A magnetic condylar cartilage osteogenic dysfunction prevention and correction device

Abstract

The present invention designs a magnetic condylar cartilage osteoblast dysfunction prevention and correction device, including a magnetic belt ring and a magnetic traction block, the magnetic belt ring is connected to the magnetic traction block by a fixed steel wire; the magnetic belt ring includes a rounded rectangular steel ring, a fixed magnetic block, a sliding magnetic block installation slot and a U-shaped slide rail, the magnetic traction block includes a traction slot and a straight slide rail, the number of the traction slots is two and they are arranged vertically in parallel, and a U-shaped slider is set at the front and rear ends of each traction slot. After the invention is installed in the patient's mouth, the static magnetic field can promote the differentiation of condylar osteoblasts and prevent condylar cartilage osteoblast dysfunction, and the principle of mutual repulsion of the same magnetic poles of permanent magnets can correct the expansion deformity. The sliding magnetic block installation slot and the traction slot can slide slightly with the magnetic field when the permanent magnet is installed for correction, and a scale is set on the straight slide rail, which is convenient for doctors to record the offset to judge the correction effect and determine the correction plan.

Description

Technical Field

[0001] The present invention relates to the technical field of oral medical instruments, and in particular to a magnetic condylar cartilage osteogenic dysfunction prevention and correction device. Background Art

[0002] The mandibular condylar cartilage is a secondary growth and development center of the maxillofacial region. It participates in the formation and movement of the temporomandibular joint and the maintenance of the lower 1 / 3 of the face height. It is related to joint movement and the composition of the oral and maxillofacial system. Osteogenesis is one of the important physiological functions of the condylar cartilage. Osteogenesis disorders can cause severe maxillofacial deformities such as micrognathia and overbite, and secondary temporomandibular joint dysfunction, which seriously affects the patient's facial beauty and quality of life. The prevention and correction of clinical maxillary deformities are closely related to condylar cartilage osteogenesis and jaw development. Condylar cartilage osteogenesis belongs to endochondral osteogenesis. During embryonic development, the development of condylar cartilage originates from Meckel's cartilage. During the development of the mandible in the embryonic period, the mandibular arch mesenchyme located on the side of Meckel's cartilage forms the mandibular body around this center through intramembranous osteogenesis, and the condylar cartilage and the cartilage of the middle line of the anterior mandible undergo endochondral osteogenesis to form the condylar and mandibular symphysis. During the process of condylar endochondral ossification in vivo, the cartilage formation template is gradually replaced by bone tissue. During this period, cartilage and bone formation are closely coordinated, and the terminal mineralization of chondrocytes is the key to osteogenesis. The terminal mineralization of condylar joint chondrocytes is very important for endochondral ossification because it forms a biological buffer zone between the non-mineralized cartilage layer on the joint surface and the underlying mineralized bone layer to cope with mechanical stress. It is the key to combining cartilage matrix degradation and osteogenesis. The mineralization of chondrocytes provides a scaffold for the formation of trabeculae and can induce the formation of cortical bone and periosteum. It is a key effector matrix that closely links cartilage and osteogenesis.

