63 results about "Iron deficient" patented technology

The invention relates to methods and compositions for identifying soybean plants that are tolerant, have improved tolerance or are susceptible to iron deficient growth conditions. The methods use molecular genetic markers to identify, select and/or construct disease-tolerant plants or identify and counterselect disease-susceptible plants. Soybean plants that display tolerance or improved tolerance to Phytophthora root rot infection that are generated by the methods of the invention are also a feature of the invention.

The invention discloses a high-power yttrium-gadolinium garnet ferrite having the composition chemical formula specifically expressed as Sm[x]Y[3-x-y-2z-p-q-q']Gd[y]Ca[2z+p+q+q']V[z]Ge[p]Sn[q]Ti[q']In[q'']Al[w]Mn[w']Fe[5-z-p-q-q'-q''-w-w'-[delta]]O[12], wherein 0<=x<=0.20, 0<=y<=3.0, 0<=z<=1.3, 0<=p<=0.9, 0<=q<=0.7, 0<=q'<=0.7, 0<=q''<=0.70, 0<=w<=1.3, 0<=w'<=0.10, 0<=delta<=0.8, and delta represents iron deficient quantity. The high-power garnet ferrite material having high spin wave line width [delta]Hk, low ferromagnetic resonance line width [delta]H and high dielectric constant epsilon' can be obtained.

The invention discloses a composite ferrum preparation for correcting iron-deficient chlorosis of fruit trees and increasing ferrum contents in fruit. The composite ferrum preparation comprises the following contents in mass percent: 60 % of commercially available composite amino acid powder, 2 % of citric acid, 16 % of ferrous sulfate, 18 % of zinc sulfate, and 4 % of surface active agent of sodium dodecyl sulfate. The composite ferrum has obvious effects on correcting iron-deficient chlorosis of fruit trees and increasing ferrum contents in fruit.

The invention discloses a nutrient solution for preventing and treating chlorosis of a camphor tree. The nutrient solution comprises the following components in concentration by mass: 0.1-2 percent ofwater-soluble iron-contained compound, 0.01-0.1 percent of ethyoxyl modified polytrisiloxane and the balance of water. The invention also discloses a method for preventing and treating chlorosis of the camphor tree by the nutrient solution. The nutrient solution is applied to the camphor tree by a leaf surface spray method. The invention can effectively solve the phenomenon of the iron deficiencychlorosis of the camphor tree.

The invention relates to a coding gene for enhancing iron deficiency tolerance and accumulation of a plant and application of the coding gene. The sequence of the coding gene for enhancing iron deficiency tolerance and accumulation of the plant is as shown in the sequence table SEQ ID No: 1. The coding gene for enhancing iron deficiency tolerance and accumulation of the plant is transferred into the plant, the coding gene for enhancing iron deficiency tolerance and accumulation of the plant is over-expressed in the plant, so that the tolerance of the plant to iron deficiency is displayed in vivo. The coding gene for enhancing iron deficiency tolerance and accumulation of the plant can provide gene resources and technical support for breeding crops under iron deficiency stress tolerance.

The present invention discloses the signaling pathway involved in erythroid repression by iron deficiency. Further disclosed is a non-toxic small-molecule compound which potently reverses the erythroid repression caused by iron deficiency. The present invention further encompasses novel compounds for inhibition of red cell production, useful, for example, in the treatment of polycythemia vera, a malignancy causing uncontrolled red cell production. These inhibitory compounds also promote megakary-ocytic lineage commitment and may therefore be useful for augmentation of platelet production. The present invention further discloses isocitrate reversal of iron deprivation.

The invention relates to a flocculant used for purifying the water quality, and in particular relates to a preparation method of solid polymeric ferric chloride silicate flocculant. The preparation method comprises the following steps of: selecting concentrated hydrochloric acid and injecting the selected concentrated hydrochloric acid into water to prepare diluted hydrochloric acid; taking liquid state sodium silicate and putting the liquid state sodium silicate into water to obtain sodium silicate water solution; adding the diluted hydrochloric acid into a reaction tank, slowing injecting the sodium silicate water solution into the reaction tank, and putting ferric trichloride into the reaction tank for a plurality of times for dissolving; standing still to form polymeric iron silicate; and micro-heating to obtain the solid polymeric ferric chloride silicate flocculant. The flocculant only contains two raw materials including Fe and Si, wherein Fe is non-toxic and is essential trace element of the human body, and iron-deficiency anemia can be caused when the human body is lack of the Fe; Si has the function of promoting development and growth; and when the metasilicic acid content in drinking water is more than 25mg/L, the drinking water belongs to mineral water and can be used for preventing diseases after being drunk for a long time, thus having the health care function of prolonging the life. The flocculant not only has excellent turbidity removal function, but also can remove harmful substances such as heavy metal, thus being beneficial to the health of human body.

