Dendrobe sobering-up tea and preparation method thereof

Abstract

The invention discloses a dendrobe sobering-up tea which is prepared from the following raw materials in parts by weight: 20-30 parts of fresh dendrobe, 20-25 parts of Japanese raisin, 9-10 parts of Hawk tea, 8-9 parts of kudzu root, 8-9 parts of olive, 3-5 parts of dried orange peel, 2-3 parts of strawberry, 3-4 parts of mung bean, 2-3 parts of lotus seed, 2-3 parts of Chinese wolfberry, 1-2 parts of ginseng, 1-2 parts of licorice root, 1-2 parts of radix rehmanniae flower, 1-2 parts of lily root, 0.01-0.02 part of sodium citrate and 0.01-0.03 part of cysteine. The reasonable formula and treatment process can be utilized to enhance the endurance of the mechanism, relieve fatigue, inhibit the stimulation of alcohol onto the nerve center, protect the brain functions and promote sleep, thereby finally achieving the effects of liver protection, detoxification and refreshing. Besides, the technique disclosed by the invention is suitable for industrial production, is applicable to both men and women at different ages, has favorable mouthfeel, satisfies the requirements for people drinking wine, and caters to the market.

Description

technical field [0001] The invention mainly relates to a hangover tea, in particular to a dendrobium hangover tea and a preparation method thereof. Background technique [0002] After alcohol is absorbed through the digestive tract, it enters the liver from the portal vein, and the main metabolic pathway (80%) is decomposed into acetaldehyde by alcohol dehydrogenase, then converted into acetic acid by acetaldehyde dehydrogenase, and finally produces carbon dioxide and water through the tricarboxylic acid cycle Or through the citric acid cycle and into other compounds. Long-term drinking can oxidize NADH into NAD in liver cells, increase lactic acid, and induce gout; and excess H + It can hinder the decomposition of fat in the mitochondria of liver cells, cause fatty liver, inhibit the progress of the tricarboxylic acid cycle in the body, block the alcohol metabolism pathway, seriously damage the normal tissues of the body, and cause a vicious circle. At the same time, ...

AI Technical Summary

Problems solved by technology

Long-term drinking can oxidize NADH into NAD in liver cells, increase lactic acid, and induce gout; and excess H + It can hinder the decomposition of fat in the mitochondria of liver cells, cause fatty liver, inhibit the progress of the tricarboxylic acid cycle in the body, block the alcohol metabolism pathway, seriously damage the normal tissues of the body, and cause a vicious circle

At the same time, excessive drinking will cause sweating, vomiting, diarrhea and other symptoms, and reduce the production of antidiuretic hormone, increase urination, resulting in a large loss of water, electrolytes, minerals and other nutrients in the body, resulting in severe water shortage and electrolyte imbalance in cells , affecting the normal metabolism of cells