[0003] According to previous literature reports, chondrocytes cultured and expanded in vitro can undergo terminal mineralization under appropriate culture conditions. Bone morphogenetic protein (BMP) signaling is a classic signaling pathway that has been widely studied to regulate the terminal mineralization of chondrocytes. BMP is an effector protein that is widely present in various tissues of the body and is closely related to the occurrence of bone and cartilage tissues in the embryonic period of vertebrates. It is also a key factor in endochondral osteogenesis after birth. Bone morphogenetic protein 2 (BMP2) is currently a widely studied BMP ligand. Studies have shown that BMP2 can induce osteogenic differentiation of mesenchymal stem cells and can induce chondrogenic differentiation of stem cells, and is deeply involved in the process of chondrocyte mineralization and osteogenesis. Studies have found that leucine-rich fibronectin transmembrane protein 3 (FLRT3) is closely related to osteogenesis. FLRT3 is a newly discovered protein that regulates the migration and differentiation of neural crest cells. It is widely expressed in human tissues and plays an important role in the regulation of the growth and development of neural tissues, embryonic ventral and limb buds. During craniofacial development, FLRT3 is expressed in the brain and eye regions. FLRT3 is also found to be localized in the mandibular blastema region and overlaps with the expression of bone morphogenetic protein 4, suggesting that FLRT3 may be involved in the growth and development of the mandible. Although there have been a certain amount of reports on the effect of magnetic field loading on the process of cartilage differentiation, such as cells from different tissues (such as stem cells) and different experimental model carriers (such as rabbit animal models and fiber scaffold models), it may be possible to partially explain the effect of magnetic fields on the chondrocyte differentiation process in time and space during the physiological process of endochondral ossification from a general and macroscopic perspective, but the effect of magnetic fields on the mineralization and osteogenesis of mandibular condylar cartilage and the exact mechanism have not yet been fully elucidated. The present invention studies the mechanism by which the magnetic field affects the expression of BMP2 and FLRT3 and then affects osteogenic differentiation, and designs a magnetic condylar cartilage osteogenic dysfunction prevention and correction device. Summary of the invention

[0004] In order to solve the above problems, the present invention designs a magnetic condylar cartilage osteogenic dysfunction prevention and correction device.

[0005] In order to solve the above technical problems and achieve the above technical effects, the present invention is implemented through the following technical contents:

[0006] A magnetic condylar cartilage osteogenic dysfunction prevention and correction device comprises a magnetic belt ring and a magnetic traction block, wherein the magnetic belt ring is connected to the magnetic traction block via a fixed steel wire;

[0007] The magnetic belt ring includes a rounded rectangular steel ring, a fixed magnetic block, a sliding magnetic block installation groove and a U-shaped slide rail, wherein the U-shaped slide rail is fixed on the two outer side walls of the steel ring, the fixed magnetic block is arranged on the outer wall of the steel ring inside the U-shaped slide rail, and the sliding magnetic block installation groove is installed in the U-shaped slide rail through a slider;

[0008] The magnetic traction block comprises a traction groove and a straight-line slide rail. The traction grooves are two in number and arranged vertically in parallel. A U-shaped slider is provided at the front and rear ends of each traction groove. The two traction grooves are slidably mounted on the straight-line slide rail via the U-shaped slider.

[0009] The fixed steel wires are respectively fixed on the ends of the I-shaped slide rail and the U-shaped slide rail to connect the magnetic belt ring and the magnetic traction block.

[0010] Furthermore, the fixed magnetic block is a permanent magnet.

[0011] Furthermore, permanent magnets with different magnetic forces can be detachably installed in the sliding magnet installation groove and the traction groove, and the permanent magnets are fixed in the sliding magnet installation groove and the traction groove by steel wires.

[0012] Furthermore, the straight-shaped slide rail is marked with scales.

[0013] Another object of the present invention is to provide a method for using a magnetic condylar cartilage osteogenic dysfunction prevention and correction device, which specifically comprises the following steps:

[0014] Step 1: Make a magnetic belt ring of appropriate size according to the patient's first premolar and second molar, and connect the magnetic belt ring and magnetic traction block through a fixed steel wire according to the shape and size of the patient's oral cavity.

[0015] Step 2: Select a permanent magnet block with a magnetic force that is suitable for the patient's correction needs and fix it in the sliding magnet block installation slot and the traction slot through a steel wire.

[0016] Step three, place the magnetic traction block on the patient's upper jaw, and fix the magnetic belt ring on the patient's first premolar and second molar. When preventing condylar cartilage osteoblastic dysfunction, the static magnetic field generated by the permanent magnet block promotes the expression of FLRT3 and BMP2 proteins in condylar chondrocytes, promotes the differentiation of condylar osteoblasts, and prevents condylar cartilage osteoblastic dysfunction. When correcting oral deformities caused by condylar cartilage osteoblastic dysfunction, according to the principle that the same poles of permanent magnets repel each other, choose to install and fix permanent magnets with the same poles as the fixed magnet in the sliding magnet installation slot and the traction slot to correct the expansion deformity by repelling the left and right.

