Fusion peptide for inhibiting tumor growth and application thereof in preparing anti-tumor medicament
A technology for fusion of peptides and tumors, applied in the direction of hybrid peptides, peptides, specific peptides, etc.
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Embodiment 1
[0039] The synthesis of embodiment 1 polypeptide (PUMA) BH3, TAT-DV3 and TAT-DV3-BH3
[0040] Experimental results show that the targeted fusion peptide of (PUMA) BH3 domain can inhibit the growth of some tumor cells and exhibit certain anti-tumor activity. Therefore, a targeted fusion peptide containing (PUMA) BH3 domain and two reference polypeptides were synthesized from Shanghai Gill Biotechnology Co., Ltd. All synthetic peptides were purified by reverse-phase high-performance liquid chromatography with a purity of >95%. The amino acid sequence was verified by MALDI-TOF mass spectrometry analysis, and FITC was added to the N-terminus of each peptide. The names of the peptides are (PUMA)BH3, TAT-DV3 and TAT-DV3-BH3, and the sequences are shown in Table 1.
[0041] Table 1:
[0042]
Embodiment 2
[0043] Example 2 Fusion peptide TAT-DV3-BH3 inhibits cell growth experiment
[0044] Select 5 cell lines from known tumor cells overexpressing CXCR4: human colon cancer cell HCT116 P53+ / + and HCT116 P53- / - , human lung adenocarcinoma cell line GLC-82, human breast cancer cell line MDA-MA-231, human gastric cancer cell line SGC-7901, two non-tumor cell lines human embryonic lung fibroblast 2BS and human embryonic kidney fibroblast HEK293 . Two of the colon cancer cells were P53 wild-type HCT116 P53+ / + , and the other is p53-deleted HCT116 P53- / - ; If the peptide we synthesized induces cell apoptosis according to the PUMA (BH3) pathway as we imagined, then there should be no significant difference in the effect of the peptide on the two strains of cells, that is to say, the occurrence of apoptosis is related to that of P53. Existence is irrelevant. The above 7 kinds of human tumor cells were subjected to cell growth inhibition experiment with polypeptide TAT-DV3-BH3. For s...
Embodiment 3
[0048] Effect of embodiment 3 (PUMA) BH3, TAT-DV3 and TAT-DV3-BH3 on cell growth
[0049] HCT116 was added at 5000 cells / well P53+ / + 、HCT116 P53- / - and HEK283 cells were inoculated in a 96-well plate, and after 24 hours of culture, the original culture medium was sucked off, and (PUMA) BH3, TAT-DV3 and TAT-DV3-BH3 were added with a final concentration of 60 μM; then cultured for 72 hours, and the polypeptide containing Add 100 μL (including 10 μL of CCK-8) culture solution to each well, and continue to cultivate for 1 hr. Use a Bio-Rad 680 microplate reader to measure the absorbance at 450 nm and the reference wavelength at 630 nm to measure the absorbance of each well and the survival rate (%) =100×(A experimental group-A blank) / (A control group-A blank), the results are shown in Table 3, figure 2 .
[0050] Table 3 Effects of peptides with a concentration of 60 μM on cell viability
[0051]
[0052] The above results indicated that neither (PUMA)BH3 acting alone nor ...
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