A predictive molecule for judging the curative effect of targeted monoclonal antibody therapy on tumor
A monoclonal antibody and targeting technology, applied in the biological field, can solve limitations and other problems, and achieve the effect of improving the quality of life and improving the curative effect
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Embodiment 1
[0050] Example 1, PRSS1 as a marker for judging whether the cells to be tested are resistant to cetuximab
[0051] 1. Identification of the molecule PRSS1 related to drug resistance
[0052] Through a large amount of literature research, a gene expression profile database (Affymetrix133A Microarrays) about 798 cell lines was found. The database is available from the Sanger Cell Line Project at http: / / www.broadinstitute.org / cgi-bin / cancer / datasets.cgi. After a large number of literature searches, it was found that among the 798 cell lines in this database, 19 cell lines (EGI-1, HCC70, COLO-205, HCT116, HT-29, LS1034, LS174T, RKO, SW620, PC-3 , KATOIII, MKN45, AGS, SAS, HuCCT1, A549, SW403, HCT15, TE-1) are cetuximab-resistant cell lines reported in the literature, 30 cell lines (MDA-MB-468, LoVo, SW48, HuH-7, SW1573, TE-5, KYSE-150, 22RV1, DU-145, MKN1, MKN28, NCI-N87, Ca9-22, CAL-33, HSC-2, HSC-3, FaDu, CAL- 39. U251, Ca-Ski, DoTc2-4510, ME-180, OMC-1, A498, ACHN, CAKI-1, S...
Embodiment 2
[0060] Example 2, PRSS1 improves the drug resistance of colon cancer cells to cetuximab
[0061] 1. Obtaining colon cancer cells overexpressing PRSS1 and colon cancer cells that interfere with PRSS1 expression
[0062] 1. Obtainment of colon cancer cells overexpressing PRSS1 full-length peptide and mature peptide
[0063] 1) Recombinant vectors and transfected cells overexpressing PRSS1
[0064] pMSCV-puro-PRSS1 is obtained by inserting the coding gene (sequence 1) of the full-length peptide of PRSS1 into the BglⅡ and SalⅠ restriction sites of pMSCV-puro (CLONTECH Company, catalog number PT3303-5, catalog #K1062-1) Carrier.
[0065] pMSCV-puro-PRSS1' is a vector obtained by inserting the coding gene of PRSS1 mature peptide (sequence 3) into the BglII and SalI restriction sites of pMSCV-puro.
[0066] pMSCV-puro-PRSS1, pMSCV-puro-PRSS1', and pMSCV-puro were transfected with LoVo (purchased from ATCC, USA, BNCC337953), respectively, to obtain colon cancer cells transfected wi...
Embodiment 3
[0116] Example 3, PRSS1 degrades the remaining monoclonal antibodies
[0117] To analyze whether PRSS can degrade other commercial monoclonal antibodies such as bevacizumab and trastuzumab.
[0118] Method is basically the same as 3 of 3 of Example 2, only cetuximab is replaced by bevacizumab (the name of the company is Roche Pharma (Schweiz) Ltd. The domestic contact is Shanghai Roche Pharmaceutical Co., Ltd., and the import registration standard: JS20110006) or trastuzumab (the name of the company is Roche Pharma (Schweiz) Ltd, the domestic contact is Shanghai Roche Pharmaceutical Co., Ltd., import registration standards: JS20110007 and JS20110008), and the colon cancer cells are HT-29.
[0119] The result is as Figure 10 As shown, PRSS1 in the cell supernatant was found to degrade bevacizumab and trastuzumab in addition to cetuximab.
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