[0017] Step 4: Regularly observe the displacement of the traction groove on the I-shaped slide rail according to the patient's correction condition, and replace the sliding magnet installation groove and the permanent magnet block in the traction groove according to the correction requirements.

[0018] The beneficial effects of the present invention are:

[0019] (1) After being installed in the patient's oral cavity, the present invention can prevent condylar cartilage osteogenic dysfunction in advance. The static magnetic field generated by the permanent magnet block can promote the expression of FLRT3 and BMP2 proteins in condylar cartilage cells, and the expression of FLRT3 and BMP2 proteins can promote the differentiation of condylar osteoblasts and prevent condylar cartilage osteogenic dysfunction.

[0020] (2) After being installed in the patient's mouth, the present invention can correct oral deformities caused by condylar cartilage osteogenic dysfunction. Based on the principle that the same magnetic poles of permanent magnets repel each other, permanent magnets with the same magnetic poles as the fixed magnets are installed and fixed in the sliding magnet installation slot and the traction slot to correct the arch expansion deformity by repelling left and right.

[0021] (3) The sliding magnet block installation slot and the traction slot designed by the present invention can replace permanent magnets with different magnetic forces, which is convenient for adjusting the magnetic force scheme according to the correction effect of the patient, with high flexibility and simple and convenient operation. When replacing the permanent magnet in the sliding magnet block installation slot and the traction slot, it is only necessary to cut the steel wire, use a magnet with the opposite magnetic pole to suck it out, and then re-insert the new permanent magnet and fix it with the steel wire. It is simple and convenient, and there is no need to remove the belt ring and the magnetic traction block.

[0022] (4) The sliding magnet installation groove and the traction groove of the present invention can slide slightly with the magnetic field when the permanent magnet is installed for correction. Compared with the traditional rigid fixed structure, the correction process is gentler and the pain caused by oral correction is reduced. A scale is set on the straight slide rail, which is convenient for doctors to record the offset of the traction groove on the straight slide rail to judge the correction effect and determine the correction plan, and quantify the correction data to facilitate data statistical analysis.

[0023] (5) The present invention has a simple structure, is made of easily available materials with low cost, has both preventive and corrective effects, is suitable for popularization and use, and can reduce the economic burden on patients. BRIEF DESCRIPTION OF THE DRAWINGS

[0024] In order to more clearly illustrate the technical solutions of the embodiments of the present invention, the accompanying drawings required for describing the embodiments will be briefly introduced below. Obviously, the accompanying drawings described below are only some embodiments of the present invention. For ordinary technicians in this field, other accompanying drawings can be obtained based on these accompanying drawings without paying creative work.

[0025] Figure 1 It is a schematic diagram of the overall structure of a device for preventing and correcting condylar cartilage osteogenic dysfunction;

[0026] Figure 2 It is a schematic diagram of the installation structure of a device for preventing and correcting condylar cartilage osteogenic dysfunction;

[0027] Figure 3It is a schematic diagram of the structure of a magnetic belt ring of a condylar cartilage osteogenic dysfunction prevention and correction device;

[0028] Figure 4 It is a schematic diagram of the structure of the magnetic traction block of the condylar cartilage osteogenesis dysfunction prevention and correction device.

[0029] In the accompanying drawings, the components represented by the reference numerals are listed as follows:

[0030] 1-magnetic belt ring, 11-steel ring, 12-fixed magnetic block, 13-U-shaped slide rail, 14-sliding magnetic block installation groove, 2-magnetic traction block, 21-traction groove, 22-straight slide rail, 23-U-shaped slider, 3-fixed steel wire. DETAILED DESCRIPTION

[0031] The following will be combined with the drawings in the embodiments of the present invention to clearly and completely describe the technical solutions in the embodiments of the present invention. Obviously, the described embodiments are only part of the embodiments of the present invention, not all of the embodiments. Based on the embodiments of the present invention, all other embodiments obtained by ordinary technicians in this field without creative work are within the scope of protection of the present invention.

