Methods and reagents for characterizing genomic editing, clonal expansion, and associated applications
A genome editing, genome technology, applied in the fields of genomics, biochemical equipment and methods, proteomics, etc., can solve problems such as sensitivity that cannot provide early selection
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example 1
[0246] In one example, an initial cell population is subjected to targeted genome editing using a CRISPR / Cas9 endonuclease system. Cas9 causes double-strand breaks in genomic DNA, which has been shown in some applications to induce cell cycle arrest through the TP53 pathway (ie, PMID 29892062, PMID 29892067). Theoretically, genome editing has lower efficiency in some cases, such as in embryonic stem cells, due to cell cycle arrest induced by double-strand break reactions, which are mediated by TP53. Those cells that did not stall after the genome editing process likely contained inactivating mutations or deletions or loss of TP53 (or other cancer driver genes) that disrupted the corresponding function and resulted in less restricted cell growth. Clonal expansion of cells carrying this mutation will outcompete neighboring cells. In this example, double-sequencing will be used to generate error-corrected consensus sequence reads of targeted double-stranded DNA molecules and det...
example 2
[0248] This example describes the use of dual sequencing to determine whether early mutations in cancer driver genes reflect the tumorigenic potential of a test carcinogen. Using model mutation inducers, this example demonstrates that dual sequencing is capable of resolving mutations in a single DNA molecule in a population. This example demonstrates that the methods of the present disclosure provide the necessary sensitivity to detect such early neoplastic clonal selection of cells in a cell population and the subsequent clonal expansion of such cells.
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