Wearable wireless intelligent posterior tibial nerve electrical stimulation system

Abstract

The invention discloses a wearable wireless intelligent posterior tibial nerve electrical stimulation system. The system comprises a stimulator and a mobile terminal unit communicated with the stimulator, wherein a MCU logical control unit is used for controlling charge and discharge of a lithium battery unit, controlling a nonvolatile memory to perform information storage, controlling a temperature measurement unit to detect the body surface temperature of a stimulation position, controlling a status indicator lamp to perform indication, controlling a low power consumption Bluetooth unit to perform wireless communication, controlling a key interaction unit to perform interaction, controlling a DAC unit to generate specific analog voltage, controlling a high-voltage BOOST circuit to open or close and indirectly controlling a constant flow source circuit to open or close, a waveform modulation circuit and output through an electrode connection unit, and performs body surface impedance detection and overcurrent overload protection. According to the wearable wireless intelligent posterior tibial nerve electrical stimulation system, stimulating current flows through the acupoint of sanyinjiao and the posterior tibial nerve, electrical pulse is generated among nerves of the spinal cord, detrusor contraction and filling of bladder of a patient can be improved, and the symptom of OABcan be treated and improved.

Description

technical field [0001] The invention belongs to the field of medical electronics and relates to a wearable wireless intelligent posterior tibial nerve electrical stimulation system. Background technique [0002] Overactive Bladder (OAB) is defined by the International Continence Society as a syndrome characterized by urinary urgency, often accompanied by frequent urination and nocturia symptoms, with or without urge incontinence, without urinary tract infection or Other definite pathological changes. Urodynamically, OAB can manifest as detrusor overactivity or other forms of urethro-bladder dysfunction. The pathogenesis of OAB is relatively complex. The urination process is controlled by higher nerve centers (such as cerebral cortex, pons, and spinal cord) and peripheral nervous systems (autonomic nerves, somatic nerves), and sensory nerves in the lower urinary tract are also involved in the regulation of urination. Abnormal function of any of the above parts may cause OAB...

AI Technical Summary

Problems solved by technology

Although bladder training methods and drug treatments are available, according to statistics, 40% of patients still cannot get satisfactory results and need to seek minimally invasive or surgical treatment

Images