[0032] Example

[0033] See also Figure 1-2As shown, a magnetic condylar cartilage osteogenic dysfunction prevention and correction device comprises a magnetic belt ring 1 fixed to a first premolar and a second molar and a magnetic traction block 2 generating a repulsive magnetic field, wherein the magnetic belt ring 1 is connected to the magnetic traction block 2 by a fixed steel wire 3, so that the magnetic traction block 2 is positioned on the upper jaw; the magnetic belt ring 1 comprises a rounded rectangular steel ring 11 fixed to the teeth, a fixed magnetic block 12 generating an expansion force, a sliding magnetic block mounting groove 14 for fixing a permanent magnet, and a U-shaped slide rail 13, wherein the fixed magnetic block 12 is a permanent magnet that can repel each other with other magnets to generate an expansion force, and the U-shaped slide rail 13 is fixed on the two outer side walls of the steel ring 11, the fixed magnetic block 12 is arranged on the outer wall of the steel ring 11 in the U-shaped slide rail 13, and the sliding magnetic block mounting groove 14 is installed in the U-shaped slide rail 13 through a slider; the magnetic traction block 2 comprises a fixed permanent magnet The traction groove 21 and the straight-line slide rail 22 capable of judging the offset are marked with scales. During the correction process, the correction situation can be analyzed by observing the offset of the traction groove 21 on the scale. There are two traction grooves 21 arranged vertically and in parallel, which can generate repulsive force on both sides of the oral cavity. A U-shaped slider 23 is arranged at the front and rear ends of each traction groove 21. The two traction grooves 21 are slidably installed on the straight-line slide rail 22 through the U-shaped slider 23, and can slide freely along the straight-line slide rail 22; the fixed steel wire 3 is respectively fixed on the end of the straight-line slide rail 22 and the U-shaped slide rail 13 to connect the magnetic belt ring 1 and the magnetic traction block 2. The sliding magnetic block mounting groove 14 and the traction groove 21 can be detachably installed with permanent magnets of different magnetic forces. The permanent magnet is fixed in the sliding magnetic block mounting groove 14 and the traction groove 21 by steel wire, and the steel wire can be cut when replaced.

[0034] The application of the present invention when used is:

[0035] When preventing condylar cartilage osteoblastic dysfunction: select a permanent magnet block with a magnetic force size suitable for the patient's correction needs and fix it in the sliding magnet block installation slot 14 and the traction slot 21 through a steel wire, place the magnetic traction block 2 on the patient's upper jaw, and fix the magnetic belt ring 1 on the patient's first premolar and second molar. The static magnetic field generated by the magnet block promotes the expression of FLRT3 and BMP2 proteins in condylar chondrocytes, promotes the differentiation of condylar osteoblasts, and prevents condylar cartilage osteoblastic dysfunction.

[0036] When correcting oral deformities caused by condylar cartilage osteogenic dysfunction: select a permanent magnet block with a magnetic force size suitable for the patient's correction needs and fix it in the sliding magnet block installation slot 14 and the traction slot 21 through a steel wire, place the magnetic traction block 2 on the patient's upper jaw, and fix the magnetic belt ring 1 on the patient's first premolar and second molar. According to the principle that the same magnetic poles of permanent magnets repel each other, select a permanent magnet with the same magnetic pole as the fixed magnet 12 to be installed and fixed in the sliding magnet block installation slot 14 and the traction slot 21 to correct the dilatation deformity by repelling left and right. Regularly observe the offset of the traction slot 21 on the straight rail 22 according to the patient's correction situation, and replace the permanent magnet blocks in the sliding magnet block installation slot 14 and the traction slot 21 according to the correction requirements.

