Pseudolarix triterpene lactone, and preparation method, pharmaceutical composition and application thereof
A composition and drug technology, applied in the field of medicine, can solve the problems of repeated symptoms and aggravation, and achieve the effect of good therapeutic effect
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Embodiment 1
[0040] Embodiment 1: Separation and structural characterization of compound 1-6
[0041] 100kg of pine seeds, pulverized, extracted 3 times by cold soaking with 95% ethanol, the first 12 hours and then twice for 2 hours each time. The extract was concentrated under reduced pressure to obtain 8.87 kg of extract, which was suspended in 20 L of water, extracted successively with petroleum ether, dichloromethane, ethyl acetate and n-butanol (each extracted 3 times), combined the extracts and recovered the solvent to obtain dichloromethane Part 3.97kg. The dichloromethane part was adsorbed on a silica gel (160-200 mesh) chromatographic column, and eluted with petroleum ether to ethyl acetate 30:1, 20:1, 10:1, 5:1, 1:1 and ethyl acetate in sequence, Six fractions of G1-G6 were obtained respectively. Among them, the G3 fraction (480g) was adsorbed on a silica gel (200-300 mesh) chromatographic column, and then eluted with petroleum ether to acetone 10:1, 8:1, 5:1, 3:1, and 1:1 to o...
experiment example 1
[0065] Experimental example 1 isolates and obtains all triterpene compounds (containing active best compound 1-6) anti-inflammatory activity test
[0066] 1. In vitro NF-κB pathway inhibition experiment
[0067] (1) Experimental principle
[0068] NF-κB is a nuclear transcription factor that plays an important role in immune and inflammatory responses by regulating the expression of inflammatory cytokines. In the resting state, the subunits p50 and p65 of NF-κB usually exist in the cytoplasm in the form of heterodimer. When pro-inflammatory stimuli (such as TNF-α) act on cells, they will activate NF-κB through the degradation and phosphorylation of IκBα, and the activated NF-κB p65 enters the nucleus and binds to DNA, and up-regulates inflammatory genes COX-2, Transcription of iNOS. NF-κB is activated and starts the transcription and expression of pro-inflammatory genes, leading to the excessive secretion of TNF-α, IL-6, IL-1β and other pro-inflammatory factors, thus aggrav...
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