This invention is a method of killing the stealthy intra-cellular
bacteria which are key to the
pathogenesis of both Acquired
Immune Deficiency Syndrome(s) (AIDS) and Cancers. It is currently believed that the
disease(s) known as AIDS are caused by a
virus, the
Human Immunodeficiency Virus (HIV). But much of the long-term (chronic) destruction of the
immune system in AIDS is actually caused by tiny, stealthy, antibiotic-
resistant bacteria. The HIV
virus weakens the
immune system so that the stealthy
bacteria can proliferate out-of-control, and the damage of AIDS is done just as much by these
bacteria as by any
virus. These very tiny L-form
Cell-Wall-Deficient (CWD) antibiotic-
resistant bacteria live within the
cytoplasm of cells, including the phagocytic cells (e.g. monocytes, macrophages, lymphocytes, neutrophils and polymorphonuclear cells) of the
immune system itself. The cellular proliferation in
Cancer is catalysed the action of the same tiny L-form bacteria. They cause the
cell nucleus to release mRNA signaling the Th1
cytokine cascade without the need for conventional signaling via, for example, CD4+ T-Lymphocytes. Some of these Cytokines and Chemokines, including, without limitation, Cellular Adhesion Molecule (
CAM), create the environment which allows the cellular proliferation to start, and then allows the cancerous growth to
gain a foothold in the body. Killing these stealthy pathogens removes the environment needed to initiate and feed the cellular proliferation commonly called ‘
Cancer,’ and prevents the chronic phase of the
disease AIDS. This invention achieves its objective partly by reducing the ability of these tiny L-form, intra-phagocytic bacteria to translate proteins within their 70S
Ribosome. The 30S and 50S subunits of the bacterial
ribosome are targeted both individually and collectively. Further, this invention activates the
innate immune system with
agonist(s) for the VDR
Nuclear Receptor, and modulates the availability of endogenous ligands to the PPAR, GCR and CB1 receptors, conditioning the immune
system to more easily recognize and kill these tiny bacterial pathogens.