[0037] The present invention uses an animal experiment on the effect of static magnetic field on the mineralization of SD rat condylar chondrocytes to study the mechanism by which static magnetic field affects the expression of BMP2 and FLRT3 proteins and thus affects the osteogenic differentiation of the condyle:

[0038] 1. Establishment of a terminal mineralization induction model of condylar chondrocytes in SD neonatal rats

[0039] In this part of the experiment, we first simulated the terminal mineralization of condylar chondrocytes in vitro using a mineralized osteogenic induction medium containing dexamethasone, sodium β-glycerophosphate, and ascorbic acid, detected the formation of osteogenic calcium nodules using Alizarin red staining, and detected the localization and expression of terminal mineralization-related factors in the BMP (bone morphogenetic protein) signaling pathway using immunofluorescence analysis.

[0040] In this study, SD rat condylar chondrocytes were used to simulate the in vitro model of the terminal mineralization stage of condylar endochondral ossification chondrocytes. It was found that the mineralized nodules increased significantly, indicating the mineralization of the cartilage matrix. Immunofluorescence staining results showed that the expression of cartilage mineralization-related factors BMP2 (bone morphogenetic protein 2) and RUNX2 (osteogenesis-specific factor) was significantly increased, indicating that the mineralization model was successfully established. Although there is a lack of real vascular invasion in vivo and the interaction between various cells during endochondral ossification in vivo, since the condylar cartilage has a special chondrocyte stratification, the mineralized matrix may be the result of calcified osteoblasts and mineralization induction fluid promoting chondrocyte mineralization.

[0041] The conclusion obtained from this experiment is that after osteogenic induction treatment, the mature mineralization-related BMP signaling pathway in chondrocytes is activated, which promotes the process of terminal mineralization of chondrocytes.

[0042] 2. Study on the role of FLRT3 (leucine-rich fibronectin transmembrane protein 3) and BMP pathway in chondrocyte mineralization

[0043] The BMP signaling pathway is closely related to the development of craniofacial bones, participates in regulating the maturation and differentiation of chondrocytes, and is the key to the formation of osteoblast precursor cells derived from cranial neural crest cells. According to recent research reports on FLRT3, it is mainly related to osteogenesis and neuronal axonogenesis, and participates in the maintenance of cell homeostasis by regulating cell migration. Whole genome sequencing found that FLRT3 is a regulatory factor for articular cartilage, and embryo experiments found that FLRT3 is located in the mandibular primordium. Therefore, we speculate that FLRT3 may be involved in the regulation of condylar cartilage, the secondary growth and development center of the maxillofacial region.

[0044] In this experiment, the terminal mineralization of condylar chondrocytes was simulated in vitro by using the first part of osteogenic induction culture medium, and the localization and expression of FLRT3 were detected by immunofluorescence analysis. Based on this differentiation induction model, Western blotting was used to observe the expression trends of cell mineralization-related proteins such as FLRT3 and BMP2 at three time points: d3, d8, and d15. The expression characteristics of FLRT3 at different stages of cell differentiation and its relationship with cartilage mineralization were analyzed, and the molecular characteristics of condylar chondrocyte mineralization in SD neonatal rats were preliminarily explored.

[0045] The condylar chondrocytes were induced, and the expression and localization of FLRT3 were detected by immunofluorescence. The results of immunofluorescence staining showed that FLRT3 was mainly located in the cytoplasm of cartilage cells. When the condylar chondrocytes were induced, the expression of FLRT3 in the chondrocytes was obviously increased. Western blotting was used to analyze the relative expression of FLRTR3 protein in the MCC (condylar chondrocytes) group and the MCC+OIM (condylar chondrocytes+osteoblastic mineralization induction medium) group of the mandibular condylar cartilage of SD neonates at three time points: 3d, 8d, and 15d. The results showed that the protein expression of FLRT3 gradually increased with the extension of time. When the chondrocytes were induced, the induction fluid could significantly promote the protein expression of FLRT3. The cells of the first generation were fused to 70% for terminal differentiation and mineralization induction. The expression of BMP2 / Runx2 signaling pathway was detected by Western blotting on d3, d8 and d15. It was found that the protein expression levels of BMP2 and Runx2 in the MCC mineralization induction group were higher than those in the MCC group on d3. The expression activity of the mineralization induction group gradually decreased from d3 to d8, and the transduction activity of the BMP2 signaling pathway was negatively correlated with the expression of FLRT3, indicating that in the mineralization microenvironment, the BMP2 signaling pathway was activated on d3, and the mineralization induction solution promoted the terminal differentiation of chondrocytes and matrix mineralization.

