Gene detecting method for hereditary risk evaluation of antioxidant ability

Abstract

The invention discloses a gene detecting method for hereditary risk evaluation of antioxidant ability, which is characterized by providing a personalized health guidance of the hereditary risk evaluation and the related disease risk aversion of the antioxidant ability for all detected persons by simultaneously detecting and analyzing the gene carrying types of SNPs sites of CAT genes, CYBA genes, NOS3 genes, SOD3 genes and PON1 genes of an individual. The method has the advantages that: before diseases appear, the risks of the diseases related to free radicals are pointed out to the detected persons from a hereditary standpoint so as to improve the self health-care awareness of the detected persons; and reasonable intervention means are actively adopted so as to achieve the aim of reducing the probability of the diseases; the pathogenic factors and mechanisms of the diseases are analyzed from the standpoint of the molecule mechanism so as to deepen the realization of the diseases, promote the making of corresponding diagnosis and treatment measures and improve the treatment effect.

Description

technical field [0001] The invention relates to a gene detection method for genetic risk assessment of antioxidant capacity, which is used to assess the strength of an individual's antioxidant capacity, and specifically prompts the subject to take appropriate measures when facing various external factors that may cause oxidative damage. The countermeasure belongs to the technical field of molecular biology. Background technique [0002] There is a very important balance system in the human body: the oxidation-reduction system, which regulates the production and metabolism of oxygen free radicals in the body, maintains the proper oxidation ability and the scavenging ability of oxygen free radicals in the body, thus ensuring the life activities carried out. [0003] Oxidation provides the human body with the energy needed for life activities, but the resulting "by-product" - excessive accumulation of oxygen free radicals will damage the normal function of the body at the mole...

AI Technical Summary

Problems solved by technology

[0011] 2. Free radicals can cause abnormal cross-linking, polymerization, and peptide chain breakage of proteins, and can also combine proteins with lipids to form polymers, losing protein functions; they may also modify amino acids on enzymes to make enzymes abnormally active;

[0016] 1. Free radicals destroy the cell membrane, leading to denaturation of the cell membrane, so that the cells cannot absorb nutrients from the outside, and cannot excrete metabolic waste in the cells, and lose their ability to resist bacteria and viruses;

[0017] 2. Free radicals attack genes that are being replicated, causing gene mutations to induce cancer;

[0022] 7. Free radicals cause the degradation of the joint membrane and synovial fluid, leading to arthritis;

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