Intravascular system and method for blood pressure control

a blood pressure control and intravascular technology, applied in the direction of internal electrodes, transvascular endocardial electrodes, therapy, etc., can solve the problems of peripheral vascular resistance, afterload, and sympathetic tone decreas
US20100211131A1Inactive Publication Date: 2010-08-19INTERVENTIONAL AUTONOMICS
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Patent Information

Authority / Receiving Office
US · United States
Current Assignee / Owner
INTERVENTIONAL AUTONOMICS
Publication Date
2010-08-19
Estimated Expiration
Not applicable · inactive patent

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Abstract

An intravascular lead is used to deliver energy for stimulating nervous system targets using energy delivery elements (e.g. electrodes) that are in direct contact with the nervous system targets. The lead may be positioned within the internal jugular vein and the nervous system targets may include the carotid artery / carotid sinus bulb and / or associated baroreceptor afferents, and / or surrounding nervous system targets in the region of the internal jugular vein, such as the carotid sinus nerve and / or associated nerve branches and / or the vagus nerve and / or associated nerve branches. Stimulation energy travels along a conductive bridge that extends from the intravascular lead to the nervous system target, or is relayed from the intravascular lead to another device disposed within or surrounding the target structure.
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Description

RELATED APPLICATIONS

[0001] This application claims the benefit of U.S. Provisional Application No. 61 / 043,070, filed Apr. 7, 2008, and U.S. Provisional Application No. 61 / 043,350, filed Apr. 7, 2008, each of which is incorporated herein by reference.FIELD OF THE INVENTION

[0002] The present invention generally relates to implantable devices and systems, and associated methods for delivering therapy to nervous system targets using components implanted within the vasculature.BACKGROUND OF THE INVENTION

[0003] Heart failure (HF) is a condition characterized by reduced cardiac output that triggers neurohormonal activation. This compensatory mechanism functions acutely to increase cardiac output and restore left ventricular (LV) functional capacity such that patients remain asymptomatic. Over time, however, sustained activation of these neurohormonal systems triggers pathologic LV remodeling and end-organ damage that ultimately drives the progression of HF.

[0004] In many people, persistent hyp...

Claims

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