[0046] Immunofluorescence results showed that FLRT3 was mainly expressed in the cytoplasm of chondrocytes, and FLRT3 was enhanced after cartilage mineralization induction treatment, suggesting that FLRT3 has a certain relationship with the cartilage mineralization differentiation process. Western blotting results showed that the mineralization induction fluid activated the expression of the BMP2 signal transduction pathway on d3 using the chondrocyte mineralization model, and then the activity decreased, but the expression was higher than that of the control group, indicating that the mineralization induction fluid promoted chondrocyte mineralization. The expression of FLRT3 in the mineralization induction group was significantly increased, and gradually increased with time in the condylar cartilage mineralization model. The trend of the control group was consistent with that of the mineralization group. During the mature mineralization period of chondrocytes, the activity of the BMP signal transduction pathway began to decrease with time, and the expression of FLRT3 gradually increased with time. The BMP2 signal and FLRT3 showed a trend of mutual antagonism.

[0047] The conclusions drawn from this experiment are: After using the condylar chondrocyte terminal mineralization induction model, it was found that FLRT3 was involved in the terminal mineralization of chondrocytes; FLRT3 and BMP2 signaling pathways negatively regulate chondrocyte mineralization.

[0048] 3. Static magnetic field affects the expression of FLRT3 and BMP2 signaling pathways during condylar chondrocyte mineralization

[0049] Studies have shown that magnetic fields can affect the BMP signaling pathway to regulate cell mineralization. The results of the second part of the experiment proved to some extent that FLRT3 is related to chondrocyte mineralization and may be involved in the regulation of the dynamic balance of chondrocyte maturation and mineralization during cartilage remodeling. Next, it is necessary to study whether FLRT3 in chondrocytes responds to static magnetic fields and whether FLRT3 and BMP2 signal transduction negatively regulate chondrocyte mineralization under the action of magnetic fields.

[0050] This experiment found through previous studies that the 280mT permanent magnetic field intensity significantly enhanced the proliferation ability of SD rat condylar chondrocytes and nasal septum chondrocytes compared with the 160mT and 360mT magnetic fields, and 280mT was selected as the optimal permanent magnetic field induced MCC in vitro culture intensity. Therefore, in order to systematically explain the signal transduction network of static magnetic field affecting the terminal mineralization of chondrocytes, this part of the experiment used 280mT permanent magnets to load the in vitro condylar chondrocyte mineralization model to maximize the simulation of clinical magnetic orthodontic correction force, and used Western blotting to detect the protein expression changes of terminal mineralization-related factors BMP2, Runx2 and FLRT3 in the BMP signaling pathway, thereby providing a theoretical basis for magnetic correction of mandibular condylar cartilage biological regulation and precise and efficient treatment. The results of the experiment are:

[0051] 1. Static magnetic field promotes the expression of FLRT3 during terminal differentiation induction

[0052] Western Blotting was used to detect the effect of static magnetic field on the expression of FLRT3 protein in condylar chondrocytes at d3, d8, and d15. It was found that the protein expression level of FLRT3 gradually increased with time, and the expression of FLRT3 after loading the static magnetic field was higher than that in the control group, suggesting that the regulation of static magnetic field on the terminal mineralization of chondrocytes may involve the participation of FLRT3.

