The resistor R39 can not only ensure the load size, but also ensure that the entire circuit does not work when the load is short-circuited.

[0027] Preferably, if Figure 2a As shown, it also includes a human body resistance detection unit, which includes an operational amplifier U1, a resistor R1, a resistor R3, a resistor R9, a capacitor C6, a capacitor C7, a voltage-stabilizing diode ZD1, and a magnetic bead inductor CZ3, wherein the sixth pin of the operational amplifier U1 is connected to the resistor R1, the resistor R3, and one end of the capacitor C7 respectively; the resistor R9, the capacitor C6, and the voltage-stabilizing diode ZD1 are connected in parallel, one end of the three is connected to the magnetic bead inductor CZ3, and the other end is grounded. In order to establish an effective therapeutic current output, the utility model will automatically perform a human skin resistance value test after the microcurrent stimulation environment is turned on. The utility model preferably adopts a dual-channel interface, Figure 2b As shown, microcurrent stimulation can be performed on multiple people at the same time.

[0028] Preferably, the field effect transistor Q1, the field effect transistor Q2, and the field effect transistor Q5 are VB2355 field effect transistors.

[0029] Preferably, the field effect transistor Q7 and the field effect transistor Q8 are VB1330 field effect transistors.

[0030] Preferably, the transistor V5 is a MMBT3904 transistor.

[0031] Preferably, the magnetic bead inductor CZ3 is a CBW160808U301T magnetic bead inductor.

[0032] Preferably, the operational amplifier U1 is one of INA132, INA148, and INA149 operational amplifiers.

[0033] Preferably, the voltage stabilizing diode ZD1 is a BZT52C3V3 voltage stabilizing diode.

[0034] The utility model outputs a current of 0.1Hz through the above circuit design, and the maximum current does not exceed a small current of 500uA. The waveform of the stimulation current is as follows Figure 3 As shown; the current acts on the patient's earlobe and other parts through the output electrode, stimulating the brain with low-intensity trace current, affecting the human brain wave activity, significantly enhancing the power of the alpha wave (tranquil state) of brain activity, achieving the effect of calming the mind, and prompting the brain to secrete a series of neurotransmitters and hormones that are closely related to diseases such as anxiety, depression, and insomnia, thereby achieving auxiliary treatment of these diseases.

[0035] A sleep device comprises the low-frequency treatment circuit described above.

[0036] Preferably, it also includes a power supply module, a pulse group suppression module, a host computer and an input-output component, wherein the power supply module is respectively connected to the pulse group suppression module, the host computer and the low-frequency treatment circuit, and the low-frequency treatment circuit and the input-output component are both connected to the host computer.

[0037] Preferably, the input and output components include but are not limited to a display, a printer, a mouse, a keyboard, and an IPAD.

[0038] The sleep monitor is powered by mains power and utilizes a pulse suppression module to mitigate the transient pulse trains of several thousand volts generated at the disconnection point of an inductive load. This interference can be conducted (and partially radiated) through the power or signal lines into the measurement and control devices, rendering the devices' digital circuits inoperable. The power supply module provides a stable power supply, powering both the host computer and the low-frequency therapy circuitry. The computer software / hardware module provides the necessary hardware and software operating environment for system operation. The software includes digitized versions of five commonly used clinical psychological assessment scales related to sleep quality, including the Pittsburgh Sleepiness Scale (PSQI), the Epworth Sleepiness Scale (ESS), the Athens Insomnia Scale (AIS), the Sleep Hygiene Awareness and Practices Scale (SHAP), and the Beliefs and Attitudes About Sleep (DBAS). The computer software / hardware module communicates effectively and in real time with the low-frequency therapy system module via a USB interface.

[0039] In this program, under the guidance of medical staff, subjects can use a mouse, keyboard, IPAD, etc. as external input devices to complete the designated assessment form. The assessment system can automatically complete the scoring and evaluation. After saving the information, it can be output using external output devices such as printers as needed. Based on the evaluation results, medical staff can use conductive rubber ear clips and electrode wires to connect the output end of the device / subject to establish an effective microcurrent stimulation environment for treatment. The dual-channel interface allows for multiple people to receive different microcurrent stimulation treatments at the same time. The treatment cycle and treatment intensity can be carried out according to the doctor's instructions, and the current intensity can be adjusted by level.

