Pharmaceutical composition for preventing and treating cerebral infarction

A composition and technology of cerebral infarction, applied in the field of medicine, can solve the problems of slow onset of effect and general therapeutic effect, and achieve the effect of no side effects, good therapeutic effect and high safety
CN110664809AActive Publication Date: 2020-01-10JIAMUSI UNIVERSITY

Patent Information

Authority / Receiving Office
CN · China
Patent Type
Applications(China)
Current Assignee / Owner
JIAMUSI UNIVERSITY
Publication Date
2020-01-10

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Abstract

The invention relates to a pharmaceutical composition. The pharmaceutical composition is prepared from phellodendrine, paeoniflorin and optional EGCG (epigallocatechin gallate). The pharmaceutical composition can prevent or treat brain tissue damage caused by local and whole cerebral ischemia, can specifically prevent or treat cerebral hemorrhage, cerebral infarction, cerebral thrombosis and otherdiseases, especially has a very good treatment effect on cerebral infarction, and is high in safety and free from side effect.
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Description

technical field

[0001] The present invention relates to the field of medicine, in particular, the present invention relates to a pharmaceutical composition comprising Phellodendriline, Paeoniflorin and optionally EGCG, a preparation method, and an application thereof for preventing and treating cerebral infarction. Background technique

[0002] Cerebral infarction, also known as ischemic stroke, refers to the localized ischemic necrosis or softening of brain tissue caused by cerebral blood supply disturbance, ischemia, and hypoxia. The common clinical types of cerebral infarction include cerebral thrombosis, lacunar infarction, and cerebral embolism. Cerebral infarction accounts for 80% of all strokes. . Cerebral infarction not only poses a great threat to human health and life, but also brings great pain and heavy burden to patients, families and society.

[0003] After the onset of cerebral infarction, under the action of various pathophysiological mechanisms, neuronal c...

Claims

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