A preparation method of medicine for treating Barrett's syndrome

Abstract

The invention discloses a medicament for treating balintss syndrome so as to solve the problem of treating balintss syndrome. The medicament is characterized by being prepared from the following raw materials: lichee seeds, ocimum tenuiflorum, poria cocos, dried orange peel, aeschynomene indica, trachelospermum jasminoides, radix puerariae, paris polyphylla, vegetable sponge of luffa, common exbucklandia stem, coking malt and coking millet sprouts. The preparation method of the medicament comprises: (1) extracting volatile oil from coarse powder of radix puerariae, ocimum tenuiflorum and aeschynomene indica for reservation; (2) crushing coking millet sprouts into fine powder for reservation; (3) performing water extraction and alcohol precipitation on lichee seeds, poria cocos, dried orange peel, trachelospermum jasminoides, vegetable sponge of luffa, paris polyphylla, common exbucklandia stem, coking malt and coking millet sprouts, drying to obtain primary materials; and (4) spraying the volatile oil into the primary materials, mixing uniformly with coking millet sprouts, sieving by a sieve of 60 meshes and performing tabletting. Clinical experiments confirm that the medicament for treating balintss syndrome has the characteristics of good treatment effect and relatively high safety, and is worth clinical application and popularization.

Description

technical field [0001] The invention relates to the field of traditional Chinese medicines, in particular to a preparation method of a medicine for treating Barrett's syndrome. Background technique [0002] Barrett's syndrome (Barrett's syndrome), also known as Barrett's ulcer, refers to the replacement of the lower esophageal mucosa by the gastric columnar epithelium. It was first described by Barrett in 1950, so it is called Barrett syndrome. Intrinsic can be congenital, but often secondary to reflux esophagitis, esophagitis and ulcers occur on the basis of gastroesophageal reflux. The pathological feature is that the acid-resistant squamous epithelium is damaged, and the acid-resistant columnar epithelium regenerates and gradually spreads upwards to involve the lower esophagus. Ulcers can penetrate the wall of the esophagus, causing mediastinal tissue fibrosis and lymphadenitis. If blood vessels are damaged, they can cause bleeding, and there may be purulent lesions in ...