Medicinal uses of plastin t
A drug, the technology of PLS3-C566A, is used in the prevention and treatment of aortic dissection to achieve the effect of improving vascular endothelial dysfunction
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Embodiment 1
[0025] Example 1 Correlation detection between aortic dissection and plastin3 in endothelial cells
[0026] In order to investigate whether aortic dissection affects the modification level of plastin3 in endothelial cells, the level of thiol-nitrosylation modification of aortic plastin3 in patients with clinical aortic dissection and cadaveric donors without cardiovascular disease was detected by biotin conversion method (biotin-switch) joint Western blot detection results are as follows figure 1 as shown, figure 1 Among them, PLS3 represents the content of plastin T (plastin3), and SNO-PLS3 represents the content of plastin T modified by thiol nitrosylation; figure 1 It can be seen that the level of thiol nitrosylation modification of plastin3 was significantly increased in patients with aortic dissection.
[0027] The Western blot detection method is a conventional technique in the art, for details, please refer to the literature: "BurnetteWN. Western blotting: electrophor...
Embodiment 2
[0031] Example 2 Inhibition of thiol-nitrosylation modification of plastin3 to improve endothelial dysfunction in aortic dissection
[0032] In order to further study whether the sulfhydryl nitrosylation modification of plastin3 is involved in the occurrence of endothelial dysfunction in aortic dissection at the cellular level, this example uses Lipofectamine 3000 (Invitrogen) in HUVEC cells and transfected PLS3-WT according to the instructions and PLS3-C566A plasmid for 24 h, and then treated with Ang II (100 nM) for 24 h to detect the change of endothelial cell function, the results are shown in the attached Figure 4-7 Shown:
[0033] in, Figure 4 Schematic diagram of overexpression of PLS3-WT and PLS3-C566A plasmids in HUVEC cells for 24 hours, and then treated with Ang II (100 nM) for 24 hours, Figure 4 It was found that overexpression of PLS3-C566A could significantly attenuate Ang II-induced lamellipodia formation in endothelial cells.
[0034] Figure 5 Schematic...
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