[0053] 2. Static magnetic field inhibits the expression of related proteins of BMP signaling pathway in terminal differentiation induction. The expression of BMP2 and Runx2 proteins in condylar chondrocytes on d3, d8, and d15 was detected by Western blotting, and it was found that the levels of BMP2 and Runx2 proteins in the mineralization microenvironment gradually decreased significantly. After loading the static magnetic field, the expression of BMP2 and Runx2 proteins in condylar chondrocytes was higher than that in the control group on the 3rd day, and the expression of BMP2 and Runx2 gradually decreased from the 8th to the 15th day, but the change trend was consistent with that in the control group. It suggests that the early static magnetic field promotes chondrocyte mineralization through BMP2 / Runx2 signaling pathway transduction, and the activity of mature mineralization signal transduction gradually decreases with the extension of time. The expression of FLRT3 shows a trend of mutual antagonism with BMP2 signal transduction.

[0054] This experiment studied the mechanism of mandibular condylar chondrocyte differentiation at the cellular level. Based on the first two experiments, a terminal mineralization induction model has been successfully established by using differentiation induction fluid. On this basis, by loading a static magnetic field, the changes in terminal mineralization-related factors related to the BMP2 signaling pathway were studied in depth, and the signal transduction mechanism of the static magnetic field on the terminal differentiation and mineralization of chondrocytes was clarified. The conclusions of this experiment are: static magnetic fields promote the expression of FLRT3 during the induction of terminal mineralization of chondrocytes; static magnetic fields inhibit the transduction of the BMP signaling pathway during the mineralization induction process; static magnetic fields affect BMP2 signal transduction, FLRT3 participates in the dynamic balance of chondrocyte mineralization, and enhances their mutual antagonistic trend.

[0055] Through the research results of the above three animal experiments on the effects of static magnetic fields on the mineralization of condylar chondrocytes in SD neonatal rats, it can be concluded that static magnetic fields affect the osteogenic differentiation of the condyle by affecting the expression of BMP2 and FLRT3 proteins, thus proving that the static magnetic field generated by the magnetic condylar cartilage osteogenic dysfunction prevention and correction device can prevent condylar cartilage osteogenic dysfunction.

[0056] In the description of this specification, the description with reference to the terms "one embodiment", "example", "specific example", etc. means that the specific features, structures, materials or characteristics described in conjunction with the embodiment or example are included in at least one embodiment or example of the present invention. In this specification, the schematic representation of the above terms does not necessarily refer to the same embodiment or example. Moreover, the specific features, structures, materials or characteristics described can be combined in any one or more embodiments or examples in a suitable manner.

[0057] The preferred embodiments of the present invention disclosed above are only used to help illustrate the present invention. The preferred embodiments do not describe all the details in detail, nor do they limit the invention to the specific implementation methods described. Obviously, many modifications and changes can be made according to the content of this specification. This specification selects and specifically describes these embodiments in order to better explain the principles and practical applications of the present invention, so that those skilled in the art can understand and use the present invention well. The present invention is limited only by the claims and their full scope and equivalents.

Claims

1. A magnetic condylar cartilage osteogenic dysfunction prevention and correction device, comprising a device body, characterized in that: The device body includes a magnetic belt ring and a magnetic traction block, and the magnetic belt ring is connected to the magnetic traction block through a fixed steel wire; the magnetic belt ring includes a rounded rectangular steel ring, a fixed magnetic block, a sliding magnetic block installation groove and a U-shaped slide rail, the U-shaped slide rail is fixed on the two outer side walls of the steel ring, the fixed magnetic block is arranged on the outer wall of the steel ring in the U-shaped slide rail, and the sliding magnetic block installation groove is installed in the U-shaped slide rail through a slider; the magnetic traction block includes a traction groove and a straight-line slide rail, the traction grooves are two in number and are arranged vertically in parallel, and a U-shaped slider is arranged at the front and rear ends of each traction groove, and the two traction grooves are slidably installed on the straight-line slide rail through the U-shaped slider; the fixed steel wire is respectively fixed to the end of the straight-line slide rail and the U-shaped slide rail to connect the magnetic belt ring and the magnetic traction block; The fixed magnetic block is a permanent magnet; Permanent magnets with different magnetic forces can be detachably installed in the sliding magnet installation groove and the traction groove, and the permanent magnets are fixed in the sliding magnet installation groove and the traction groove by steel wires; The straight-shaped slide rail is marked with scales.

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