The invention discloses a complementary deoxyribonucleic acid (cDNA) sequence related to iron deficiency of a plant and an encoded protein and application thereof. A full-length cDNA of a ZmFDR gene is cloned from an iron-deficient induced corn root and the structural characteristic of the full-length cDNA and the functional complementation of the full-length cDNA in yeast are analyzed. The analysis of the structural characteristic indicates that the full length of the ZmFDR gene is 3.3 kb, the gene comprises two open reading frames which are 1,068 bp and 648 bp respectively and the latter is named as ZmFDR4 (SEQ ID NO:1). The encoded protein (SEQ ID NO:2) has four transmembrane domains and has a signal peptide which belongs to a secretory protein and has a similar structure domain to a Flip protein in an III type secretory system. Extensive homology comparison indicates that no homology sequence is found in a higher plant. A yeast functional complementation experiment proves that the expression of the cDNA sequence in the yeast can transfer iron ions effectively.

The invention relates to the technical field of soil fertilizer and in particular provides a compound iron-supplementing agent applicable to a saline and alkaline land as well as preparation and application methods thereof. By combining inorganic iron salt and a fermentation technology, inorganic iron participates in a metabolism process of microorganisms and is converted into an organic chelatingform; the iron with the organic state has relatively good permeability and dissolubility so that the absorption rate and the utilization rate of plants in the saline and alkaline land on an iron element can be improved, and physiological diseases, caused by iron deficiency, on the plants are prevented and alleviated.

The present invention generally relates to iron-polysaccharide (polydextrose) complexes and methods for the preparation thereof. In particular, the present invention relates to iron-polydextrose complexes and methods for the preparation thereof. The inventive complexes are suitable for use in various compositions, including particulate compositions that may be incorporated in various forms of administration, including tablets, capsules, and intravenous formulations. The complexes and compositions of the present invention are suitable for use in the treatment of iron deficient anemia. Advantageously, in addition to providing iron supplementation the complexes and compositions of the present invention provide certain advantages based on the presence of polydextrose (e.g., its low glycemic index and high fiber content).

The invention discloses a Zea mays zinc iron-regulated transporter ZmITR1 gene and its applications. The Zea mays zinc iron-regulated transporter ZmITR1 gene is separated from Zea mays, and the cDNA sequence of the gene is represented by SEQ ID No.1. Subcellular localization shows that the zinc iron-regulated transporter is localized in the plasma membrane and endoplasmic reticulum of a cell, has zinc and iron absorption, transportation and storage effects in plant cells, and particles in detoxification of zinc and iron ions. Quantitative real-time RT-PCR expression analysis shows expression the expression of the ZmITR1 in zinc or iron deficient root and overground parts rises, the expression in the later stage of embryo growth obviously rises, and the ZmITR1 plays an important role in the regulation of the embryo growth. Yeast complementation experiments show that the ZmITR1 gene has a zinc and iron complementation ability. The separated ZmITR1 gene has important application prospects in the regulation of zinc and iron absorption, transportation and storage of plants, promotion of the embryo growth and increase of the content of zinc and iron in grains of food crops.

The present invention discloses the signaling pathway involved erythroid repression by iron deficiency. Further disclosed is anon-toxic small-molecule compound which potently reverses the erythroid repression caused by iron deficiency. The present invention further encompasses novel compounds for inhibition of red cell production, useful, for example, in the treatment of polycythemia vera, a malignancy causing uncontrolled red cell production. These inhibitory compounds also promote megakaryocytic lineage commitment and may therefore be useful for augmentation of platelet production. The present invention further discloses isocitrate reversal of iron deprivation.

The invention discloses an oral nano iron supplement and a preparation method thereof, and effect of the oral nano iron supplement on intestinal flora of rats. The oral nano iron supplement is formedby wrapping iron oxide crystals with a pharmaceutical excipient cellulose. The cellulose comprises sodium carboxymethylcellulose, calcium carboxymethylcellulose, croscarmellose sodium, hydroxy propylcellulose, hydroxypropyl methyl cellulose or cellulose acetate. The iron supplement is used in a rat model with iron-deficiency anemia, can supplement hemoglobin to complete treatment of anemia in 3 weeks, and has higher curative effect and lower side effect than a traditional oral chalybeate ferrous sulfate. Through high-throughput sequencing for intestinal flora of rats, the anaylysis of 16S rRNA shows that the oral iron supplement of the invention can significantly increase abundance and diversity of intestinal flora of rats, promote to restore a steady state of the intestinal flora, facilitate normal growth of the rats and has great application potential in a human body compared to an iron-deficient group and an iron-supplementing group with ferrous sulfate.

