Application of Antiplatelet Thrombolysin in Preparation of Drugs for Treating Anemia
An anti-platelet and thrombolytic technology, applied in the field of medicine, can solve the problem of meaningless etiological treatment, achieve the effect of reducing aggregation and prolonging survival time
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Embodiment 1
[0030] Example 1 Antiplatelet Thrombolysin Reduces Platelet-Neutrophil Aggregation in Mouse Model of Sickle Cell Anemia Induced by TNF-α
[0031] Neutrophils (neutrophil) play a very important role in the non-specific cellular immune system of the blood. Activated neutrophils reach the lesion site through chemotaxis, and the bactericidal substances they carry will be released in large quantities locally, causing tissue damage through their respective destructive effects, and further causing organ disorders. After neutrophils invade an organ, they will Induces accumulation of neutrophils in other vital organs, resulting in multi-organ damage. At the same time, platelet selective receptors mediate the binding and adhesion of activated platelets and neutrophils, neutrophils aggregate, and accelerate tissue damage; on the other hand, platelets and neutrophils adhere to promote platelet aggregation and mediate vascular occlusion , at the same time, the cell membrane of neutrophils...
Embodiment 2
[0039] Example 2 Therapeutic effect of antiplatelet thrombolysin on sickle cell anemia mice
[0040] Chimeric control and Berkeley mice were generated by transplanting Berkeley mouse bone marrow into 6-week-old lethally irradiated WT mice. Chimerism was confirmed by PCR and electrophoretic analysis 3 months after transplantation. These chimeric Berkeley The mice are sickle cell anemic mice. The mice that have successfully established the model were taken, and severe inflammation was induced by intraperitoneal injection of 500ng TNF-α, and single-blind administration was used after 3 hours of induction. The administration groups included: IgG control (1.5 μg / g), antiPDI (BD34, 1.5 μg / g) , BSA (25ng / g), anfibatide (25ng / g), antiPDI (1.5μg / g)+anfibatide (25ng / g).
[0041] After administration, the intravital microscope was used to observe the survival time of each mouse during the study period. The recording time started from the injection of TNFα and ended when the mice died or...
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