Biomarker signatures and uses thereof
a biomarker and signature technology, applied in the field of biomarkers, can solve the problems of insufficient diagnosis, inability to solve key unmet clinical problems, and inability to use multiplexed serum biomarker signatures to achieve the goal of reducing the risk of lupus, and achieve good conjugate and good reaction colour
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[0256]Serum profiling using antibody microarrays can facilitate our understanding of disease by identifying panels of disease-specific biomarkers, thereby enhancing our ability to diagnose and treat such diseases. Here, we performed a comprehensive survey of serologic proteome in human SLE using our in-house developed human recombinant single-chain fragment variable (scFv) microarray technology. We have indentified 20 serum biomarker signatures, that discriminates between SLE patients and healthy controls with a high specificity and sensitivity. When targeting the individual clinical subgroups of SLE we observed that differences in protein expression patterns depend on the activity and severity of the SLE symptoms. Serum proteome of patients with SLE glomerulonephritis (SLE3) showed the most significant difference compared to healthy one. Furthermore, the levels of some serum proteins, were highly correlated with clinical measure of disease activity (SLEDAI), as well as ...
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[0303]Systemic lupus erythematosus (SLE) is a potentially fatal autoimmune multisystem disease, characterized by chronic inflammations of various tissues and internal organs. The underlying mechanisms, not yet fully understood, involve immunological deficiencies as well as genetic, environmental and hormonal factors (1). Due to heterogeneous symptoms and an unpredictable course of disease, the management of SLE is one of the greatest challenges within the field of rheumatology (2).
[0304]Associated with poor long-term prognosis (3), SLE nephritis is one of the more severe manifestations of SLE that occurs in about 50% of all patients (1). Commonly, it is a result of antibody-antigen complex deposition in the glomerulus, i.e. the capillary network of the nephron, where the initial blood filtration takes place. These complexes activate the complement system which mediates local inflammatory events (4).
[0305]Standard treatment involves immunosuppressive agent cyclophosphamide. However, ...
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