The invention provides a food for infantile iron deficiency and a preparation method thereof. The food comprises the following raw materials in part by mass: 40-60 parts of walnut kernel, 10-30 parts of lilium brownii, 10-30 parts of semen nelumbinis and 5-20 parts of peanut kernel. The invention provides the food for infantile iron deficiency and the preparation method thereof, wherein the supplementary food is reasonable in formula, simple in production process, unique in flavor, good in sensory quality and rich in nutrition; the selected raw materials are rich in iron and are supplemented with other natural raw materials for promoting iron absorption, so that infants can absorb iron in supplementary food more quickly and effectively. The food is pollution-free, free of additives, and is a natural infant food which is easy to absorb, high in quality and inexpensive, and healthy.

The present invention relates to an iron-based composition, for use in conditions of total or relative iron deficiency. In particular, the present invention relates to a solid composition, preferablyin the form of powder or granules, for use in the treatment of disorders or diseases related to or derived from an iron deficiency. The composition of the present invention is suitable for pediatric subjects, adolescents, athletes, men, women, pregnant women and elderly. Finally, the present invention relates to a process for preparing said solid composition.

The present invention relates to methods and compounds for regulating or enhancing erythropoiesis and iron metabolism, and for treating or preventing iron deficiency and anemia of chronic disease.

The present invention provides methods and compositions for identifying soybean plants that are tolerant or have improved tolerance, or those that are susceptible to, iron deficient growth conditions. The methods use molecular markers to identify, select, and/or introgress genetic loci modulating phenotypic expression of an iron deficiency tolerance trait in soybean plant breeding. Methods are provided for screening germplasm entries for the performance and expression of this trait.

The invention discloses a vitamin and mineral tablet for pregnant women. The vitamin and mineral tablet includes a tablet core and a film coating layer, and the tablet core includes the following rawmaterials in parts by mass: 200-300 parts of calcium carbonate, 50-100 parts of tricalcium phosphate, 100-150 parts of magnesium carbonate, 4-8 parts of ferrous fumarate, 6-12 parts of polypeptide chelated iron, 15-30 parts of polysaccharide chelated iron, 0.1-0.2 part of vitamin D3, 0.3-0.5 part of folic acid, 3-5 parts of L-ascorbic acid, 0.5-1 part of tocopherol, 100-150 parts of inulin, and 300-400 parts of auxiliary materials. The vitamin and mineral tablet for the pregnant women provided by the invention can comprehensively supplement various vitamins and minerals required during pregnancy, and in particular the vitamin and mineral tablet can promote absorption of iron elements by a body, thereby effectively preventing the occurrence of iron deficiency during the pregnancy.

The invention discloses an iron-deficient culture medium for improving hydrogen production of microalgae, which is prepared by removing FeSO4. 7H2O in a TAP culture solution and adding NaHSO3. By optimizing an existing hydrogen production culture medium formula (a TAP culture solution added with NaHSO3), it is found that in an iron-deficient TAP culture solution added with NaHSO3, the hydrogen production amount of chlamydomonas reinhardtii 124 is increased by more than two times compared with an existing system added with NaHSO3, one culture medium composition is reduced, and cost is reduced.

The invention relates to the technical field of medicines, in particular to an application of an iron agent in preparation of anti-hepatoma metastasis drugs. The iron agent has the advantages that research finds that hepatoma iron deficiency can significantly promote hepatoma metastasis, in-vivo imaging photographing observation shows that supplementing iron to mice significantly inhibits in-situhepatoma metastasis to lung and significantly inhibits the number and distribution range of lung hepatoma cells, and it is indicated that supplementing iron has an anti-hepatoma lung metastasis effect, and the iron agent has a good clinical application prospect.

The invention relates to application of a coding gene for increasing the iron content of plants and increasing the tolerance to iron deficiency stress, and belongs to the technical field of bioengineering. The coding gene sequence for increasing the iron content of plants and increasing the tolerance to iron deficiency stress is shown in a sequence table SEQ ID No: 1. According to the invention, the coding gene for increasing the iron content of the plant and increasing the tolerance to iron deficiency stress is transferred into the plant, so that the coding gene for increasing the iron content in the plant is over-expressed, and the plant shows the tolerance to iron deficiency and has higher iron content than that of a common plant. According to the invention, the coding gene capable of increasing the iron content of plants and increasing the tolerance to iron deficiency stress can provide gene resources and technical support for iron deficiency stress tolerance breeding of crops.