Application of artemisinin in targeted inhibition of myeloid-derived suppressor cells and preparation of tumor immunotherapy drugs
An immunotherapy drug and inhibitory cell technology, applied in the field of biomedicine, can solve problems such as artemisinin therapy combined with immunotherapy that have not been reported in research, and achieve the effect of less toxic side effects and promoting apoptosis.
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Embodiment 1
[0022] (1) In vitro detection of the effect of artemisinin treatment on the aggregation and apoptosis of MDSCs
[0023] Freshly isolated bone marrow cells from C57BL / 6 mice were adjusted to a cell density of 1×10 with RPMI 1640 medium containing 10% FBS 6 / ml, 40ng / ml GM-CSF and 40ng / ml IL-6 were added accordingly. After thorough mixing, the cell mixture was plated in a 24-well plate and cultured at 37°C, 5% CO 2 in the incubator. ART (50 μM, 100 μM, 300 μM, 500 μM) was added to the third day of culture, and DMSO was used as the control group to detect the proportion and apoptosis of MDSCs by flow cytometry.
[0024] (2) In vitro detection of the effect of artemisinin treatment on releasing the inhibitory effect of MDSCs on T cells
[0025]The MDSCs isolated above were treated with 100 μM artemisinin for 12 hours, and Gr-1 was separated and purified by magnetic beads + cell. Spleen of C57BL / 6 mice was aseptically isolated and purified by flow cytometry to obtain spleen CD...
Embodiment 2
[0028] Analysis of transcriptome sequencing results showed that MDSCs inhibited the expression of M2-type pathway-related genes under the action of artemisinin compared with the control group DMSO. Western Blot and qRT-PCR were used to detect the expression of immunosuppressive factor gene ARG1 and M1 gene iNOS in MDSCs cells, and the levels of arginase and nitric oxide in MDSCs cells were detected by commercial kits.
[0029] figure 2 : Artemisinin can polarize MDSCs from M2-type tumor-promoting phenotype to M1-type anti-tumor phenotype.
[0030] Figures A-B: Transcriptome sequencing showed that under the action of artemisinin, compared with the control group DMSO, the ARG1 immunosuppressive gene of MDSCs was down-regulated, and the expression of related genes in the M2 pathway was inhibited. Figure C: qRT-PCR detection of the expression of M1-type macrophage marker genes and M2-type macrophage marker genes in MDSCs, the results showed that compared with the control group D...
Embodiment 3
[0032] Two tumor models, B16F10 and Hepa1-6, were established in C57BL / 6 mice to verify the effect of artemisinin on tumor growth in vivo, and combined with anti-PD-L1 antibody to enhance anti-PD-L1 immunotherapy.
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