Renal artery sympathetic nerve activity measurement system

Abstract

The invention provides a renal artery sympathetic nerve activity measurement system. The system comprises blood analysis and measurement equipment, an interventional catheter, a flexible expansion part which is arranged at the position close to the other end part of the interventional catheter, is communicated with the interventional catheter and is used for controllably stimulating the renal artery sympathetic nerve activity through expansion at the renal artery so as to promote secretion to form related to-be-tested blood, and various blood detection sensors which are arranged on the interventional catheter, are close to the flexible expansion part and are used for collecting to-be-detected blood, converting the to-be-detected blood into various corresponding signals and feeding the various corresponding signals back to the blood analysis and measurement equipment, wherein various signals of the blood to be measured are analyzed through the blood analysis and measurement equipment to form corresponding physicochemical indexes reflecting renal artery sympathetic nerve activity. The technical scheme has the beneficial effects that the whole measurement system is high in controllability, simple in testing condition as long as the flexible expansion part is expanded to the wall of the renal arterial blood vessel. Furthermore, the activity of renal artery sympathetic nerves can be accurately determined based on specific physicochemical indexes.

Description

technical field [0001] The invention relates to the technical field of medical devices, in particular to a renal artery sympathetic nerve activity measurement system. Background technique [0002] The RAAS system is the renin-angiotensin-aldosterone system. The classic RAAS includes: the juxtaglomerular cells of the glomerular afferent artery secrete renin, activate angiotensinogen produced in the liver to produce angiotensin I, and then convert enzymes in the pulmonary circulation to produce angiotensin II (ATII). ATII is the main effector substance of RAAS, acting on angiotensin II receptor 1, causing arteriole smooth muscle to contract, stimulating the adrenal cortex to secrete aldosterone, and increasing the secretion of norepinephrine through the positive feedback of the presynaptic membrane of the sympathetic nerve terminal , these effects can increase blood pressure. [0003] Although there is an analysis of the overall regulation results of the RAAS regulation syst...

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