Application of (-)-jorunnamycin A in inhibiting growth of bladder tumor cells and application method of (-)-jorunnamycin A
A technology for bladder tumors and application methods, applied in the application field of inhibiting the growth of bladder tumor cells, to achieve the effect of inhibiting the growth of bladder cancer cells and having a good inhibitory effect
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Embodiment 1
[0040] The small molecule (-)-jorunnamycin A of the marine biological extract mentioned in the present invention has the following chemical structural formula:
[0041]
[0042] Regarding the inhibitory effect of (-)-jorunnamycin A on different types of cells, the inventors conducted the following experiments:
[0043] Take human embryonic kidney cells H293T, human colon cancer cells SW480, human glioma cells U251, bladder cancer cells SW780, bladder cancer cells T24, and bladder cancer cells UMUC3 as samples, and culture them in 96-well plates at an initial density of 25%. , when the density grew to 50-55%, different amounts of (-)-jorunnamycin A were added to make the final concentrations respectively 0, 20, 40, 60, 80, 100, 200, 300, 400 nM.
[0044] After culturing for 24 hours, the residual viability of the cells was detected by the CCK8 method and plotted (the cell viability without (-)-jorunnamycinA was taken as 100%).
[0045] attached figure 1 It is a diagram sho...
Embodiment 2
[0050] In Example 2, the present inventors further investigated the inhibitory effect of (-)-jorunnamycin A on muscle-invasive bladder cancer, and the specific steps are as follows:
[0051] Take a cell sample and culture it into a 96-well plate according to the initial density of 25%. When the density grows to 50-55%, add (-)-jorunnamycin A at a concentration of 0-200nM. After culturing for 24 hours, use the CCK8 method to detect the cells Remaining viability, and the half-inhibitory concentration IC50 of (-)-jorunnamycin A for the muscle-invasive bladder cancer cell line T24 was 54.1nM, and the half-inhibitory concentration IC50 for the muscle-invasive bladder cancer cell line UMUC3 was 83.6nM, as attached figure 2 shown.
Embodiment 3
[0053] The present invention further investigates (-)-jorunnamycin A to inhibit the division ability of muscle-invasive bladder cancer cell T24 cell, and the results are shown in the attached image 3 shown.
[0054] The specific method is as follows: One of the main characteristics of cancer cells is rapid mitosis, and the premise of cell mitosis is DNA replication. Therefore, the effect on DNA replication rate is an important experimental method to evaluate the tumor suppressor effect of drugs. In the present invention, the EdU kit method (the EdU kit is purchased from Beyuntian Company) is used to detect the inhibitory effect of (-)-jorunnamycin A on the DNA replication of bladder cancer cells. From attached image 3 It can be seen from the results in that (-)-jorunnamycin A can effectively inhibit the DNA synthesis of bladder cancer cells in a concentration-dependent manner.
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