Pulmonary artery support

Abstract

The invention relates to the technical field of medical appliances and discloses a pulmonary artery support. The pulmonary artery support comprises a circumferential-dilatable metal support; and an isolating membrane, wherein the isolating membrane wraps the metal support so as to isolate the metal support from an external environment, and the circumferential tensile strength of the isolating membrane is lower than the axial tensile strength of the isolating membrane. According to the pulmonary artery support disclosed by the embodiments of the invention, the diameter of the support can be dilated according to diameter changes of blood vessels so as to meet the requirements of the blood vessels on supporting performance after being enlarged, and a metal portion of the support can be isolated from a blood vessel environment, so that the problem that the interior of the bracket is subjected to restenosis is effectively solved.

Description

technical field [0001] The invention relates to the technical field of medical devices, in particular to a pulmonary artery stent. Background technique [0002] Pulmonary artery stenosis is more common in complex congenital heart disease. With the development of percutaneous interventional therapy, implantation of an implant for the treatment of pulmonary artery stenosis has become an effective treatment for pulmonary artery stenosis. The application of a pulmonary artery stenosis implant for interventional surgery has shown good results in pulmonary artery stenosis, right ventricular outflow tract stenosis, channel stenosis after corrective complex congenital heart disease, systemic pulmonary shunt conduit and collateral vessel stenosis. The specific interventional operation is to introduce a special guide wire, catheter, and an implant for treating pulmonary artery stenosis into the human body under the guidance of medical imaging equipment, and perform surgical treatment ...

AI Technical Summary

Problems solved by technology

However, traditional vascular stents cannot meet the changes in vessel size over time; secondly, the local stimulation of traditional vascular stent implantation on the pulmonary artery will further lead to intimal hyperplasia, resulting in in-stent restenosis; thirdly, because the physiological structure of the pulmonary artery is located in the The right ventricle has an outflow outlet, so when the heart contracts to pump blood out of the ventricle and into the pulmonary artery, the blood flow velocity and blood pressure are relatively high. After the traditional vascular stent is implanted, it is easy to shift under the force of the blood flow impact, resulting in failure

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