Application of gene FOXD4 to preparation of diagnosis reagent kit for acute myelocytic leukemia and reagent kit
A diagnostic kit and acute myeloid technology, applied in the fields of biomedical technology and hematological leukemia, can solve the problem of no research report on the expression of FOXD4 gene family members
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Embodiment 1
[0043] We first validated Kras in an AML animal model G12D / + ; In CD2Cre transgenic mice, myeloid / monocytic progenitor cells in the bone marrow and myeloid leukemia cells in the spleen up-regulated the FOXD4 signaling pathway gene ( figure 1 ). (A, B) Quantitative qPCR to determine the difference in transcript levels of candidate genes. RNA from control and Kras G12D / + ; CD2Cre + myeloid / monocytic progenitors in bone marrow (A) or Gr1 in spleen + Mac1 + Extracted from myeloid leukemia cells (B). The relative expression level is expressed as the expression fold relative to GAPDH.
[0044] The result was that qPCR verified the gene expression of the granulocyte TGF-β pathway in AML mice was elevated ( figure 1 A, B). qPCR results proved that Tgfβr1, Tgfβ1, Foxd4 in Kras G12D / + ; CD2Cre + myeloid / monocytic progenitors and Gr1 + Mac1 + Highly expressed in myeloid leukemia cells ( figure 1 A, B), while the expression of Hoxa3, foxa2, and foxa3 are less different.
Embodiment 2
[0046] To evaluate the gene expression level of FOXD4 in peripheral blood of clinical acute myeloid leukemia patients.
[0047] Experimental materials: Collect peripheral blood samples from 8 patients with acute myeloid leukemia, and collect peripheral blood samples from 3 normal people at the same time, and use fluorescence real-time quantitative PCR to carry out classical molecular biology experiment verification (qRT-PCR).
[0048] Experimental results: The target band was determined by melting curve analysis and electrophoresis, and the ΔΔCT method was used for relative quantification. The results are as follows: figure 2 shown. Compared with normal peripheral blood samples, the expression of FOXD4 gene family members FOXD4(A) and FOXD4L1(B) in the peripheral blood of 3 clinical myeloid leukemia patients was up-regulated by q-PCR. There was no significant difference between FOXD4L4 and FOXD4L5.
Embodiment 3
[0050] The expression of FOXD4 in myeloid leukemia patients in Oncomine database.
[0051] We found in the Oncomine database that the expression of FOXD4 gene was positively correlated with the exacerbation stage of acute myeloid leukemia and chronic myeloid leukemia ( image 3 ).
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