Combination therapy
A technology for binding molecules and binding domains, which can be used in drug combinations, antibody medical components, chemical instruments and methods, etc., and can solve problems such as reducing a variety of cytokines
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[0277] As stated above, the present invention relates to combination therapy for the treatment of cancer comprising the administration of:
[0278] (1) A molecule capable of binding PD-1 or a natural ligand of PD-1; and
[0279] (2) Molecules capable of mediating redirected killing of target cells (eg, diabodies, BiTe, bispecific antibodies, etc.).
[0280] The invention also relates to pharmaceutical compositions comprising such molecule(s).
[0281] As used herein, the term "administration" refers to providing such molecules in relative doses and in temporal proximity to provide the recipient with binding to PD-1 or a natural ligand of PD-1 and to target cells ( For example, both redirected killing of cancer cells or of pathogen-infected cells).
[0282] With respect to molecules capable of binding PD-1 or natural ligands of PD-1, the present invention particularly relates to wherein such molecules possess an epitope that immunospecifically binds PD-1 to inhibit (i.e., blo...
Embodiment 1
[0973] Combination Therapy Study: LOX-IMVI Tumor Model
[0974] To illustrate the principles of the present invention, combination therapy studies were performed using a reconstituted tumor model in which LOX-IMVI human metastatic melanoma cancer cells were injected subcutaneously into MHCI reconstituted with human PBMC - / - in mice. Mice were then administered vehicle or the following treatments:
[0975] (1) Humanized anti-human PD-1 antibody: hPD-1 mAb7(1.2) IgG4(P), this type of antibody is a molecule capable of binding PD-1; and / or
[0976] (2) CD3 x B7-H3 bispecific diabody DART-A, this type of diabody is a molecule that can bind to a cell surface molecule (ie, CD3) of effector cells and can bind to a cancer antigen (ie, B7-H3), This can mediate redirected killing of B7-H3-expressing cancer cells.
[0977] The amino acid sequences of such administered molecules are described above. Table 11 shows the parameters of the study. Each group consisted of 6 female mice. Fo...
Embodiment 2
[0981] Combination Therapy Study: Detroit562 Tumor Model
[0982] To further illustrate the principles of the present invention, a combination therapy study was performed using a reconstituted tumor model in which Detroit562 human metastatic pharyngeal carcinoma cells were subcutaneously injected into MHCI reconstituted with human PBMC - / - in mice. Mice were then administered vehicle control, 1 mg / kg hPD-1 mAb7(1.2) IgG4(P), 0.5 mg / kg DART-A or 1 mg / kg hPD-1 mAb7(1.2) IgG4(P) and 0.5 mg / kg hPD-1 mAb7(1.2) IgG4(P) kg DART-A both. Table 12 shows the parameters of the study. Each group consisted of 8 male mice. For all groups, mice received 5 x 10 6 Detroit562 cancer cell (ID) and 10 6 Human PBMC (IP; administered on study day 0). Treatment (molecule(s) or vehicle administered starting on study day 7) was provided weekly for four weeks (Q7Dx4) or every 14 days for up to two doses (Q14Dx2); via Doses were administered intravenously.
[0983]
[0984] Measure tumor volum...
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