Methods of preventing and/or treating cognitive impairment and neuroinflammation associated with aging
A technology for cognitive impairment and aging, applied in the field of 2-[4-phenyl]-1H-benzimidazole, which can solve the problems that the role of microglial cells has not yet been reported
- Summary
- Abstract
- Description
- Claims
- Application Information
AI Technical Summary
Problems solved by technology
Method used
Image
Examples
Embodiment 1
[0038] Example 1. Ppargc1a activator ZLN005 inhibits metabolic dysfunction of microglial cells in aged mice
[0039] Aged animals were treated orally with 0.5% methylcellulose (vehicle) or 25 mg / kg ZLN005 (Sigma) (in vehicle) 3 times a week for 15 weeks, starting at 37 weeks of age. Treated old animals (n=6), untreated old animals (n=6) and young animals (20 weeks old, n=6) were sacrificed after drug treatment and their brain tissues were collected. Brain tissues of sacrificed animals perfused with PBS were digested with collagenase I and analyzed by flow cytometry (Ginhoux et al. "Science" 330:841-5, 2010). Brain microglial cells were treated with 2-NBDG (2-(N-(7-nitrobenzo-2-oxa-1,2-oxadiazol-4-yl)amino)-2-deoxyglucose, Invitrogen ) and anti-Slc2a1 antibody (RnD) for flow cytometry harvesting (LSRII, BD) and analysis (FlowJo).
[0040] The results are summarized in figure 1 Middle; Y-axis represents % of microglia expressing the glucose transporter Slc2a1 or having taken...
Embodiment 2
[0041] Example 2. Ppargc1a activator ZLN005 inhibits the production of inflammatory cytokines in aged mouse microglial cells
[0042] Aged animals were treated orally with 0.5% methylcellulose (vehicle) or 25 mg / kg ZLN005 (Sigma) (in vehicle) 3 times a week for 15 weeks, starting at 37 weeks of age. Treated old animals (n=6), untreated old animals or senescent animals (n=6) and young animals (20 weeks old, n=6) were sacrificed after drug treatment and their brains were collected organize. Brain tissues of sacrificed animals perfused with PBS were digested with collagenase I and analyzed by flow cytometry (Ginhoux et al. "Science" 330:841-5, 2010). Brain microglia were phenotyped with fluorochrome-labeled CCL2 antibody and TNF-α (Biolegend) for flow cytometric harvest (LSRII, BD) and analysis (FlowJo).
[0043] The results are summarized in figure 2 Middle; Y-axis represents % of microglia expressing CCL2 or TNF-α. Statistical analysis was performed by ANOVA. The results ...
Embodiment 3
[0044] Example 3. Ppargc1a activator ZLN005 inhibits systemic inflammation in aged mice
[0045] Serum TNF-α levels in young mice, vehicle-treated aged mice, and ZLN-treated aged mice (from Example 2) were measured by ELISA to determine TNF-α levels indicative of systemic inflammation. The results are summarized in Table 3; the Y-axis represents the concentration of serum TNF-α. The results showed that by administering ZLN005 to aged animals, serum TNF-α levels in these treated and aged animals were reduced compared to vehicle-treated aged animals, thereby suppressing systemic inflammation (p-value<0.05). Statistical analysis was performed using an unpaired t-test.
PUM
Login to View More Abstract
Description
Claims
Application Information
Login to View More 