[0040] It should be noted that, in this document, relational terms such as "first" and "second" are used only to distinguish one entity or operation from another entity or operation, and do not necessarily require or imply any actual relationship or order between these entities or operations. Moreover, the terms "comprises," "comprising," or any other variations thereof are intended to cover non-exclusive inclusion, so that a process, method, article, or device comprising a series of elements includes not only those elements, but also other elements not explicitly listed, or elements inherent to such process, method, article, or device. In the absence of further limitations, an element defined by the phrase "comprising a ..." does not exclude the presence of other identical elements in the process, method, article, or device comprising the element.

[0041] The above description of the disclosed embodiments will enable one skilled in the art to implement or use the present invention. Various modifications to these embodiments will be readily apparent to one skilled in the art, and the general principles defined herein may be implemented in other embodiments without departing from the spirit or scope of the present invention. Therefore, the present invention is not limited to the embodiments shown herein but is intended to conform to the widest scope consistent with the principles and novel features disclosed herein.

Claims

1. A low-frequency treatment circuit, characterized in that: include: A current output unit includes a resistor R29, a resistor R37, a transistor V5, a field-effect transistor Q1, a field-effect transistor Q2, a field-effect transistor Q5, a field-effect transistor Q7, a field-effect transistor Q8, and a current output interface J1, wherein one end of the resistor R37 is connected to the MCU, and the other end is connected to the base of the transistor V5, the collector of the transistor V5 is connected to the gate of the field-effect transistor Q5 after passing through the resistor R29, the source of the field-effect transistor Q5 is respectively connected to the source of the field-effect transistor Q1 and the source of the field-effect transistor Q2, and the drains of the field-effect transistor Q1 and the field-effect transistor Q2 are respectively connected to the field-effect transistor Q7, the field-effect transistor Q8, and the current output interface J1.

2. The low-frequency therapeutic circuit according to claim 1, wherein: It also includes a human body resistance detection unit, which includes an operational amplifier U1, a resistor R1, a resistor R3, a resistor R9, a capacitor C6, a capacitor C7, a Zener diode ZD1, and a magnetic bead inductor CZ3, wherein the sixth pin of the operational amplifier U1 is connected to the resistor R1, the resistor R3 and one end of the capacitor C7 respectively; the resistor R9, the capacitor C6, and the Zener diode ZD1 are connected in parallel, one end of the three is connected to the magnetic bead inductor CZ3, and the other end is grounded.

3. The low-frequency therapeutic circuit according to claim 1, wherein: The field effect transistor Q1, the field effect transistor Q2, and the field effect transistor Q5 are VB2355 field effect transistors.

4. The low-frequency therapeutic circuit according to claim 1, wherein: The field effect transistor Q7 and the field effect transistor Q8 are VB1330 field effect transistors.

5. The low-frequency therapeutic circuit according to claim 1, wherein: The transistor V5 is a MMBT3904 transistor.

6. The low-frequency therapeutic circuit according to claim 2, wherein: The magnetic bead inductor CZ3 is a CBW160808U301T magnetic bead inductor.

7. The low-frequency therapeutic circuit according to claim 2, wherein: The operational amplifier U1 is one of INA132, INA148, and INA149 operational amplifiers.

8. The low-frequency therapeutic circuit according to claim 2, wherein: The voltage stabilizing diode ZD1 is a BZT52C3V3 voltage stabilizing diode.

9. A sleep instrument, characterized in that: The invention comprises the low-frequency therapeutic circuit described in any one of claims 1-8.

10. The sleep device according to claim 9, wherein: It also includes a power supply module, a pulse group suppression module, a host computer and input and output components, wherein the power supply module is respectively connected to the pulse group suppression module, the host computer and the low-frequency treatment circuit, and the low-frequency treatment circuit and the input and output components are both connected to the host computer.

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